@article{WickKruegerHohmann2005,
  author    = {Wick, Ditmar and Kr{\"u}ger, Tom and Hohmann, Andreas},
  title     = {Komplexe Bewegungsanalysen zum R{\"u}ckenstart bei nationalen Spitzenschwimmern},
  year      = {2005},
  abstract  = {Beobachtungen auf nationalem und internationalem Niveau haben ergeben, dass sich die Starttechnik in den letzten Jahren ver{\"a}ndert hat. So zeigen Analysen zum Zusammenhang von Block-, Flug- und Teilzeiten {\"u}ber 7,5; 10 und 15m und den erzielten Gesamtzeiten im 100-m-R{\"u}cken-schwimmen, dass k{\"u}rzere Startzeiten mit besseren Schwimmleistungen einher gehen (Cossor \& Mason, 2001). Insbesondere die Dauer der Flugzeit h{\"a}ngt negativ mit der Gesamtschwimmzeit zusammen, so dass die Flugzeit und -weite optimiert werden sollte.},
  language  = {de}
}
@article{HohmannFehrKirstenetal.2008,
  author    = {Hohmann, Andreas and Fehr, Ulrich and Kirsten, Robert and Kr{\"u}ger, Tom},
  title     = {Biomechanical analysis of the backstroke start technique in swimming},
  issn      = {1612-5770},
  year      = {2008},
  language  = {en}
}
@article{WickKruegerHohmann2003,
  author    = {Wick, Ditmar and Kr{\"u}ger, Tom and Hohmann, Andreas},
  title     = {Biomechanische Prinzipien als Kriterium der Effektivit{\"a}t von Grab- und Trackstart im Schwimmen},
  year      = {2003},
  abstract  = {Beobachtungen bei nationalen und internationalen Schwimmwettk{\"a}mpfen haben ergeben, dass sich die Starttechnik in den vergangenen Jahren zun{\"a}chst vom ehemals dominierenden Grabstart zum Trackstart ver{\"a}ndert hatte. Bei den Europameisterschaften 2002 in Berlin setzte jedoch eine Trendumkehr ein. An sieben Hochleistungsschwimmern wird untersucht, ob das biomechanische Prinzip der optimalen Tendenz im Beschleunigungsverlauf bei der Ausf{\"u}hrung von Grab- und Trackstarts optimal genutzt wird.},
  language  = {de}
}
@article{HolzBoeckerSchlierBaumeisteretal.2014,
  author    = {Holz, Nathalie E. and Boecker-Schlier, Regina and Baumeister, Sarah and Hohm, Erika and Zohsel, Katrin and Buchmann, Arlette F. and Blomeyer, Dorothea and Jennen-Steinmetz, Christine and Hohmann, Sarah and Wolf, Isabella and Plichta, Michael M. and Meyer-Lindenberg, Andreas and Banaschewski, Tobias and Brandeis, Daniel and Laucht, Manfred},
  title     = {Effect of prenatal exposure to tobacco smoke on inhibitory control neuroimaging results from a 25-Year prospective study},
  series = {JAMA psychiatry},
  volume    = {71},
  journal   = {JAMA psychiatry},
  number    = {7},
  publisher = {American Veterinary Medical Association},
  address   = {Chicago},
  issn      = {2168-622X},
  doi       = {10.1001/jamapsychiatry.2014.786},
  pages     = {786 -- 796},
  year      = {2014},
  abstract  = {IMPORTANCE: There is accumulating evidence relating maternal smoking during pregnancy to attention-deficit/hyperactivity disorder (ADHD) without elucidating specific mechanisms. Research investigating the neurobiological underpinnings of this disorder has implicated deficits during response inhibition. Attempts to uncover the effect of prenatal exposure to nicotine on inhibitory control may thus be of high clinical importance. MAIN OUTCOMES AND MEASURES: Functional magnetic resonance imaging response, morphometric data, lifetime ADHD symptoms, and novelty seeking. RESULTS: Participants prenatally exposed to nicotine exhibited a weaker response in the anterior cingulate cortex (t(168) = 4.46; peak Montreal Neurological Institute [MNI] coordinates x = -2, y = 20, z = 30; familywise error [FWE]-corrected P = .003), the right inferior frontal gyrus (t(168) = 3.65; peak MNI coordinates x = 44, y = 38, z = 12; FWE-corrected P = .04), the left inferior frontal gyrus (t(168) = 4.09; peak MNI coordinates x = -38, y = 36, z = 8; FWE-corrected P = .009), and the supramarginal gyrus (t(168) = 5.03; peak MNI coordinates x = 64, y = -28, z = 22; FWE-corrected P = .02) during the processing of the NoGo compared to neutral stimuli, while presenting a decreased volume in the right inferior frontal gyrus. These findings were obtained irrespective of the adjustment of confounders, ADHD symptoms, and novelty seeking. There was an inverse relationship between inferior frontal gyrus activity and ADHD symptoms and between anterior cingulate cortex activity and novelty seeking. CONCLUSIONS AND RELEVANCE: These findings point to a functional involvement of prenatal exposure to tobacco smoke in neural alterations similar to ADHD, which underlines the importance of smoking prevention treatments.},
