@article{WarringtonBeaumontHorikoshietal.2019,
  author    = {Warrington, Nicole and Beaumont, Robin and Horikoshi, Momoko and Day, Felix R. and Helgeland, {\O}yvind and Laurin, Charles and Bacelis, Jonas and Peng, Shouneng and Hao, Ke and Feenstra, Bjarke and Wood, Andrew R. and Mahajan, Anubha and Tyrrell, Jessica and Robertson, Neil R. and Rayner, N. William and Qiao, Zhen and Moen, Gunn-Helen and Vaudel, Marc and Marsit, Carmen and Chen, Jia and Nodzenski, Michael and Schnurr, Theresia M. and Zafarmand, Mohammad Hadi and Bradfield, Jonathan P. and Grarup, Niels and Kooijman, Marjolein N. and Li-Gao, Ruifang and Geller, Frank and Ahluwalia, Tarunveer Singh and Paternoster, Lavinia and Rueedi, Rico and Huikari, Ville and Hottenga, Jouke-Jan and Lyytik{\"a}inen, Leo-Pekka and Cavadino, Alana and Metrustry, Sarah and Cousminer, Diana L. and Wu, Ying and Thiering, Elisabeth Paula and Wang, Carol A. and Have, Christian Theil and Vilor-Tejedor, Natalia and Joshi, Peter K. and Painter, Jodie N. and Ntalla, Ioanna and Myhre, Ronny and Pitk{\"a}nen, Niina and van Leeuwen, Elisabeth M. and Joro, Raimo and Lagou, Vasiliki and Richmond, Rebecca C. and Espinosa, Ana and Barton, Sheila J. and Inskip, Hazel M. and Holloway, John W. and Santa-Marina, Loreto and Estivill, Xavier and Ang, Wei and Marsh, Julie A. and Reichetzeder, Christoph and Marullo, Letizia and Hocher, Berthold and Lunetta, Kathryn L. and Murabito, Joanne M. and Relton, Caroline L. and Kogevinas, Manolis and Chatzi, Leda and Allard, Catherine and Bouchard, Luigi and Hivert, Marie-France and Zhang, Ge and Muglia, Louis J. and Heikkinen, Jani and Morgen, Camilla S. and van Kampen, Antoine H. C. and van Schaik, Barbera D. C. and Mentch, Frank D. and Langenberg, Claudia and Scott, Robert A. and Zhao, Jing Hua and Hemani, Gibran and Ring, Susan M. and Bennett, Amanda J. and Gaulton, Kyle J. and Fernandez-Tajes, Juan and van Zuydam, Natalie R. and Medina-Gomez, Carolina and de Haan, Hugoline G. and Rosendaal, Frits R. and Kutalik, Zolt{\´a}n and Marques-Vidal, Pedro and Das, Shikta and Willemsen, Gonneke and Mbarek, Hamdi and M{\"u}ller-Nurasyid, Martina and Standl, Marie and Appel, Emil V. R. and Fonvig, Cilius Esmann and Trier, Caecilie and van Beijsterveldt, Catharina E. M. and Murcia, Mario and Bustamante, Mariona and Bon{\`a}s-Guarch, S{\´i}lvia and Hougaard, David M. and Mercader, Josep M. and Linneberg, Allan and Schraut, Katharina E. and Lind, Penelope A. and Medland, Sarah Elizabeth and Shields, Beverley M. and Knight, Bridget A. and Chai, Jin-Fang and Panoutsopoulou, Kalliope and Bartels, Meike and S{\´a}nchez, Friman and Stokholm, Jakob and Torrents, David and Vinding, Rebecca K. and Willems, Sara M. and Atalay, Mustafa and Chawes, Bo L. and Kovacs, Peter and Prokopenko, Inga and Tuke, Marcus A. and Yaghootkar, Hanieh and Ruth, Katherine S. and Jones, Samuel E. and Loh, Po-Ru and Murray, Anna and Weedon, Michael N. and T{\"o}njes, Anke and Stumvoll, Michael and Michaelsen, Kim Fleischer and Eloranta, Aino-Maija and Lakka, Timo A. and van Duijn, Cornelia M. and Kiess, Wieland and Koerner, Antje and Niinikoski, Harri and Pahkala, Katja and Raitakari, Olli T. and Jacobsson, Bo and Zeggini, Eleftheria and Dedoussis, George V. and Teo, Yik-Ying and Saw, Seang-Mei and Montgomery, Grant W. and Campbell, Harry and Wilson, James F. and Vrijkotte, Tanja G. M. and Vrijheid, Martine and de Geus, Eco J. C. N. and Hayes, M. Geoffrey and Kadarmideen, Haja N. and Holm, Jens-Christian and Beilin, Lawrence J. and Pennell, Craig E. and Heinrich, Joachim and Adair, Linda S. and Borja, Judith B. and Mohlke, Karen L. and Eriksson, Johan G. and Widen, Elisabeth E. and