@book{IhleGroenWalteretal.2012,
  author    = {Ihle, Wolfgang and Groen, Gunter and Walter, Daniel and Esser, G{\"u}nter and Petermann, Franz},
  title     = {Depression},
  series = {Leitfaden Kinder- und Jugendpsychotherapie},
  volume    = {16},
  journal   = {Leitfaden Kinder- und Jugendpsychotherapie},
  publisher = {Hogrefe},
  address   = {G{\"o}ttingen},
  isbn      = {978-3-8017-2381-1},
  pages     = {IX, 162 S.},
  year      = {2012},
  language  = {de}
}
@article{BuchmannSchmidBlomeyeretal.2009,
  author    = {Buchmann, Arlette F. and Schmid, Brigitte and Blomeyer, Dorothea and Becker, Katja and Treutlein, Jens and Zimmermann, Ulrich S. and Jennen-Steinmetz, Christine and Schmidt, Martin H. and Esser, G{\"u}nter and Banaschewski, Tobias and Rietschel, Marcella and Schumann, Gunter and Laucht, Manfred},
  title     = {Impact of age at first drink on vulnerability to alcohol-related problems : testing the marker hypothesis in a prospective study of young adults},
  issn      = {0022-3956},
  doi       = {10.1016/j.jpsychires.2009.02.006},
  year      = {2009},
  abstract  = {There is ample evidence that the early initiation of alcohol use is a risk factor for the development of later alcohol-related problems. The purpose of the current study was to examine whether this association can be explained by indicators of a common underlying susceptibility or whether age at drinking onset may be considered as an independent predictor of later drinking behavior, suggesting a potential causal relationship. Participants were drawn from a prospective cohort study of the long-term outcomes of early risk factors followed up from birth onwards. Structured interviews were administered to 304 participants to assess age at first drink and current drinking behavior. Data on risk factors, including early family adversity, parental alcohol use, childhood psychopathology and stressful life events, were repeatedly collected during childhood using standardized parent interviews. In addition, information on genotype was considered. Results confirmed previous work demonstrating that hazardous alcohol consumption is related to early-adolescent drinking onset. A younger age of first drink was significantly predicted by 5-HTTLPR genotype and the degree of preceding externalizing symptoms, and both factors were related to increased consumption or harmful alcohol use at age 19. However, even after controlling for these potential explanatory factors, earlier age at drinking onset remained a strong predictor of heavy alcohol consumption in young adulthood. The present longitudinal study adds to the current literature indicating that the early onset - adult hazardous drinking association cannot solely be attributed to shared genetic and psychopathologic risk factors as examined in this study.},
  language  = {en}
}
@article{BlomeyerTreutleinEsseretal.2007,
  author    = {Blomeyer, Dorothea and Treutlein, Jens and Esser, G{\"u}nter and Schmidt, Martin H. and Schumann, Gunter and Laucht, Manfred},
  title     = {Interaction between CRHR1 gene and stressful life events predicts adolescent heavy alcohol use},
  issn      = {0006-3223},
  year      = {2007},
  abstract  = {Background: Recent animal research suggests that alterations in the corticotropin releasing hormone receptor 1 (CRHR1) may lead to heavy alcohol use following repeated stress. The aim of this study was to examine interactions between two haplotype-tagging single nucleotide polymorphisms (SNPs) covering the CRHR1 gene and adverse life events on heavy drinking in adolescents. Methods: Data were available from the Mannheim Study of Children at Risk, an ongoing cohort study of the long-term outcome of early risk factors followed since birth. At age 15 years, 280 participants (135 males, 145 females) completed a self-report questionnaire measuring alcohol use and were genotyped for two SNPs (rs242938, rs1876831) of CRHR1. Assessment of negative life events over the past three years was obtained by a standardized interview with the parents. Results: Adolescents homozygous for the C allele of rs1876831 drank higher maximum amounts of alcohol per occasion and had greater lifetime rates of heavy drinking in relation to negative life events than individuals carrying the T allele. No gene X environment interactions were found for regular drinking and between rs242938 and stressful life events. Conclusions: These findings provide first evidence in humans that the CRHR1 gene interacts with exposure to stressful life events to predict heavy alcohol use in adolescents.},
  language  = {en}
}
@article{HolzBoeckerSchlierBuchmannetal.2017,
  author    = {Holz, Nathalie E. and Boecker-Schlier, Regina and Buchmann, Arlette F. and Blomeyer, Dorothea and Jennen-Steinmetz, Christine and Baumeister, Sarah and Plichta, Michael M. and Cattrell, Anna and Schumann, Gunter and Esser, G{\"u}nter and Schmidt, Martin and Buitelaar, Jan and Meyer-Lindenberg, Andreas and Banaschewski, Tobias and Brandeis, Daniel and Laucht, Manfred},
  title     = {Ventral striatum and amygdala activity as convergence sites for early adversity and conduct disorder},
  series = {Frontiers in human neuroscience},
  volume    = {12},
  journal   = {Frontiers in human neuroscience},
  number    = {2},
  publisher = {Oxford Univ. Press},
  address   = {Oxford},
  issn      = {1749-5016},
  doi       = {10.1093/scan/nsw120},
  pages     = {261 -- 272},
  year      = {2017},