  language  = {en}
}
@article{HolzBoeckerSchlierBuchmannetal.2016,
  author    = {Holz, Nathalie and Boecker-Schlier, Regina and Buchmann, Arlette F. and Blomeyer, Dorothea and Baumeister, Sarah and Hohmann, Sarah and Jennen-Steinmetz, Christine and Wolf, Isabella and Rietschel, Marcella and Witt, Stephanie H. and Plichta, Michael M. and Meyer-Lindenberg, Andreas and Schmidt, Martin H. and Esser, G{\"u}nter and Banaschewski, Tobias and Brandeis, Daniel and Laucht, Manfred},
  title     = {Evidence for a Sex-Dependent MAOAx Childhood Stress Interaction in the Neural Circuitry of Aggression},
  series = {Cerebral cortex},
  volume    = {26},
  journal   = {Cerebral cortex},
  publisher = {Oxford Univ. Press},
  address   = {Cary},
  issn      = {1047-3211},
  doi       = {10.1093/cercor/bhu249},
  pages     = {904 -- 914},
  year      = {2016},
  abstract  = {Converging evidence emphasizes the role of an interaction between monoamine oxidase A (MAOA) genotype, environmental adversity, and sex in the pathophysiology of aggression. The present study aimed to clarify the impact of this interaction on neural activity in aggression-related brain systems. Functional magnetic resonance imaging was performed in 125 healthy adults from a high-risk community sample followed since birth. DNA was genotyped for the MAOA-VNTR (variable number of tandem repeats). Exposure to childhood life stress (CLS) between the ages of 4 and 11 years was assessed using a standardized parent interview, aggression by the Youth/Young Adult Self-Report between the ages of 15 and 25 years, and the VIRA-R (Vragenlijst Instrumentele En Reactieve Agressie) at the age of 15 years. Significant interactions were obtained between MAOA genotype, CLS, and sex relating to amygdala, hippocampus, and anterior cingulate cortex (ACC) response, respectively. Activity in the amygdala and hippocampus during emotional face-matching increased with the level of CLS in male MAOA-L, while decreasing in male MAOA-H, with the reverse pattern present in females. Findings in the opposite direction in the ACC during a flanker NoGo task suggested that increased emotional activity coincided with decreased inhibitory control. Moreover, increasing amygdala activity was associated with higher Y(A)SR aggression in male MAOA-L and female MAOA-H carriers. Likewise, a significant association between amygdala activity and reactive aggression was detected in female MAOA-H carriers. The results point to a moderating role of sex in the MAOAx CLS interaction for intermediate phenotypes of emotional and inhibitory processing, suggesting a possible mechanism in conferring susceptibility to violence-related disorders.},
  language  = {en}
}
@article{HohmannZohselBuchmannetal.2016,
  author    = {Hohmann, Sarah and Zohsel, Katrin and Buchmann, Arlette F. and Blomeyer, Dorothea and Holz, Nathalie and Boecker-Schlier, Regina and Jennen-Steinmetz, Christine and Rietschel, Marcella and Witt, Stephanie H. and Schmidt, Martin H. and Esser, G{\"u}nter and Meyer-Lindenberg, Andreas and Banaschewski, Tobias and Brandeis, Daniel and Hohm, Erika and Laucht, Manfred},
  title     = {Interacting effect of MAOA genotype and maternal prenatal smoking on aggressive behavior in young adulthood},
  series = {Journal of neural transmission},
  volume    = {123},
  journal   = {Journal of neural transmission},
  publisher = {Springer},