Hattersley, Andrew T. and Spector, Tim D. and Kaehoenen, Mika and Viikari, Jorma S. and Lehtimaeki, Terho and Boomsma, Dorret I. and Sebert, Sylvain and Vollenweider, Peter and Sorensen, Thorkild I. A. and Bisgaard, Hans and Bonnelykke, Klaus and Murray, Jeffrey C. and Melbye, Mads and Nohr, Ellen A. and Mook-Kanamori, Dennis O. and Rivadeneira, Fernando and Hofman, Albert and Felix, Janine F. and Jaddoe, Vincent W. V. and Hansen, Torben and Pisinger, Charlotta and Vaag, Allan A. and Pedersen, Oluf and Uitterlinden, Andre G. and Jarvelin, Marjo-Riitta and Power, Christine and Hypponen, Elina and Scholtens, Denise M. and Lowe, William L. and Smith, George Davey and Timpson, Nicholas J. and Morris, Andrew P. and Wareham, Nicholas J. and Hakonarson, Hakon and Grant, Struan F. A. and Frayling, Timothy M. and Lawlor, Debbie A. and Njolstad, Pal R. and Johansson, Stefan and Ong, Ken K. and McCarthy, Mark I. and Perry, John R. B. and Evans, David M. and Freathy, Rachel M.},
  title     = {Maternal and fetal genetic effects on birth weight and their relevance to cardio-metabolic risk factors},
  series = {Nature genetics},
  volume    = {51},
  journal   = {Nature genetics},
  number    = {5},
  publisher = {Nature Publ. Group},
  address   = {New York},
  organization    = {EGG Consortium},
  issn      = {1061-4036},
  pages     = {804 -- +},
  year      = {2019},
  abstract  = {Birth weight variation is influenced by fetal and maternal genetic and non-genetic factors, and has been reproducibly associated with future cardio-metabolic health outcomes. In expanded genome-wide association analyses of own birth weight (n = 321,223) and offspring birth weight (n = 230,069 mothers), we identified 190 independent association signals (129 of which are novel). We used structural equation modeling to decompose the contributions of direct fetal and indirect maternal genetic effects, then applied Mendelian randomization to illuminate causal pathways. For example, both indirect maternal and direct fetal genetic effects drive the observational relationship between lower birth weight and higher later blood pressure: maternal blood pressure-raising alleles reduce offspring birth weight, but only direct fetal effects of these alleles, once inherited, increase later offspring blood pressure. Using maternal birth weight-lowering genotypes to proxy for an adverse intrauterine environment provided no evidence that it causally raises offspring blood pressure, indicating that the inverse birth weight-blood pressure association is attributable to genetic effects, and not to intrauterine programming.},
  language  = {en}
}
@misc{KaminskiSchlagenhaufRappetal.2018,
  author    = {Kaminski, Jakob A. and Schlagenhauf, Florian and Rapp, Michael A. and Awasthi, Swapnil and Ruggeri, Barbara and Deserno, Lorenz and Banaschewski, Tobias and Bokde, Arun L. W. and Bromberg, Uli and B{\"u}chel, Christian and Quinlan, Erin Burke and Desrivi{\`e}res, Sylvane and Flor, Herta and Frouin, Vincent and Garavan, Hugh and Gowland, Penny and Ittermann, Bernd and Martinot, Jean-Luc and Paill{\`e}re Martinot, Marie-Laure and Nees, Frauke and Papadopoulos Orfanos, Dimitri and Paus, Tom{\´a}š and Poustka, Luise and Smolka, Michael N. and Fr{\"o}hner, Juliane H. and Walter, Henrik and Whelan, Robert and Ripke, Stephan and Schumann, Gunter and Heinz, Andreas},
  title     = {Epigenetic variance in dopamine D2 receptor},
  series = {Postprints der Universit{\"a}t Potsdam : Mathematisch-Naturwissenschaftliche Reihe},
  journal   = {Postprints der Universit{\"a}t Potsdam : Mathematisch-Naturwissenschaftliche Reihe},
  number    = {950},
  issn      = {1866-8372},