  abstract  = {Childhood family adversity (CFA) increases the risk for conduct disorder (CD) and has been associated with alterations in regions of affective processing like ventral striatum (VS) and amygdala. However, no study so far has demonstrated neural converging effects of CFA and CD in the same sample. At age 25 years, functional MRI data during two affective tasks, i.e. a reward (N = 171) and a face-matching paradigm (N = 181) and anatomical scans (N = 181) were acquired in right-handed currently healthy participants of an epidemiological study followed since birth. CFA during childhood was determined using a standardized parent interview. Disruptive behaviors and CD diagnoses during childhood and adolescence were obtained by diagnostic interview (2-19 years), temperamental reward dependence was assessed by questionnaire (15 and 19 years). CFA predicted increased CD and amygdala volume. Both exposure to CFA and CD were associated with a decreased VS response during reward anticipation and blunted amygdala activity during face-matching. CD mediated the effect of CFA on brain activity. Temperamental reward dependence was negatively correlated with CFA and CD and positively with VS activity. These findings underline the detrimental effects of CFA on the offspring's affective processing and support the importance of early postnatal intervention programs aiming to reduce childhood adversity factors.},
  language  = {en}
}
@article{BlomeyerBuchmannLascorzetal.2013,
  author    = {Blomeyer, Dorothea and Buchmann, Arlette F. and Lascorz, Jesus and Zimmermann, Ulrich S. and Esser, G{\"u}nter and Desrivieres, Sylvane and Schmidt, Martin H. and Banaschewski, Tobias and Schumann, Gunter and Laucht, Manfred},
  title     = {Association of PER2 genotype and stressful life events with alcohol drinking in young adults},
  series = {PLoS one},
  volume    = {8},
  journal   = {PLoS one},
  number    = {3},
  publisher = {PLoS},
  address   = {San Fransisco},
  issn      = {1932-6203},
  doi       = {10.1371/journal.pone.0059136},
  pages     = {7},
  year      = {2013},
  abstract  = {Background: Clock genes govern circadian rhythms and shape the effect of alcohol use on the physiological system. Exposure to severe negative life events is related to both heavy drinking and disturbed circadian rhythmicity. The aim of this study was 1) to extend previous findings suggesting an association of a haplotype tagging single nucleotide polymorphism of PER2 gene with drinking patterns, and 2) to examine a possible role for an interaction of this gene with life stress in hazardous drinking. Methods: Data were collected as part of an epidemiological cohort study on the outcome of early risk factors followed since birth. At age 19 years, 268 young adults (126 males, 142 females) were genotyped for PER2 rs56013859 and were administered a 45-day alcohol timeline follow-back interview and the Alcohol Use Disorders Identification Test (AUDIT). Life stress was assessed as the number of severe negative life events during the past four years reported in a questionnaire and validated by interview. Results: Individuals with the minor G allele of rs56013859 were found to be less engaged in alcohol use, drinking at only 72\% of the days compared to homozygotes for the major A allele. Moreover, among regular drinkers, a gene x environment interaction emerged (p = .020). While no effects of genotype appeared under conditions of low stress, carriers of the G allele exhibited less hazardous drinking than those homozygous for the A allele when exposed to high stress. Conclusions: These findings may suggest a role of the circadian rhythm gene PER2 in both the drinking patterns of young adults and in moderating the impact of severe life stress on hazardous drinking in experienced alcohol users. However, in light of the likely burden of multiple tests, the nature of the measures used and the nominal evidence of interaction, replication is needed before drawing firm conclusions.},
  language  = {en}
}
@article{LauchtBeckerFranketal.2008,
  author    = {Laucht, Manfred and Becker, Katja and Frank, Josef and Schmidt, Martin H. and Esser, G{\"u}nter and Treutlein, Jens and Skowronek, Markus H. and Schumann, Gunter},
  title     = {Genetic variation in dopamine pathways differentially associated with smoking progression in adolescence},
  issn      = {0890-8567},
  doi       = {10.1097/Chi.0b013e31816bff77},
  year      = {2008},
  abstract  = {Objective: To clarify the nature of the association between dopamine genes and smoking by examining whether genetic variability in components of the dopamine pathway could explain refined phenotypes in adolescent smoking progression. Method: Data are from an ongoing prospective study of the long-term outcome of early risk factors studied since birth. At age 15 years, 220 participants (108 males, 112 females) completed a self-report questionnaire measuring smoking behavior and were genotyped for five dopamine gene variants. Results: Smoking initiation was related to allelic variation in the dopamine D-4 receptor gene (DRD4), whereas smoking continuation and dependence showed association with the dopamine D-2 receptor gene (DRD2). Adolescents with the seven-repeat allele of the common DRD4 exon 3 polymorphism had rates of ever smoking that were significantly higher than in those with other genotypes. Once smoking started, carriers of the T allele of a single nucleotide polymorphism of DRD2 (rs4648317) reported higher rates of current smoking and scored higher on nicotine dependence than their allelic counterparts. Among current smokers, intention to quit was significantly lower in adolescents homozygous for the 10-repeat allele of the common dopamine transporter 3 untranslated region polymorphism. Conclusions: Our results provide preliminary evidence of genetic influences on different stages of smoking and suggest the importance of specific dopamine genes in smoking progression in adolescence.},
