@article{Tzoref2018,
  author    = {Tzoref, Shani},
  title     = {Knowing the Heart of the Stranger},
  series = {Interpretation : a journal of Bible and theology},
  volume    = {72},
  journal   = {Interpretation : a journal of Bible and theology},
  number    = {2},
  publisher = {Sage Publ.},
  address   = {Thousand Oaks},
  issn      = {0020-9643},
  doi       = {10.1177/0020964317749540},
  pages     = {119 -- 131},
  year      = {2018},
  abstract  = {With its exhortation "You shall also love the stranger (gēr), for you were strangers (gēr{\^i}m) in the land of Egypt" (Deut 10:19), the book of Deuteronomy helps cultivate a healthy and appreciative sense of past hardship, current prosperity, progress, and relative privilege. In contemporary culture, where the term "privilege" has become an unfortunate source of contention, Deuteronomy might point a way for recognition of one's relative privilege in regard to an Other as a basis for gratitude and responsibility. This essay argues that we have gained "privilege" after having been immigrants and strangers in a strange land. Privilege could become an empowering and challenging exercise of counting one's blessings and considering how these could be used for the benefit of others, including strangers in our land.},
  language  = {en}
}
@article{MorrisKassamBerczetal.2019,
  author    = {Morris, Mackenzie C. and Kassam, Farzaan and Bercz, Aron and Beckmann, Nadine and Schumacher, Fabian and Gulbins, Erich and Makley, Amy T. and Goodman, Michael D.},
  title     = {The Role of Chemoprophylactic Agents in Modulating Platelet Aggregability After Traumatic Brain Injury},
  series = {Journal of surgical research},
  volume    = {244},
  journal   = {Journal of surgical research},
  publisher = {Elsevier},
  address   = {San Diego},
  issn      = {0022-4804},
  doi       = {10.1016/j.jss.2019.06.022},
  pages     = {1 -- 8},
  year      = {2019},
  abstract  = {Background: The pathophysiology behind the subacute but persistent hypercoagulable state after traumatic brain injury (TBI) is poorly understood but contributes to morbidity induced by venous thromboembolism. Because platelets and their microvesicles have been hypothesized to play a role in post-traumatic hypercoagulability, administration of commonly used agents may ameliorate this coagulability. We hypothesized that utilization of aspirin, ketorolac, amitriptyline, unfractionated heparin, or enoxaparin would modulate the platelet aggregation response after TBI. Methods: Concussive TBI was induced by weight drop. Mice were then randomized to receive aspirin, ketorolac, amitriptyline, heparin, enoxaparin, or saline control at 2 and 8 h after TBI. Mice were sacrificed at 6 or 24 h after injury to determine coagulability by rotational thromboelastometry (ROTEM), platelet function testing with impedance aggregometry, and microvesicle enumeration. Platelet sphingolipid metabolites were analyzed by mass spectrometry. Results: ROTEM demonstrated increased platelet contribution to maximum clot firmness at 6 h after TBI in mice that received aspirin or amitriptyline, but this did not persist at 24 h. By contrast, adenosine diphosphate- and arachidonic acid-induced platelet aggregation at 6 h was significantly lower in mice receiving ketorolac, aspirin, and amitriptyline compared with mice receiving saline at 6 h after injury and only arachidonic acid-initiated platelet aggregation was decreased by aspirin at 24 h. There were no differences in microvesicle production at either time point. Platelet sphingosine-1-phosphate levels were decreased at 6 h in the group receiving amitriptyline and increased at 24 h along with platelet ceramide levels at 24 h in the amitriptyline group. Conclusion: After TBI, amitriptyline decreased platelet aggregability and increased contribution to clot in a manner similar to aspirin. The amitriptyline effects on platelet function and sphingolipid metabolites may represent a possible role of the acid sphingomyelinase in the hypercoagulability observed after injury. In addition, inhibition of platelet reactivity may be an underappreciated benefit of low molecular weight heparins, such as enoxaparin. (C) 2019 Elsevier Inc. All rights reserved.},
  language  = {en}
}