  doi       = {10.25932/publishup-42568},
  url       = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-425687},
  pages     = {13},
  year      = {2018},
  abstract  = {Genetic and environmental factors both contribute to cognitive test performance. A substantial increase in average intelligence test results in the second half of the previous century within one generation is unlikely to be explained by genetic changes. One possible explanation for the strong malleability of cognitive performance measure is that environmental factors modify gene expression via epigenetic mechanisms. Epigenetic factors may help to understand the recent observations of an association between dopamine-dependent encoding of reward prediction errors and cognitive capacity, which was modulated by adverse life events. The possible manifestation of malleable biomarkers contributing to variance in cognitive test performance, and thus possibly contributing to the "missing heritability" between estimates from twin studies and variance explained by genetic markers, is still unclear. Here we show in 1475 healthy adolescents from the IMaging and GENetics (IMAGEN) sample that general IQ (gIQ) is associated with (1) polygenic scores for intelligence, (2) epigenetic modification of DRD2 gene, (3) gray matter density in striatum, and (4) functional striatal activation elicited by temporarily surprising reward-predicting cues. Comparing the relative importance for the prediction of gIQ in an overlapping subsample, our results demonstrate neurobiological correlates of the malleability of gIQ and point to equal importance of genetic variance, epigenetic modification of DRD2 receptor gene, as well as functional striatal activation, known to influence dopamine neurotransmission. Peripheral epigenetic markers are in need of confirmation in the central nervous system and should be tested in longitudinal settings specifically assessing individual and environmental factors that modify epigenetic structure.},
  language  = {en}
}
@article{KaminskiSchlagenhaufRappetal.2018,
  author    = {Kaminski, Jakob A. and Schlagenhauf, Florian and Rapp, Michael A. and Awasthi, Swapnil and Ruggeri, Barbara and Deserno, Lorenz and Banaschewski, Tobias and Bokde, Arun L. W. and Bromberg, Uli and B{\"u}chel, Christian and Quinlan, Erin Burke and Desrivieres, Sylvane and Flor, Herta and Frouin, Vincent and Garavan, Hugh and Gowland, Penny and Ittermann, Bernd and Martinot, Jean-Luc and Martinot, Marie-Laure Paillere and Nees, Frauke and Orfanos, Dimitri Papadopoulos and Paus, Tomas and Poustka, Luise and Smolka, Michael N. and Fr{\"o}hner, Juliane H. and Walter, Henrik and Whelan, Robert and Ripke, Stephan and Schumann, Gunter and Heinz, Andreas},
  title     = {Epigenetic variance in dopamine D2 receptor},
  series = {Translational Psychiatry},
  volume    = {8},
  journal   = {Translational Psychiatry},
  publisher = {Nature Publ. Group},
  address   = {New York},
  organization    = {IMAGEN Consortium},
  issn      = {2158-3188},
  doi       = {10.1038/s41398-018-0222-7},
  pages     = {11},
  year      = {2018},
  abstract  = {Genetic and environmental factors both contribute to cognitive test performance. A substantial increase in average intelligence test results in the second half of the previous century within one generation is unlikely to be explained by genetic changes. One possible explanation for the strong malleability of cognitive performance measure is that environmental factors modify gene expression via epigenetic mechanisms. Epigenetic factors may help to understand the recent observations of an association between dopamine-dependent encoding