  language  = {en}
}
@article{MeiserEsser2017,
  author    = {Meiser, Susanne and Esser, G{\"u}nter},
  title     = {Interpersonal Stress Generation-A Girl Problem?},
  series = {Journal of early adolescence},
  volume    = {39},
  journal   = {Journal of early adolescence},
  number    = {1},
  publisher = {Sage Publ.},
  address   = {Thousand Oaks},
  issn      = {0272-4316},
  doi       = {10.1177/0272431617725197},
  pages     = {41 -- 66},
  year      = {2017},
  abstract  = {To provide further insight into stress generation patterns in boys and girls around puberty, this study investigated longitudinal reciprocal relations between depressive symptoms, dysfunctional attitudes, and stress generation, the process by which individuals contribute to the occurrence of stress in interpersonal contexts (e.g., problematic social interactions) or in noninterpersonal contexts (e.g., achievement problems). A community sample of N = 924 German children and early adolescents (51.8\% male) completed depressive symptoms and dysfunctional attitudes measures at T1 and again 20 months later (T2). Stressful life events were reported at T2. Dysfunctional attitudes were unrelated to stress generation. Interpersonal, but not noninterpersonal, dependent stress partially mediated the relationship between initial and later depressive symptoms, with girls being more likely to generate interpersonal stress in response to depressive symptoms. Findings underscore the role of interpersonal stress generation in the early development of depressive symptomatology, and in the gender difference in depression prevalence emerging around puberty.},
  language  = {en}
}
@article{HolzBoeckerSchlierJennenSteinmetzetal.2018,
  author    = {Holz, Nathalie E. and Boecker-Schlier, Regina and Jennen-Steinmetz, Christine and Hohm, Erika and Buchmann, Arlette F. and Blomeyer, Dorothea and Baumeister, Sarah and Plichta, Michael M. and Esser, G{\"u}nter and Schmidt, Martin and Meyer-Lindenberg, Andreas and Banaschewski, Tobias and Brandeis, Daniel and Laucht, Manfred},
  title     = {Early maternal care may counteract familial liability for psychopathology in the reward circuitry},
  series = {Social Cognitive and Affective Neuroscience},
  volume    = {13},
  journal   = {Social Cognitive and Affective Neuroscience},
  number    = {11},
  publisher = {Oxford Univ. Press},
  address   = {Oxford},
  issn      = {1749-5016},
  doi       = {10.1093/scan/nsy087},
  pages     = {1191 -- 1201},
  year      = {2018},
  abstract  = {Reward processing is altered in various psychopathologies and has been shown to be susceptible to genetic and environmental influences. Here, we examined whether maternal care may buffer familial risk for psychiatric disorders in terms of reward processing. Functional magnetic resonance imaging during a monetary incentive delay task was acquired in participants of an epidemiological cohort study followed since birth (N = 172, 25 years). Early maternal stimulation was assessed during a standardized nursing/playing setting at the age of 3 months. Parental psychiatric disorders (familial risk) during childhood and the participants' previous psychopathology were assessed by diagnostic interview. With high familial risk, higher maternal stimulation was related to increasing activation in the caudate head, the supplementary motor area, the cingulum and the middle frontal gyrus during reward anticipation, with the opposite pattern found in individuals with no familial risk. In contrast, higher maternal stimulation was associated with decreasing caudate head activity during reward delivery and reduced levels of attention deficit hyperactivity disorder (ADHD) in the high-risk group. Decreased caudate head activity during reward anticipation and increased activity during delivery were linked to ADHD. These findings provide evidence of a long-term association of early maternal stimulation on both adult neurobiological systems of reward underlying externalizing behavior and ADHD during development.},
  language  = {en}
}
@article{BoeckerSchlierHolzHohmetal.2017,
  author    = {Boecker-Schlier, Regina and Holz, Nathalie E. and Hohm, Erika and Zohsel, Katrin and Blomeyer, Dorothea and Buchmann, Arlette F. and Baumeister, Sarah and Wolf, Isabella and Esser, G{\"u}nter and Schmidt, Martin H. and Meyer-Lindenberg, Andreas and Banaschewski, Tobias and Brandeis, Daniel and Laucht, Manfred},
  title     = {Association between pubertal stage at first drink and neural reward processing in early adulthood},
  series = {Addiction biology},
  volume    = {22},
  journal   = {Addiction biology},
  publisher = {Wiley},
  address   = {Hoboken},
  issn      = {1355-6215},
  doi       = {10.1111/adb.12413},
  pages     = {1402 -- 1415},
  year      = {2017},
