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Objective: The incompatible response hypothesis states that inducing incompatible emotional states mitigates the effect of situational risk factors on aggressive behavior. The current study extended this approach to situated aggression control to withdrawal-related negative emotions. We proposed that even a negative affective state can be incompatible with aggression if its basic motivational orientation counteracts the approach orientation underlying anger and aggression. Specifically, we predicted that although it is inherently negative, sadness may reduce anger-driven aggressive behavior. Method: An experiment was conducted (N = 149) in which half the participants were angered by means of a frustrating number-sequences task, whereas the other half were asked to engage in a similar but nonfrustrating task. To counteract anger-driven aggressive behavior, sadness was induced in half the participants by asking them to recall a sad personal episode. Participants in the no-sadness group recalled an affectively neutral episode. Finally, participants were asked to choose the difficulty level of the number sequences that would ostensibly be assigned to future participants, with the number of difficult sequences chosen indicating the strength of the aggressive response. Results: As predicted, the induction of sadness buffered anger-related aggressive behavior. Anger translated into aggression in the control condition but not in the sadness condition. The aggression-inhibiting effect of the experience of sadness was found to be driven by the compensating coactivation of anger and sadness. Conclusions: The results support the extension of the incompatible response hypothesis to withdrawal-related negative emotions and shed further light on the underlying processes.
Using two clinical samples of patients, the presented studies examined the construct validity of the recently revised Anxiety Sensitivity Index-3 (ASI-3). Confirmatory factor analyses established a clear three-factor structure that corresponds to the postulated subdivision of the construct into correlated somatic, social, and cognitive components. Participants with different primary clinical diagnoses differed from each other on the ASI-3 subscales in theoretically meaningful ways. Specifically, the ASI-3 successfully discriminated patients with anxiety disorders from patients with nonanxiety disorders. Moreover, patients with panic disorder or agoraphobia manifested more somatic concerns than patients with other anxiety disorders and patients with nonanxiety disorders. Finally, correlations of the ASI-3 scales with other measures of clinical symptoms and negative affect substantiated convergent and discriminant validity. Substantial positive correlations were found between the ASI-3 Somatic Concerns and body vigilance, between Social Concerns and fear of negative evaluation and socially inhibited behavior, and between Cognitive Concerns and depression symptoms, anxiety, fear of negative evaluation, and subjective complaints. Moreover, Social Concerns correlated negatively with dominant and intrusive behavior. Results are discussed with respect to the contribution of the ASI-3 to the assessment of anxiety-related disorders.