of reward prediction errors and cognitive capacity, which was modulated by adverse life events. The possible manifestation of malleable biomarkers contributing to variance in cognitive test performance, and thus possibly contributing to the "missing heritability" between estimates from twin studies and variance explained by genetic markers, is still unclear. Here we show in 1475 healthy adolescents from the IMaging and GENetics (IMAGEN) sample that general IQ (gIQ) is associated with (1) polygenic scores for intelligence, (2) epigenetic modification of DRD2 gene, (3) gray matter density in striatum, and (4) functional striatal activation elicited by temporarily surprising reward-predicting cues. Comparing the relative importance for the prediction of gIQ in an overlapping subsample, our results demonstrate neurobiological correlates of the malleability of gIQ and point to equal importance of genetic variance, epigenetic modification of DRD2 receptor gene, as well as functional striatal activation, known to influence dopamine neurotransmission. Peripheral epigenetic markers are in need of confirmation in the central nervous system and should be tested in longitudinal settings specifically assessing individual and environmental factors that modify epigenetic structure.},
  language  = {en}
}
@misc{KaminskiSchlagenhaufRappetal.2018,
  author    = {Kaminski, Jakob and Schlagenhauf, Florian and Rapp, Michael A. and Awasthi, Swapnil and Ruggeri, Barbara and Deserno, Lorenz and Laura, Daedelow and Banaschewski, Tobias and Bokde, Arun and Quinlan, Erin Burke and Buechel, Christian and Bromberg, Uli and Desrivieres, Sylvane and Flor, Herta and Frouin, Vincent and Garavan, Hugh and Gowland, Penny and Ittermann, Bernd and Martinot, Jean-Luc and Martinot, Marie-Laure Paillere and Nees, Frauke and Orfanos, Dimitri Papadopoulos and Paus, Tomas and Poustka, Luise and Smolka, Michael and Froehner, Juliane and Walter, Henrik and Whelan, Robert and Ripke, Stephan and Schumann, Gunter and Heinz, Andreas},
  title     = {Variance in Dopaminergic Markers},
  series = {Biological psychiatry : a journal of psychiatric neuroscience and therapeutics ; a publication of the Society of Biological Psychiatry},
  volume    = {83},
  journal   = {Biological psychiatry : a journal of psychiatric neuroscience and therapeutics ; a publication of the Society of Biological Psychiatry},
  number    = {9},
  publisher = {Elsevier},
  address   = {New York},
  organization    = {IMAGEN Consortium},
  issn      = {0006-3223},
  doi       = {10.1016/j.biopsych.2018.02.311},
  pages     = {S118 -- S118},
  year      = {2018},
  language  = {en}
}
@article{YoungUedaGuehretal.2018,
  author    = {Young, Linda and Ueda, Kiyoshi and G{\"u}hr, Markus and Bucksbaum, Philip H. and Simon, Marc and Mukamel, Shaul and Rohringer, Nina and Prince, Kevin C. and Masciovecchio, Claudio and Meyer, Michael and Rudenko, Artem and Rolles, Daniel and Bostedt, Christoph and Fuchs, Matthias and Reis, David A. and Santra, Robin and Kapteyn, Henry and Murnane, Margaret and Ibrahim, Heide and Legare, Francois and Vrakking, Marc and Isinger, Marcus and Kroon, David and Gisselbrecht, Mathieu and W{\"o}rner, Hans Jakob and Leone, Stephen R.},
  title     = {Roadmap of ultrafast x-ray atomic and molecular physics},
  series = {Journal of physics : B, Atomic, molecular and optical physics},
  volume    = {51},
  journal   = {Journal of physics : B, Atomic, molecular and optical physics},
  number    = {3},
  publisher = {IOP Publ. Ltd.},
  address   = {Bristol},
  issn      = {0953-4075},
  doi       = {10.1088/1361-6455/aa9735},
  pages     = {45},
  year      = {2018},