  abstract  = {Puberty is a critical time period during human development. It is characterized by high levels of risk-taking behavior, such as increased alcohol consumption, and is accompanied by various neurobiological changes. Recent studies in animals and humans have revealed that the pubertal stage at first drink (PSFD) significantly impacts drinking behavior in adulthood. Moreover, neuronal alterations of the dopaminergic reward system have been associated with alcohol abuse or addiction. This study aimed to clarify the impact of PSFD on neuronal characteristics of reward processing linked to alcohol-related problems. One hundred sixty-eight healthy young adults from a prospective study covering 25 years participated in a monetary incentive delay task measured with simultaneous EEG-fMRI. PSFD was determined according to the age at menarche or Tanner stage of pubertal development, respectively. Alcohol-related problems in early adulthood were assessed with the Alcohol Use Disorder Identification Test (AUDIT). During reward anticipation, decreased fMRI activation of the frontal cortex and increased preparatory EEG activity (contingent negative variation) occurred with pubertal compared to postpubertal first alcohol intake. Moreover, alcohol-related problems during early adulthood were increased in pubertal compared to postpubertal beginners, which was mediated by neuronal activation of the right medial frontal gyrus. At reward delivery, increased fMRI activation of the left caudate and higher feedback-related EEG negativity were detected in pubertal compared to postpubertal beginners. Together with animal findings, these results implicate PSFD as a potential modulator of psychopathology, involving altered reward anticipation. Both PSFD timing and reward processing might thus be potential targets for early prevention and intervention.},
  language  = {en}
}
@article{KucianZuberKohnetal.2018,
  author    = {Kucian, Karin and Zuber, Isabelle and Kohn, Juliane and Poltz, Nadine and Wyschkon, Anne and Esser, G{\"u}nter and von Aster, Michael G.},
  title     = {Relation Between Mathematical Performance, Math Anxiety, and Affective Priming in Children With and Without Developmental Dyscalculia},
  series = {Frontiers in psychology},
  volume    = {9},
  journal   = {Frontiers in psychology},
  publisher = {Frontiers Research Foundation},
  address   = {Lausanne},
  issn      = {1664-1078},
  doi       = {10.3389/fpsyg.2018.00263},
  pages     = {13},
  year      = {2018},
  abstract  = {Many children show negative emotions related to mathematics and some even develop mathematics anxiety. The present study focused on the relation between negative emotions and arithmetical performance in children with and without developmental dyscalculia (DD) using an affective priming task. Previous findings suggested that arithmetic performance is influenced if an affective prime precedes the presentation of an arithmetic problem. In children with DD specifically, responses to arithmetic operations are supposed to be facilitated by both negative and mathematics-related primes (= negative math priming effect). We investigated mathematical performance, math anxiety, and the domain-general abilities of 172 primary school children (76 with DD and 96 controls). All participants also underwent an affective priming task which consisted of the decision whether a simple arithmetic operation (addition or subtraction) that was preceded by a prime (positive/negative/neutral or mathematics-related) was true or false. Our findings did not reveal a negative math priming effect in children with DD. Furthermore, when considering accuracy levels, gender, or math anxiety, the negative math priming effect could not be replicated. However, children with DD showed more math anxiety when explicitly assessed by a specific math anxiety interview and showed lower mathematical performance compared to controls. Moreover, math anxiety was equally present in boys and girls, even in the earliest stages of schooling, and interfered negatively with performance. In conclusion, mathematics is often associated with negative emotions that can be manifested in specific math anxiety, particularly in children with DD. Importantly, present findings suggest that in the assessed age group, it is more reliable to judge math anxiety and investigate its effects on mathematical performance explicitly by adequate questionnaires than by an affective math priming task.},
  language  = {en}
}
@article{HohmZohselSchmidtetal.2017,
  author    = {Hohm, Erika and Zohsel, Katrin and Schmidt, Martin H. and Esser, G{\"u}nter and Brandeis, Daniel and Banaschewski, Tobias and Laucht, Manfred},
  title     = {Beeintr{\"a}chtigter Start ins Leben},
  series = {Kindheit und Entwicklung},
  volume    = {26},
  journal   = {Kindheit und Entwicklung},
  publisher = {Hogrefe},
  address   = {G{\"o}ttingen},
  issn      = {0942-5403},
  doi       = {10.1026/0942-5403/a000234},
  pages     = {210 -- 220},
  year      = {2017},
  abstract  = {Postpartale Depressionen sind h{\"a}ufige und schwerwiegende psychische Erkrankungen mit ung{\"u}nstigem Einfluss auf die kindliche Entwicklung. Als Haupttransmissionsweg gilt die fr{\"u}he Mutter-Kind-Interaktion. {\"U}ber die langfristigen Auswirkungen auf die Kinder im Erwachsenenalter und die Rolle der Interaktion liegen kaum Ergebnisse vor. Im Rahmen der Mannheimer Risikokinderstudie wurden postpartale Depressionen bis zwei Jahre nach der Geburt erfasst. Die kindliche Entwicklung wurde fortlaufend und die Mutter-Kind-Interaktion im Alter von 3 Monaten standardisiert erhoben. 28 Kinder postpartal depressiver und 107 Kinder gesunder M{\"u}tter konnten mit 25 Jahren untersucht werden. Beeintr{\"a}chtigungen der kognitiven und psychischen Entwicklung bei Kindern postpartal depressiver M{\"u}tter waren bis ins Erwachsenenalter nachweisbar. Responsives bzw. sensitives m{\"u}tterliches Verhalten wirkte der negativen Entwicklung entgegen. Dies betont die Bedeutung einer hohen Qualit{\"a}t der Mutter-Kind-Interaktion f{\"u}r die Entwicklung von Risikokindern.},
  language  = {de}