  address   = {Wien},
  issn      = {0300-9564},
  doi       = {10.1007/s00702-016-1582-x},
  pages     = {885 -- 894},
  year      = {2016},
  abstract  = {Findings on the etiology of aggressive behavior have provided evidence for an effect both of genetic factors, such as variation in the monoamine oxidase A (MAOA) gene, and adverse environmental factors. Recent studies have supported the existence of gene × environment interactions, with early experiences playing a key role. In the present study, the effects of prenatal nicotine exposure, MAOA genotype and their interaction on aggressive behavior during young adulthood were examined. In a sample of 272 young adults (129 males, 143 females) from an epidemiological cohort study, smoking during pregnancy was measured with a standardized parent interview at the offspring's age of 3 months. Aggressive behavior was assessed between the ages of 19 and 25 years using the Young Adult Self-Report. DNA was genotyped for the MAOA 5\&\#8242; untranslated region variable number of tandem repeats polymorphism (VNTR). Results revealed a significant interaction between MAOA and smoking during pregnancy, indicating higher levels of aggressive behavior in young adults carrying the MAOA low-expressing genotype who had experienced prenatal nicotine exposure (n = 8, p = .025). In contrast, in carriers of the MAOA high-expressing genotype, maternal smoking during pregnancy had no effect on aggressive behavior during young adulthood (n = 20, p = .145). This study extends earlier findings demonstrating an interaction between MAOA genotype and prenatal nicotine exposure on aggressive behavior into young adulthood. The results point to the long-term adverse effects of smoking during pregnancy on the offspring's mental health, possibly underlining the importance of smoking cessation during pregnancy. According to the nature of the study (particularly sample size and power), analyses are exploratory and results need to be interpreted cautiously.},
  language  = {en}
}
@article{HolzBoeckerSchlierHohmetal.2015,
  author    = {Holz, Nathalie E. and Boecker-Schlier, Regina and Hohm, Erika and Zohsel, Katrin and Buchmann, Arlette F. and Blomeyer, Dorothea and Jennen-Steinmetz, Christine and Baumeister, Sarah and Hohmann, Sarah and Wolf, Isabella and Plichta, Michael M. and Esser, G{\"u}nter and Schmidt, Martin and Meyer-Lindenberg, Andreas and Banaschewski, Tobias and Brandeis, Daniel and Laucht, Manfred},
  title     = {The Long-Term Impact of Early Life Poverty on Orbitofrontal Cortex Volume in Adulthood: Results from a Prospective Study Over 25 Years},
  series = {Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology},
  volume    = {40},
  journal   = {Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology},
  number    = {4},
  publisher = {Nature Publ. Group},
  address   = {London},
  issn      = {0893-133X},
  doi       = {10.1038/npp.2014.277},
  pages     = {996 -- 1004},
  year      = {2015},
  abstract  = {Converging evidence has highlighted the association between poverty and conduct disorder (CD) without specifying neurobiological pathways. Neuroimaging research has emphasized structural and functional alterations in the orbitofrontal cortex (OFC) as one key mechanism underlying this disorder. The present study aimed to clarify the long-term influence of early poverty on OFC volume and its association with CD symptoms in healthy participants of an epidemiological cohort study followed since birth. At age 25 years, voxel-based morphometry was applied to study brain volume differences. Poverty (0 = non-exposed (N = 134), I = exposed (N = 33)) and smoking during pregnancy were determined using a standardized parent interview, and information on maternal responsiveness was derived from videotaped mother infant interactions at the age of 3 months. CD symptoms were assessed by diagnostic interview from 8 to 19 years of age. Information on life