  abstract  = {X-ray free-electron lasers (XFELs) and table-top sources of x-rays based upon high harmonic generation (HHG) have revolutionized the field of ultrafast x-ray atomic and molecular physics, largely due to an explosive growth in capabilities in the past decade. XFELs now provide unprecedented intensity (10(20) W cm(-2)) of x-rays at wavelengths down to similar to 1 Angstrom, and HHG provides unprecedented time resolution (similar to 50 attoseconds) and a correspondingly large coherent bandwidth at longer wavelengths. For context, timescales can be referenced to the Bohr orbital period in hydrogen atom of 150 attoseconds and the hydrogen-molecule vibrational period of 8 femtoseconds; wavelength scales can be referenced to the chemically significant carbon K-edge at a photon energy of similar to 280 eV (44 Angstroms) and the bond length in methane of similar to 1 Angstrom. With these modern x-ray sources one now has the ability to focus on individual atoms, even when embedded in a complex molecule, and view electronic and nuclear motion on their intrinsic scales (attoseconds and Angstroms). These sources have enabled coherent diffractive imaging, where one can image non-crystalline objects in three dimensions on ultrafast timescales, potentially with atomic resolution. The unprecedented intensity available with XFELs has opened new fields of multiphoton and nonlinear x-ray physics where behavior of matter under extreme conditions can be explored. The unprecedented time resolution and pulse synchronization provided by HHG sources has kindled fundamental investigations of time delays in photoionization, charge migration in molecules, and dynamics near conical intersections that are foundational to AMO physics and chemistry. This roadmap coincides with the year when three new XFEL facilities, operating at Angstrom wavelengths, opened for users (European XFEL, Swiss-FEL and PAL-FEL in Korea) almost doubling the present worldwide number of XFELs, and documents the remarkable progress in HHG capabilities since its discovery roughly 30 years ago, showcasing experiments in AMO physics and other applications. Here we capture the perspectives of 17 leading groups and organize the contributions into four categories: ultrafast molecular dynamics, multidimensional x-ray spectroscopies; high-intensity x-ray phenomena; attosecond x-ray science.},
  language  = {en}
}
@misc{BattegayBierwirthDubrauetal.2006,
  author    = {Battegay, Caspar and Bierwirth, Sabine and Dubrau, Alexander and Feinberg, Anat and Franz-Klauser, Olivia and Gr{\"o}zinger, Elvira and Gr{\"o}zinger, Karl E. and Haußig, Hans-Michael and Hessing, Jakob and Horch, Hans-Otto and Hudak-Lazi{\c{c}}, Susanne and J{\"u}tte, Daniel and J{\"u}tte, Robert and Lutz, Edith and Madsen, Hendrik and Riemer, Nathanael and Schl{\"o}r, Joachim},
  title     = {PaRDeS : Zeitschrift der Vereinigung f{\"u}r J{\"u}dische Studien e.V. = Themenheft zum 150. Todestag von Heinrich Heine},
  number    = {12},
  editor    = {Riemer, Nathanael},
  publisher = {Universit{\"a}tsverlag Potsdam},
  address   = {Potsdam},
  isbn      = {978-3-939469-19-3},
  issn      = {1862-7684},
  url       = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus-14636},
  year      = {2006},