}
@article{HohmLauchtZohseletal.2017,
  author    = {Hohm, Erika and Laucht, Manfred and Zohsel, Katrin and Schmidt, Martin H. and Esser, G{\"u}nter and Brandeis, Daniel and Banaschewski, Tobias},
  title     = {Resilienz und Ressourcen im Verlauf der Entwicklung},
  series = {Kindheit und Entwicklung},
  volume    = {26},
  journal   = {Kindheit und Entwicklung},
  publisher = {Hogrefe},
  address   = {G{\"o}ttingen},
  issn      = {0942-5403},
  doi       = {10.1026/0942-5403/a000236},
  pages     = {230 -- 239},
  year      = {2017},
  abstract  = {Anhand von Daten der Mannheimer Risikokinderstudie, die sich mit der langfristigen Entwicklung von Kindern mit unterschiedlichen Risikobelastungen besch{\"a}ftigt, wird gezeigt, wie Schutzfaktoren aufseiten des Kindes und seines famili{\"a}ren Umfelds im Verlauf der Entwicklung wirksam werden und zur Entstehung von Resilienz beitragen k{\"o}nnen. Eine besondere Rolle kommt dabei positiven fr{\"u}hen Eltern-Kind-Beziehungen zu (sowohl Mutter- als auch Vater-Kind-Interaktionen). Daneben spielen auch Interaktionserfahrungen im Alter von zwei Jahren des Kindes eine bedeutsame Rolle; diese sch{\"u}tzen Risikokinder davor, eine ung{\"u}nstige Entwicklung zu nehmen und tragen dazu bei, dass sich Kinder, die in psychosozialen Hochrisikofamilien aufwachsen, trotz ung{\"u}nstiger „Startbedingungen" positiv entwickeln. Neben Merkmalen der sozialen Umwelt nehmen auch sprachliche, sozial-emotionale und internale Kompetenzen des Kindes im Entwicklungsverlauf eine wichtige Rolle ein. Diese Kompetenzen erm{\"o}glichen es Risikokindern auch unter widrigen Lebensumst{\"a}nden (psychosoziale Hochrisikofamilien, Aufwachsen in Armutsverh{\"a}ltnissen) erfolgreich zu bestehen. Dar{\"u}ber hinaus zeigt die Arbeit, dass Resilienz ein Pers{\"o}nlichkeitsmerkmal ist, das ab dem fr{\"u}hen Erwachsenenalter eine hohe Stabilit{\"a}t besitzt. Mit diesen Befunden verweist die Arbeit auf die große Bedeutung der Resilienz bei der Vorhersage der langfristigen Entwicklung von Risikokindern.},
  language  = {de}
}
@misc{HohmZohselSchmidtetal.2017,
  author    = {Hohm, Erika and Zohsel, Katrin and Schmidt, Martin H. and Esser, G{\"u}nter and Brandeis, Daniel and Banaschewski, Tobias and Laucht, Manfred},
  title     = {Beeintr{\"a}chtigter Start ins Leben},
  series = {Postprints der Universit{\"a}t Potsdam : Humanwissenschaftliche Reihe},
  journal   = {Postprints der Universit{\"a}t Potsdam : Humanwissenschaftliche Reihe},
  number    = {692},
  issn      = {1866-8364},
  doi       = {10.25932/publishup-43340},
  url       = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-433406},
  pages     = {37},
  year      = {2017},
  abstract  = {Postpartale Depressionen sind h{\"a}ufige und schwerwiegende psychische Erkrankungen mit ung{\"u}nstigem Einfluss auf die kindliche Entwicklung. Als Haupttransmissionsweg gilt die fr{\"u}he Mutter-Kind-Interaktion. {\"U}ber die langfristigen Auswirkungen auf die Kinder im Erwachsenenalter und die Rolle der Interaktion liegen kaum Ergebnisse vor. Im Rahmen der Mannheimer Risikokinderstudie wurden postpartale Depressionen bis zwei Jahre nach der Geburt erfasst. Die kindliche Entwicklung wurde fortlaufend und die Mutter-Kind-Interaktion im Alter von 3 Monaten standardisiert erhoben. 28 Kinder postpartal depressiver und 107 Kinder gesunder M{\"u}tter konnten mit 25 Jahren untersucht werden. Beeintr{\"a}chtigungen der kognitiven und psychischen Entwicklung bei Kindern postpartal depressiver M{\"u}tter waren bis ins Erwachsenenalter nachweisbar. Responsives bzw. sensitives m{\"u}tterliches Verhalten wirkte der negativen Entwicklung entgegen. Dies betont die Bedeutung einer hohen Qualit{\"a}t der Mutter-Kind-Interaktion f{\"u}r die Entwicklung von Risikokindern.},
  language  = {de}
}
@misc{HohmLauchtZohseletal.2017,
  author    = {Hohm, Erika and Laucht, Manfred and Zohsel, Katrin and Schmidt, Martin H. and Esser, G{\"u}nter and Brandeis, Daniel and Banaschewski, Tobias},
  title     = {Resilienz und Ressourcen im Verlauf der Entwicklung},
  series = {Postprints der Universit{\"a}t Potsdam : Humanwissenschaftliche Reihe},
  journal   = {Postprints der Universit{\"a}t Potsdam : Humanwissenschaftliche Reihe},
  number    = {608},
  issn      = {1866-8364},
  doi       = {10.25932/publishup-43307},
  url       = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-433072},
  pages     = {230 -- 239},
  year      = {2017},
  abstract  = {Anhand von Daten der Mannheimer Risikokinderstudie, die sich mit der langfristigen Entwicklung von Kindern mit unterschiedlichen Risikobelastungen besch{\"a}ftigt, wird gezeigt, wie Schutzfaktoren aufseiten des Kindes und seines famili{\"a}ren Umfelds im Verlauf der Entwicklung wirksam werden und zur Entstehung von Resilienz beitragen k{\"o}nnen. Eine besondere Rolle kommt dabei positiven fr{\"u}hen Eltern-Kind-Beziehungen zu (sowohl Mutter- als auch Vater-Kind-Interaktionen). Daneben spielen auch Interaktionserfahrungen im Alter von zwei Jahren des Kindes eine bedeutsame Rolle; diese sch{\"u}tzen Risikokinder davor, eine ung{\"u}nstige Entwicklung zu nehmen und tragen dazu bei, dass sich Kinder, die in psychosozialen Hochrisikofamilien aufwachsen, trotz ung{\"u}nstiger „Startbedingungen" positiv entwickeln. Neben Merkmalen der sozialen Umwelt nehmen auch sprachliche, sozial-emotionale und internale Kompetenzen des Kindes im Entwicklungsverlauf eine wichtige Rolle ein. Diese Kompetenzen erm{\"o}glichen es Risikokindern auch unter widrigen Lebensumst{\"a}nden (psychosoziale Hochrisikofamilien, Aufwachsen in Armutsverh{\"a}ltnissen) erfolgreich zu bestehen. Dar{\"u}ber hinaus zeigt die Arbeit, dass Resilienz ein Pers{\"o}nlichkeitsmerkmal ist, das ab dem fr{\"u}hen Erwachsenenalter eine hohe Stabilit{\"a}t besitzt. Mit diesen Befunden verweist die Arbeit auf die große Bedeutung der Resilienz bei der Vorhersage der langfristigen Entwicklung von Risikokindern.},