stress was acquired at each assessment and childhood maltreatment was measured using retrospective self-report at the age of 23 years. Analyses were adjusted for sex, parental psychopathology and delinquency, obstetric adversity, parental education, and current poverty. Individuals exposed to early life poverty exhibited a lower OFC volume. Moreover, we replicated previous findings of increased CD symptoms as a consequence of childhood poverty. This effect proved statistically mediated by OFC volume and exposure to life stress and smoking during pregnancy, but not by childhood maltreatment and maternal responsiveness. These findings underline the importance of studying the impact of early life adversity on brain alterations and highlight the need for programs to decrease income-related disparities.},
  language  = {en}
}
@article{Hohmann2000,
  author    = {Hohmann, Andreas},
  title     = {Aktuelle Aspekte der Leistungsdiagnostik im Sportspiel},
  isbn      = {3-88020-364-4},
  year      = {2000},
  language  = {de}
}
@article{SeidelHohmannDiercksetal.2000,
  author    = {Seidel, Ilka and Hohmann, Andreas and Diercks, B. and Daum, M. and L{\"u}hnenschloß, D.},
  title     = {Die individuelle Handballeistung im Nachwuchsbereich : Pfadanalysen zum Einfluss grundlegender Leistungsvoraussetzungen},
  isbn      = {3-88020-364-4},
  year      = {2000},
  language  = {de}
}
@article{Hohmann2000,
  author    = {Hohmann, Andreas},
  title     = {DEL-Analyse : eine Methode zur Trainingswirkungsanalyse},
  year      = {2000},
  language  = {de}
}
@article{SeidelHohmann1999,
  author    = {Seidel, Ilka and Hohmann, Andreas},
  title     = {Ein Forschungsprojekt zum sportlichen Talent},
  isbn      = {3- 88020-343-1},
  year      = {1999},
  language  = {de}
}
@article{Hohmann1999,
  author    = {Hohmann, Andreas},
  title     = {Anwendungs- und Grundlagenorientierung in der Trainings- und Bewegungsforschung},
  isbn      = {3- 88020-343-1},
  year      = {1999},
  language  = {de}
}
@book{HohmannLamesLetzelter1999,
  author    = {Hohmann, Andreas and Lames, Martin and Letzelter, Manfred},
  title     = {Einf{\"u}hrung in die Trainingswissenschaft},
  volume    = {2099},
  publisher = {Limpert},
  address   = {Wiebelsheim},
  isbn      = {3-8252-2099-0},
  pages     = {250 S.},
  year      = {1999},
  language  = {de}
}
@article{Hohmann1999,
  author    = {Hohmann, Andreas},
  title     = {The influence of strength, speed, motor coordination and technique on the performance in crawl sprint},
  isbn      = {951-39-0607-8},
  year      = {1999},
  language  = {en}
}
@article{HohmannHamacher1999,
  author    = {Hohmann, Andreas and Hamacher, D.},
  title     = {Isokinetisches Krafttraining mit Knie-Total-Endoprothese(TEP)-Patienten in der Anschlussheilbehandlung},
  isbn      = {3-88020-341-5},
  year      = {1999},
  language  = {de}
}
@article{HohmannDiercksLuehnenschlossetal.1999,
  author    = {Hohmann, Andreas and Diercks, B. and L{\"u}hnenschloß, D. and Seidel, Ilka and Wichmann, E.},
  title     = {Criteria of talent in sport},
  year      = {1999},
  language  = {en}
}
@article{HohmannDiercksLuehnenschlossetal.1999,
  author    = {Hohmann, Andreas and Diercks, B. and L{\"u}hnenschloß, D. and Seidel, Ilka and Griebsch, A.},
  title     = {The structure of physical abilities in sprint running},
  year      = {1999},
  language  = {en}
}
@article{Hohmann1999,
  author    = {Hohmann, Andreas},
  title     = {Speed abilities in swimming : diagnosis of some components influencing the performance in crawl sprint},
  year      = {1999},
  language  = {en}
}
@article{Hohmann1999,
  author    = {Hohmann, Andreas},
  title     = {Feldforschung in der Trainingswissenschaft},
  isbn      = {3-89001-323-6},
  year      = {1999},
  language  = {de}
}
@article{Hohmann1999,
  author    = {Hohmann, Andreas},
  title     = {Trainingswissenschaft},
  isbn      = {3-89676-183-8},
  year      = {1999},
  language  = {de}
}