  abstract  = {Aus dem Inhalt dieser Ausgabe: Artikel und Miszellen: Karl E. Gr{\"o}zinger: Manfred Voigts zum Sechzigsten - 2.5.2006 Caspar Battegay: Wie nicht erinnern? - Die Frage nach der J{\"u}dischkeit in Heinrich Heines autobiographischen Texten Edith Lutz: Heinrich Heine im "Verein f{\"u}r Cultur und Wissenschaft der Juden" Jakob Hessing: Wahrheit und Dichtung - Die Damaskusaff{\"a}re und Heines Der Rabbi von Bacharach Elvira Gr{\"o}zinger: Im Venusberg. Zu Gesundheit und Krankheit bei Heinrich Heine zwischen Eros und Thanatos Sabine Bierwirth: Meilenstein der Zeitgeschichtsschreibung: Heinrich Heines Berichte {\"u}ber die Judenverfolgung in Damaskus 1840 Hans Otto Horch: Die unheilbar große Br{\"u}derkrankheit - Zum programmatischen Zeitgedicht Das neue Israelitische Hospital zu Hamburg von H. Heine Hendrik Madsen: Vom {\"U}berleben der toten Schrift - Eine medientheoretisch orientierte Lekt{\"u}re des Jeremiabuches Manfred Voigts: Moses Mendelssohn und Franz Kafka: Die Heilige Schrift Olivia Franz-Klauser: Samaritanerforschung im 19. Jahrhundert: Die Anf{\"a}nge der historischen Kritik im Schatten religi{\"o}ser Vorurteile, gezeigt an der Rezeption Moritz Heidenheims (1824-1898) Rezensionen: Mark R. Cohen: Unter Kreuz und Halbmond. Die Juden im Mittelalter (Daniel J{\"u}tte) Erika Timm: Historische jiddische Semantik. (Karl E. Gr{\"o}zinger) David B. Ruderman, Giuseppe Veltri (Hrsg.): Cultural Intermediaries. Jewish Intellectuals in Early Modern Italy (Daniel J{\"u}tte) Gian Maria Varanini, Reinhold C. Mueller (Hrsg.): Ebrei nella Terraferma veneta del Quattrocento (Daniel J{\"u}tte) Stefanie B. Siegmund: The Medici State and the Ghetto of Florence (Daniel J{\"u}tte) Johannes Mordstein: Selbstbewußte Untert{\"a}nigkeit (Robert J{\"u}tte) Werner Heegewaldt, Oliver Sander (Hrsg.): Salomo Sachs (Elvira Gr{\"o}zinger) Philipp Theisohn: Die Urbarkeit der Zeichen. Zionismus und Literatur - eine andere Poetik der Moderne (Joachim Schl{\"o}r) Jascha Nemtsov: Die Neue J{\"u}dische Schule in der Musik (Susanne Hudak-Lazi{\c{c}}) Ines Sonder: Gartenst{\"a}dte in Eretz Israel. (Robert J{\"u}tte) Cilly Kugelmann (Hrsg.): Weihnukka. Geschichten von Weihnachten und Chanukka. (Daniel J{\"u}tte) Moshe Zimmermann, Yotam Hotam (Hrsg.): Zweimal Heimat. Die Jeckes zwischen Mitteleuropa und Nahost. (Anat Feinberg) Birgit Schlachter: Schreibweisen der Abwesenheit. (Elvira Gr{\"o}zinger) J{\"u}dische Studien in aller Welt: Juden und Judentum im Iran - Einige zuf{\"a}llige und weniger zuf{\"a}llige Reiseeindr{\"u}cke (Hans-Michael Haußig) Nachrichten R{\"u}ckblicke},
  language  = {de}
}
@article{KalkuhlFernandezMilanSchwerhoffetal.2018,
  author    = {Kalkuhl, Matthias and Fernandez Milan, Blanca and Schwerhoff, Gregor and Jakob, Michael and Hahnen, Maren and Creutzig, Felix},
  title     = {Can land taxes foster sustainable development?},
  series = {Land use policy : the international journal covering all aspects of land use},
  volume    = {78},
  journal   = {Land use policy : the international journal covering all aspects of land use},
  publisher = {Elsevier Science Publishers Ltd.},
  address   = {Oxford},
  issn      = {0264-8377},
  doi       = {10.1016/j.landusepol.2018.07.008},
  pages     = {338 -- 352},
  year      = {2018},
  abstract  = {Economists argue that land rent taxation is an ideal form of taxation as it causes no deadweight losses. Nevertheless, pure land rent taxation is rarely applied. This paper revisits the case of land taxation for developing countries. We first provide an up-to-date review on land taxation in development countries, including feasibility and implementation challenges. We then simulate land tax reforms for Rwanda, Peru, Nicaragua and Indonesia, based on household surveys. We find that (i) land taxes provide a substantial untapped potential for tax revenues at minimal deadweight losses; that (ii) linear land value taxes tend to put a high relative burden on poor households as land ownership is pervasive; (iii) non-linear tax schemes could avoid adverse effects on the poor; and that (iv) with technological advances, administrative costs of land taxes have reduced substantially and are outweighed by tax revenues and co-benefits of formalized land tenure. Enforcement and compliance remain, however, a key challenge.},