  language  = {de}
}
@misc{Esser2020,
  author    = {Esser, G{\"u}nter},
  title     = {Rezension zu: Geissler, Julia; Vloet, Timo D.; Romanos, Marcel; Zwanzger, Ulrike; Jans, Thomas: Verhaltenstherapie bei ADHS im Jugendalter : ein modular aufgebautes Therapieprogramm. - G{\"o}ttingen: Hogrefe, 102 S. - (Therapeutische Praxis, Bd. 94). - ISBN: 978-3-8017-2979-0},
  series = {Zeitschrift f{\"u}r Klinische Psychologie und Psychotherapie},
  volume    = {49},
  journal   = {Zeitschrift f{\"u}r Klinische Psychologie und Psychotherapie},
  number    = {2},
  publisher = {Hogrefe},
  address   = {G{\"o}ttingen},
  issn      = {1616-3443},
  doi       = {10.1026/1616-3443/a000551},
  pages     = {138 -- 139},
  year      = {2020},
  language  = {de}
}
@article{VianaWackermannFurtadoEsseretal.2006,
  author    = {Viana-Wackermann, Paula C. and Furtado, Erikson F. and Esser, G{\"u}nter and Schmidt, Martin H. and Laucht, Manfred},
  title     = {Lower P300 amplitude in eight-year-old offspring of alcoholic fathers with a delinquent history},
  issn      = {0940-1334},
  doi       = {10.1007/s00406-006-0709-8},
  year      = {2006},
  abstract  = {The aim of the present study was to investigate the P300 amplitude as a possible vulnerability marker in children of alcoholic (COA) fathers with and without paternal delinquency. Event-related potentials (ERPs) of 122 children aged 8 years (63 boys, 59 girls) were compared depending on father's alcoholism subtype: 30 COAs without paternal delinquency, 10 COAs with paternal delinquency, and 82 children of non-alcoholic and non-delinquent fathers. ERPs were recorded from Fz, Cz, and Pz, using an auditory oddball paradigm. Sinus tones of 60 dB HL were presented binaurally at 1,000 Hz (standard stimulus) and 2,000 Hz (target stimulus), at a relative frequency ratio of 80:20. Two trial blocks of 250 stimuli each were collected. Results indicated that only COAs with paternal delinquency displayed significant differences from the control group, characterized by reduced P300 amplitude at frontal site and in the second trial block. Thus, the combination of fathers' alcoholism and delinquency was more likely to relate to attenuated P300 amplitude in the offspring than paternal alcoholism alone. Our results suggest that both alcoholic and delinquent family history appear to play a role in P300 amplitude reduction in the offspring.},
  language  = {en}
}
@article{EsserWyschkon2007,
  author    = {Esser, G{\"u}nter and Wyschkon, Anne},
  title     = {Bedeutung komorbider St{\"o}rung im Kindesalter f{\"u}r den Langzeitverlauf der ADHS},
  isbn      = {978-3-17-019081-8},
  year      = {2007},
  language  = {de}
}
@article{MaierLangeHoraceketal.2007,
  author    = {Maier, A. and Lange, Sabine and Horacek, U. and Weinrich, D. and Esser, G{\"u}nter},
  title     = {Psychische St{\"o}rungen und Entwicklungsauff{\"a}lligkeiten fr{\"u}her erkennen mit neuem Kita- Vorsorgebogen},
  year      = {2007},
  language  = {de}
}
@article{IhleLehmannEsser2008,
  author    = {Ihle, Wolfgang and Lehmann, Katrin and Esser, G{\"u}nter},
  title     = {Legale und illegale Substanzen im Jugendalter : Konsum, problematischer Gebrauch, risikoerh{\"o}hende und risikomindernde Faktoren bei 14- bis 15-J{\"a}hrigen},
  issn      = {0942-5403},
  doi       = {10.1026/0942-5403.17.1.5},
  year      = {2008},
  abstract  = {Use and misuse, age of first substance use, and prevalence of problematic use of legal (alcohol, nicotine) and illegal (cannabis etc.) drugs in adolescence were analyzed. The risk and protective factors for substance misuse were investigated. A representative sample of 246 14-15 year-olds were examined (20\% of the population of a defined German region, cross-sectional design, self-rating inventory). Prevalence rates for problematic use were 19.9\% for alcohol, 48\% for nicotine, and 16.7\% for illegal drugs. Mean age of first substance use was age 12 for legal drugs and age 14 for illegal drugs. 4,9\% of the adolescents showed concurrent problematic use of nicotine, alcohol, and illegal drugs (male- female ratio: 5,2:1). Problematic substance use could be predicted best by a combination of risk factors (school failure, low level for availability of illegal drugs in the neighbourhood) and protective factors (participating a lot of sport, positive family climate). Suggestions for universal, selective, and indicated preventive interventions were derived.},
  language  = {de}
}
@article{LauchtHohmEsseretal.2007,
  author    = {Laucht, Manfred and Hohm, E. and Esser, G{\"u}nter and Schmidt, Martin H. and Becker, Katja},
  title     = {Association between ADHD and smoking in adolescence : shared genetic, environmental and psychopathological factors},
  issn      = {0300-9564},
  doi       = {10.1007/s00702-007-0703-y},
  year      = {2007},
