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During the second phase of the Alpine Fault, Deep Fault Drilling Project (DFDP) in the Whataroa River, South Westland, New Zealand, bedrock was encountered in the DFDP-2B borehole from 238.5–893.2 m Measured Depth (MD). Continuous sampling and meso- to microscale characterisation of whole rock cuttings established that, in sequence, the borehole sampled amphibolite facies, Torlesse Composite Terrane-derived schists, protomylonites and mylonites, terminating 200–400 m above an Alpine Fault Principal Slip Zone (PSZ) with a maximum dip of 62°. The most diagnostic structural features of increasing PSZ proximity were the occurrence of shear bands and reduction in mean quartz grain sizes. A change in composition to greater mica:quartz + feldspar, most markedly below c. 700 m MD, is inferred to result from either heterogeneous sampling or a change in lithology related to alteration. Major oxide variations suggest the fault-proximal Alpine Fault alteration zone, as previously defined in DFDP-1 core, was not sampled.
The organic market is characterized by remarkable disparities, and confusion persists about which motives drive organic consumption. To understand them, this research introduces the idea that the same consumer motives can exert different and potentially opposite impacts when organic consumption patterns unfold. The proposed multistage theory of differential effects distinguishes a participation stage, when consumers decide whether to purchase organic at all, and an expenditure stage, when consumers decide about how much of their budget to spend on organic products across purchases. An analysis of shopping patterns of approximately 14,000 households confirms the proposed differential influences: Other-oriented motives (care for others and the environment) support participation but impede sustained expenditures. Only self-oriented motives (hedonism) foster both participation and expenditures. The results pinpoint the need to rethink organic consumption as a stage-specific problem, which opens up new perspectives for managers about an old but persistent problem.
There are two fundamental ways in which consumers can express their concerns and obligations for society through their consumption decisions: They can boycott companies that they deem to be irresponsible or they may deliberately buy from companies that they perceive to act responsibly (‘buycott’). It has been largely ignored that individuals are driven by different motivational mechanisms to join boycotts and buycotts (punishment vs. reward of corporate behaviors), and thus, these mechanisms have disparate implications for the participating individual (e.g., high vs. low subjective costs because of a restriction in consumption habits). This paper fills this void and develops a framework suggesting that the extent to which consumers translate their concerns and obligations for society into a willingness to boycott and/or buycott is bounded by self-interest. Using a unique, representative sample of 1833 German consumers, this study reveals that the effects of environmental concerns and universalism on buycotting are amplified by hedonism, while the effects of social concern on buycotting and boycotting are attenuated by hedonism and simplicity, respectively. These results have far-reaching implications for organizations and policy planners who aim to change corporate behavior.
A catalog of genetic loci associated with kidney function from analyses of a million individuals
(2019)
Chronic kidney disease (CKD) is responsible for a public health burden with multi-systemic complications. Through transancestry meta-analysis of genome-wide association studies of estimated glomerular filtration rate (eGFR) and independent replication (n = 1,046,070), we identified 264 associated loci (166 new). Of these,147 were likely to be relevant for kidney function on the basis of associations with the alternative kidney function marker blood urea nitrogen (n = 416,178). Pathway and enrichment analyses, including mouse models with renal phenotypes, support the kidney as the main target organ. A genetic risk score for lower eGFR was associated with clinically diagnosed CKD in 452,264 independent individuals. Colocalization analyses of associations with eGFR among 783,978 European-ancestry individuals and gene expression across 46 human tissues, including tubulo-interstitial and glomerular kidney compartments, identified 17 genes differentially expressed in kidney. Fine-mapping highlighted missense driver variants in 11 genes and kidney-specific regulatory variants. These results provide a comprehensive priority list of molecular targets for translational research.