  language  = {en}
}
@article{vanderMeijReimannVornehmetal.2019,
  author    = {van der Meij, Marijn W. and Reimann, Tony and Vornehm, V. K. and Temme, Arnaud J. A. M. and Wallinga, Jakob and van Beek, Roy and Sommer, Michael},
  title     = {Reconstructing rates and patterns of colluvial soil redistribution in agrarian (hummocky) landscapes},
  series = {Earth surface processes and landforms : the journal of the British Geomorphological Research Group},
  volume    = {44},
  journal   = {Earth surface processes and landforms : the journal of the British Geomorphological Research Group},
  number    = {12},
  publisher = {Wiley},
  address   = {Hoboken},
  issn      = {0197-9337},
  doi       = {10.1002/esp.4671},
  pages     = {2408 -- 2422},
  year      = {2019},
  abstract  = {Humans have triggered or accelerated erosion processes since prehistoric times through agricultural practices. Optically stimulated luminescence (OSL) is widely used to quantify phases and rates of the corresponding landscape change, by measuring the last moment of daylight exposure of sediments. However, natural and anthropogenic mixing processes, such as bioturbation and tillage, complicate the use of OSL as grains of different depositional ages become mixed, and grains become exposed to light even long after the depositional event of interest. Instead, OSL determines the stabilization age, indicating when sediments were buried below the active mixing zone. These stabilization ages can cause systematic underestimation when calculating deposition rates. Our focus is on colluvial deposition in a kettle hole in the Uckermark region, northeastern Germany. We took 32 samples from five locations in the colluvium filling the kettle hole to study both spatial and temporal patterns in colluviation. We combined OSL dating with advanced age modelling to determine the stabilization age of colluvial sediments. These ages were combined with an archaeological reconstruction of historical ploughing depths to derive the levels of the soil surface at the moment of stabilization; the deposition depths, which were then used to calculate unbiased deposition rates. We identified two phases of colluvial deposition. The oldest deposits (similar to 5 ka) were located at the fringe of the kettle hole and accumulated relatively slowly, whereas the youngest deposits (<0.3 ka) rapidly filled the central kettle hole with rates of two orders of magnitude higher. We suggest that the latter phase is related to artificial drainage, facilitating accessibility in the central depression for agricultural practices. Our results show the need for numerical dating techniques that take archaeological and soil-geomorphological information into account to identify spatiotemporal patterns of landscape change, and to correctly interpret landscape dynamics in anthropogenically influenced hilly landscapes. (c) 2019 The Authors. Earth Surface Processes and Landforms Published by John Wiley \& Sons Ltd.},
  language  = {en}
}
@article{FegerFajolLebedevaetal.2013,
  author    = {Feger, Martina and Fajol, Abul and Lebedeva, Aleksandra and Meissner, Adrian and Michael, Diana and V{\"o}lkl, Jakob and Alesutan, Ioana and Schleicher, Erwin and Reichetzeder, Christoph and Hocher, Berthold and Qadri, Syed M. and Lang, Florian},
  title     = {Effect of Carbon Monoxide Donor CORM-2 on Vitamin D-3 Metabolism},