  abstract  = {The present study aimed to examine the extent to which the co-occurrence of ADHD and smoking in adolescents could be attributed to common genetic, environmental and psychopathological factors. Data are from an ongoing prospective study of the outcome of early risk factors. At age 15 years, 305 adolescents completed self-report questionnaires measuring tobacco consumption and deviant peer affiliations. Lifetime psychiatric diagnoses were obtained using standardized interviews. DNA was genotyped for the dopamine D4 receptor (DRD4) gene exon III polymorphism. Adolescents with a lifetime diagnosis of ADHD displayed significantly higher smoking activity than non-ADHD controls. A major component of this association could be accounted for by deviant peer affiliations and the comorbidity with oppositional-defiant and conduct disorder, while a minor part was attributable to DRD4 in males but not in females. These findings suggest that the association of ADHD with smoking relies on risk factors shared by the two behaviors.},
  language  = {en}
}
@article{EsserFreybergerHoffmannetal.2006,
  author    = {Esser, G{\"u}nter and Freyberger, H. J. and Hoffmann, S. O. and Hoyer, J. and Richter, R. and Harfst, T.},
  title     = {Anpassung der Psychotherapie-Richtlinien zur Zulassung psychotherapeutischer Verfahren und Methoden},
  year      = {2006},
  language  = {de}
}
@article{EsserLange2006,
  author    = {Esser, G{\"u}nter and Lange, Sabine},
  title     = {Unbeschriebene Entwicklungsst{\"o}rung des Lesens und Rechtschreibens},
  isbn      = {978- 3-406-54106-3},
  year      = {2006},
  language  = {de}
}
@article{WyschkonEsser2006,
  author    = {Wyschkon, Anne and Esser, G{\"u}nter},
  title     = {Klinische Kinder- und Jugendlichen-Psychologie : Forschungslinien und offene Fragen in den letzten vier Jahren},
  year      = {2006},
  language  = {de}
}
@article{LauchtHohmEsseretal.2005,
  author    = {Laucht, Manfred and Hohm, E. and Esser, G{\"u}nter and Schmidt, Martin H.},
  title     = {Elevated risk of smoking in children with externalizing disorders},
  issn      = {1616-3443},
  year      = {2005},
  abstract  = {Background: Several studies have reported higher smoking rates among adolescents with externalizing disorders (attention-deficit hyperactivity disorder and conduct disorder) as compared to healthy controls. Objective: To follow the association between childhood externalizing disorders and smoking during development, to determine the type of problems most strongly related to later tobacco use, and to control for the influence of covarying factors. Methods: Participants were from a longitudinal study of a birth cohort of 384 children born with different perinatal and psychosocial risks. Standardized assessments of behavioral disorders between 2 and 11 years and of tobacco use at age 15 were obtained. Results: 15-year-olds with externalizing disorders between 2 and 11 years reported higher tobacco use than those without a history of disorder. This association could be followed back into early childhood and held up even after controlling for covariates. Conclusions: The findings suggest that childhood externalizing disorders may represent an independent risk factor for elevated tobacco use in adolescence},
  language  = {en}
}
@article{EsserLaucht2005,
  author    = {Esser, G{\"u}nter and Laucht, Manfred},
  title     = {Sind junge M{\"u}tter ein Risiko f{\"u}r die Kindesentwicklung?},
  year      = {2005},
  language  = {de}
}
@article{EsserGrueningKrugetal.2005,
  author    = {Esser, G{\"u}nter and Gr{\"u}ning, Th. and Krug, W. and May, P. and Meiers, K. and Trebert, M.},
  title     = {Wie Kindern mit Lernschwierigkeiten wirksam helfen?},
  year      = {2005},
  language  = {de}
}
@article{EsserLauchtSchmidt2005,
  author    = {Esser, G{\"u}nter and Laucht, Manfred and Schmidt, M. H.},
  title     = {Modell der Entstehung von Substanzmissbrauch : stellt die Fr{\"u}hkindheit die Weichen?},
  isbn      = {3-525-46237-9},
  year      = {2005},
  language  = {de}
}
@article{HirschbergerEsser2005,
  author    = {Hirschberger, E. and Esser, G{\"u}nter},
  title     = {Auditive selektive Aufmerksamkeit bei geriatrischen Patienten mit einem visuellen Neglect},
  isbn      = {3-89967-220-8},
  year      = {2005},
  language  = {de}
}
@article{EsserLangeWyschkon2006,
  author    = {Esser, G{\"u}nter and Lange, Sabine and Wyschkon, Anne},
  title     = {Bausteine der Diagnostik : Multimethodale Diagnostik},
  isbn      = {978-3-932096-43-3},
  year      = {2006},
  language  = {de}
}
@article{LangeEsser2005,
  author    = {Lange, Sabine and Esser, G{\"u}nter},
  title     = {Auditiv-sprachliche St{\"o}rung der Informationsverarbeitung bei Lese-Rechtschreib-St{\"o}rung.},
  isbn      = {3-89967-220-8},
  year      = {2005},
  language  = {de}
}
@article{LauchtHomEsseretal.2005,
  author    = {Laucht, Manfred and Hom, Erika and Esser, G{\"u}nter and Schmidt, Martin H.},
  title     = {Erh{\"o}htes Raucherrisiko von Kindern mit Aufmerksamkeits- und Verhaltensst{\"o}rungen},
  year      = {2005},
  language  = {de}
}
@article{LayIhleEsseretal.2005,
  author    = {Lay, Barbara and Ihle, Wolfgang and Esser, G{\"u}nter and Schmidt, Martin H.},
  title     = {Juvenile-episodic, continued or adult-onset delinquency? Risk conditions analysed in a cohort of children followed up to the age of 25 years},
  year      = {2005},
  language  = {en}
}
@article{SchneiderMuellerEsser2005,
  author    = {Schneider, A. and M{\"u}ller, D. and Esser, G{\"u}nter},
  title     = {EEG-Koh{\"a}renzanalyse zur Untersuchung eines Automatisierungsdefizits bei Lese-Rechtschreib-St{\"o}rung und ADHS : eine Pilotstudie},
  isbn      = {3-89967-220-8},
  year      = {2005},
  language  = {de}
}
@article{IhleEsserSchmidt2005,
  author    = {Ihle, Wolfgang and Esser, G{\"u}nter and Schmidt, M. H.},
  title     = {Aggressiv-dissoziale St{\"o}rungen und rechtsextreme Einstellungen : Pr{\"a}valenz, Geschlechtsunterschiede, Verlauf und Risikofaktoren},