  series = {Kidney \& blood pressure research : official organ of the Gesellschaft f{\"u}r Nephrologie},
  volume    = {37},
  journal   = {Kidney \& blood pressure research : official organ of the Gesellschaft f{\"u}r Nephrologie},
  number    = {4-5},
  publisher = {Karger},
  address   = {Basel},
  issn      = {1420-4096},
  doi       = {10.1159/000355730},
  pages     = {496 -- 505},
  year      = {2013},
  abstract  = {Background/Aims: Carbon monoxide (CO) interferes with cytochrome-dependent cellular functions and acts as gaseous transmitter. CO is released from CO-releasing molecules (CORM) including tricarbonyl-dichlororuthenium (II) dimer (CORM-2), molecules considered for the treatment of several disorders including vascular dysfunction, inflammation, tissue ischemia and organ rejection. Cytochrome P450-sensitive function include formation of 1,25-dihydroxyvitamin D-3 (1,25(OH)(2)D-3) by renal 25-hydroxyvitamin D-3 1-alpha-hydroxylase (Cyp27b1). The enzyme is regulated by PTH, FGF23 and klotho. 1,25(OH)(2)D-3 regulates Ca2+ and phosphate transport as well as klotho expression. The present study explored, whether CORM-2 influences 1,25(OH)(2)D-3 formation and klotho expression. Methods: Mice were treated with intravenous CORM-2 (20 mg/kg body weight). Plasma 1,25(OH)(2)D-3 and FGF23 concentrations were determined by ELISA, phosphate, calcium and creatinine concentrations by colorimetric methods, transcript levels by quantitative RT-PCR and protein expression by western blotting. Fgf23 mRNA transcript levels were further determined in rat osteosarcoma UMR106 cells without or with prior treatment for 24 hours with 20 mu M CORM-2. Results: CORM-2 injection within 24 hours significantly increased FGF23 plasma levels and decreased 1,25(OH)(2)D-3 plasma levels, renal Cyp27b1 gene expression as well as renal klotho protein abundance and transcript levels. Moreover, treatment of UMR106 cells with CORM-2 significantly increased Fgf23 transcript levels. Conclusion: CO-releasing molecule CORM-2 enhances FGF23 expression and release and decreases klotho expression and 1,25(OH)(2)D-3 synthesis.},
  language  = {en}
}
@article{ReichsteinBahnCiaisetal.2013,
  author    = {Reichstein, Markus and Bahn, Michael and Ciais, Philippe and Frank, Dorothea and Mahecha, Miguel D. and Seneviratne, Sonia I. and Zscheischler, Jakob and Beer, Christian and Buchmann, Nina and Frank, David C. and Papale, Dario and Rammig, Anja and Smith, Pete and Thonicke, Kirsten and van der Velde, Marijn and Vicca, Sara and Walz, Ariane and Wattenbach, Martin},
  title     = {Climate extremes and the carbon cycle},
  series = {Nature : the international weekly journal of science},
  volume    = {500},
  journal   = {Nature : the international weekly journal of science},
  number    = {7462},
  publisher = {Nature Publ. Group},
  address   = {London},
  issn      = {0028-0836},
  doi       = {10.1038/nature12350},
  pages     = {287 -- 295},
  year      = {2013},
  abstract  = {The terrestrial biosphere is a key component of the global carbon cycle and its carbon balance is strongly influenced by climate. Continuing environmental changes are thought to increase global terrestrial carbon uptake. But evidence is mounting that climate extremes such as droughts or storms can lead to a decrease in regional ecosystem carbon stocks and therefore have the potential to negate an expected increase in terrestrial carbon uptake. Here we explore the mechanisms and impacts of climate extremes on the terrestrial carbon cycle, and propose a pathway to improve our understanding of present and future impacts of climate extremes on the terrestrial carbon budget.},
  language  = {en}
}