  year      = {2005},
  language  = {de}
}
@article{AurichEsserMayetal.2005,
  author    = {Aurich, Eberhard and Esser, G{\"u}nter and May, P. and Meiers, K.},
  title     = {Gesellschaftliche Bedeutung der Schriftsprachkompetenz und M{\"o}glichkeiten ihrer F{\"o}rderung},
  year      = {2005},
  language  = {de}
}
@article{DopfnerEsser2004,
  author    = {Dopfner, M. and Esser, G{\"u}nter},
  title     = {Guidelines for assessment and psychotherapy},
  issn      = {0942-5403},
  year      = {2004},
  abstract  = {This editorial summarizes the currant state of development of guidelines for the assessment and treatment of mental disorders in children and adolescents. The aims of guidelines and criteria for the quality of guidelines are discussed. This special issue intends to be a starting point for the development of guidelines for psychological and psychotherapeutic disciplines in the German-speaking countries},
  language  = {en}
}
@article{IhleAhleJahnkeetal.2004,
  author    = {Ihle, Wolfgang and Ahle, M. E. and Jahnke, D{\"o}rte and Esser, G{\"u}nter},
  title     = {Guidelines for the assessment and psychotherapy of depressive disorders in childhood and adolescence: An evidence-based discussion paper},
  issn      = {0942-5403},
  year      = {2004},
  abstract  = {An outline of evidence-based guidelines for the assessment and treatment of depressive disorders in childhood and adolescence is presented. Depressive disorders in children and adolescents are marked by core symptoms similar to those seen in adults, although symptom expression varies greatly with developmental stage. These disorders are common, especially in adolescence, chronic, and recurrent, and are associated with comorbid conditions such as anxiety disorders, conduct disorders, and substance use disorders. Effective treatment approaches for the prevention of depressive disorders and the acute treatment of mild and moderate depressive disorders are available. The psychotherapeutic interventions of choice are currently cognitive behavioral therapy (CBT) and interpersonal therapy (IPT). The antidepressants of choice are currently selective serotonin reuptake inhibitors (SSRI). Especially on relapse prevention and the evaluation of the combination of psychotherapy with antidepressant medication further studies are necessary},
  language  = {en}
}
@article{EsserBlanzGeiseletal.2005,
  author    = {Esser, G{\"u}nter and Blanz, Bernhard and Geisel, B. and Laucht, Manfred},
  title     = {MEI Manneimer Elterninterview},
  isbn      = {3-8017-1860-3},
  year      = {2005},
  language  = {de}
}
@article{EsserBlanzIhleetal.2004,
  author    = {Esser, G{\"u}nter and Blanz, Bernhard and Ihle, Wolfgang and Schmidt, M. H.},
  title     = {Modell und Entstehung des Substanzmissbrauchs},
  isbn      = {3-525-49075-6},
  year      = {2004},
  language  = {de}
}
@misc{EsserWyschkonSchmidt2004,
  author    = {Esser, G{\"u}nter and Wyschkon, Anne and Schmidt, Martin H.},
  title     = {Anmerkungen},
  year      = {2004},
  language  = {de}
}
@article{DoepfnerEsser2004,
  author    = {D{\"o}pfner, Manfred and Esser, G{\"u}nter},
  title     = {Leitlinien zur Diagnostik und Psychotherapie},
  year      = {2004},
  language  = {de}
}
@article{Esser2004,
  author    = {Esser, G{\"u}nter},
  title     = {Verhaltenstherapie bei Kindern und Jugendlichen},
  isbn      = {978-3-932096-45-7},
  year      = {2004},
  language  = {de}
}
@article{HartmannWillnerEsser2004,
  author    = {Hartmann, Hellmut and Willner, Hans and Esser, G{\"u}nter},
  title     = {Ist die Aufmerksamkeits-Interaktions-Therapie (AIT) effektiv bei fr{\"u}hkindlichem Autismus?},
  year      = {2004},
  language  = {de}
}
@article{IhleAhleJahnkeetal.2004,
  author    = {Ihle, Wolfgang and Ahle, M. E. and Jahnke, D{\"o}rte and Esser, G{\"u}nter},
  title     = {Leitlinien zur Diagnostik und Psychologie von depressiven St{\"o}rungen im Kindes- und Jugendalter : ein evidenzbasierter Diskussionsvorschlag},
  year      = {2004},
  language  = {de}
}
@article{IhleAhleJahnkeetal.2004,
  author    = {Ihle, Wolfgang and Ahle, M. E. and Jahnke, D{\"o}rte and Esser, G{\"u}nter},
  title     = {Leitlinien zur Diagnostik und Psychotherapie von depressiven St{\"o}rungen im Kindes- und Jugendalter : ein evidenzbasierter Diskussionsvorschlag},
  year      = {2004},
  language  = {de}
}
@article{IhleEsserLauchtetal.2004,
  author    = {Ihle, Wolfgang and Esser, G{\"u}nter and Laucht, Manfred and Schmidt, M. H.},
  title     = {Depressive St{\"o}rungen und aggressiv-dissoziale St{\"o}rungen im Kindes- und Jugendalter : Pr{\"a}valenz, Verlauf und Risikofaktoren},
  year      = {2004},
  language  = {de}
}
@article{LauchtSchmidtEsser2004,
  author    = {Laucht, Manfred and Schmidt, M. H. and Esser, G{\"u}nter},
  title     = {The development of at-risk children in early life},
  year      = {2004},
  language  = {en}
}
@article{LauchtSchmidtEsser2003,
  author    = {Laucht, Manfred and Schmidt, Martin and Esser, G{\"u}nter},
  title     = {Fr{\"u}hkindliche Regulationsst{\"o}rungen: Vorl{\"a}ufer von Verhaltensst{\"o}rungen des sp{\"a}teren Kindesalters?},
  isbn      = {3- 456-84036-5},
  year      = {2003},
  language  = {de}
}
@article{Esser2003,
  author    = {Esser, G{\"u}nter},
  title     = {Verhaltensdiagnostik},
  isbn      = {3-13-126082-3},
  year      = {2003},
  language  = {de}
}
@article{EsserWyschkon2003,
  author    = {Esser, G{\"u}nter and Wyschkon, Anne},
  title     = {Entwicklungsdiagnostik im Vorschulalter},
  year      = {2003},
  language  = {de}
}