@misc{CostardStadieRitteretal.2011,
  author    = {Costard, Sylvia and Stadie, Nicole and Ritter, Christiane and Moll, Kristina and Landerl, Karin and Kohnen, Saskia and Kentner, Gerrit and Bethmann, Anja and Scheich, Henning and Brechmann, Andr{\´e} and De Kok, D{\"o}rte and Berger, Frauke and Sticher, Heike and Czepluch, Christine and M{\"a}tzener, Flurina and Wilmes, Stefanie and Hadert, Sandra and Frank, Ulrike and M{\"a}der, Mark and Westermann, Antje and Meinusch, Miriam and Neumann, Sandra and D{\"u}sterh{\"o}ft, Stefanie and Posse, Dorothea and Puritz, Caroline and Seidl, Rainer Ottis and Etzien, Maria and Machleb, Franziska and Lorenz, Antje and H{\"o}ger, Maria and Schr{\"o}der, Astrid and Busch, Tobias and Heide, Judith and Tagoe, Tanja and Watermeyer, Melanie and H{\"o}hle, Barbara and Kauschke, Christina},
  title     = {Spektrum Patholinguistik = Schwerpunktthema: Lesen lernen: Diagnostik und Therapie bei St{\"o}rungen des Leseerwerbs},
  number    = {4},
  editor    = {Hanne, Sandra and Fritzsche, Tom and Ott, Susan and Adelt, Anne},
  publisher = {Universit{\"a}tsverlag Potsdam},
  address   = {Potsdam},
  organization    = {Verband f{\"u}r Patholinguistik e. V. (vpl)},
  isbn      = {978-3-86956-145-5},
  issn      = {1869-3822},
  doi       = {10.25932/publishup-5155},
  url       = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus-53146},
  year      = {2011},
  abstract  = {Am 20. November 2010 fand an der Universit{\"a}t Potsdam das 4. Herbsttreffen Patholinguistik statt. Die Konferenzreihe wird regelm{\"a}ßig seit 2007 vom Verband f{\"u}r Patholinguistik e.V. (vpl) durchgef{\"u}hrt. Der vorliegende Tagungsband ver{\"o}ffentlicht die Hauptvortr{\"a}ge des Herbsttreffens zum Thema "Lesen lernen: Diagnostik und Therapie bei St{\"o}rungen des Leseerwerbs". Des Weiteren sind die Beitr{\"a}ge promovierender bzw. promovierter PatholinguistInnen sowie der Posterpr{\"a}sentationen enthalten.},
  language  = {de}
}
@misc{PasslackSchmiedchenRailaetal.2016,
  author    = {Paßlack, Nadine and Schmiedchen, Bettina and Raila, Jens and Schweigert, Florian J. and Stumpff, Friederike and Kohn, Barbara and Neumann, Konrad and Zentek, J{\"u}rgen},
  title     = {Impact of increasing dietary calcium levels on calcium excretion and vitamin D metabolites in the blood of healthy adult cats},
  series = {Postprints der Universit{\"a}t Potsdam : Mathematisch-Naturwissenschaftliche Reihe},
  journal   = {Postprints der Universit{\"a}t Potsdam : Mathematisch-Naturwissenschaftliche Reihe},
  number    = {542},
  issn      = {1866-8372},
  doi       = {10.25932/publishup-41130},
  url       = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-411302},
  pages     = {19},
  year      = {2016},
  abstract  = {Background Dietary calcium (Ca) concentrations might affect regulatory pathways within the Ca and vitamin D metabolism and consequently excretory mechanisms. Considering large variations in Ca concentrations of feline diets, the physiological impact on Ca homeostasis has not been evaluated to date. In the present study, diets with increasing concentrations of dicalcium phosphate were offered to ten healthy adult cats (Ca/phosphorus (P): 6.23/6.02, 7.77/7.56, 15.0/12.7, 19.0/17.3, 22.2/19.9, 24.3/21.6 g/kg dry matter). Each feeding period was divided into a 10-day adaptation and an 8-day sampling period in order to collect urine and faeces. On the last day of each feeding period, blood samples were taken. Results Urinary Ca concentrations remained unaffected, but faecal Ca concentrations increased (P < 0.001) with increasing dietary Ca levels. No effect on whole and intact parathyroid hormone levels, fibroblast growth factor 23 and calcitriol concentrations in the blood of the cats were observed. However, the calcitriol precursors 25(OH)D-2 and 25(OH)D-3, which are considered the most useful indicators for the vitamin D status, decreased with higher dietary Ca levels (P = 0.013 and P = 0.033). Increasing dietary levels of dicalcium phosphate revealed an acidifying effect on urinary fasting pH (6.02) and postprandial pH (6.01) (P < 0.001), possibly mediated by an increase of urinary phosphorus (P) concentrations (P < 0.001). Conclusions In conclusion, calcitriol precursors were linearly affected by increasing dietary Ca concentrations. The increase in faecal Ca excretion indicates that Ca homeostasis of cats is mainly regulated in the intestine and not by the kidneys. Long-term studies should investigate the physiological relevance of the acidifying effect observed when feeding diets high in Ca and P.},
  language  = {en}
}
@article{PasslackSchmiedchenRailaetal.2016,
  author    = {Passlack, Nadine and Schmiedchen, Bettina and Raila, Jens and Schweigert, Florian J. and Stumpff, Friederike and Kohn, Barbara and Neumann, Konrad and Zentek, Juergen},
  title     = {Impact of Increasing Dietary Calcium Levels on Calcium Excretion and Vitamin D Metabolites in the Blood of Healthy Adult Cats},
  series = {PLoS one},
  volume    = {11},
  journal   = {PLoS one},
  publisher = {PLoS},
  address   = {San Fransisco},
  issn      = {1932-6203},
  doi       = {10.1371/journal.pone.0149190},
  pages     = {47 -- 67},
  year      = {2016},
  abstract  = {Background Dietary calcium (Ca) concentrations might affect regulatory pathways within the Ca and vitamin D metabolism and consequently excretory mechanisms. Considering large variations in Ca concentrations of feline diets, the physiological impact on Ca homeostasis has not been evaluated to date. In the present study, diets with increasing concentrations of dicalcium phosphate were offered to ten healthy adult cats (Ca/phosphorus (P): 6.23/6.02, 7.77/7.56, 15.0/12.7, 19.0/17.3, 22.2/19.9, 24.3/21.6 g/kg dry matter). Each feeding period was divided into a 10-day adaptation and an 8-day sampling period in order to collect urine and faeces. On the last day of each feeding period, blood samples were taken. Results Urinary Ca concentrations remained unaffected, but faecal Ca concentrations increased (P < 0.001) with increasing dietary Ca levels. No effect on whole and intact parathyroid hormone levels, fibroblast growth factor 23 and calcitriol concentrations in the blood of the cats were observed. However, the calcitriol precursors 25(OH)D-2 and 25(OH)D-3, which are considered the most useful indicators for the vitamin D status, decreased with higher dietary Ca levels (P = 0.013 and P = 0.033). Increasing dietary levels of dicalcium phosphate revealed an acidifying effect on urinary fasting pH (6.02) and postprandial pH (6.01) (P < 0.001), possibly mediated by an increase of urinary phosphorus (P) concentrations (P < 0.001). Conclusions In conclusion, calcitriol precursors were linearly affected by increasing dietary Ca concentrations. The increase in faecal Ca excretion indicates that Ca homeostasis of cats is mainly regulated in the intestine and not by the kidneys. Long-term studies should investigate the physiological relevance of the acidifying effect observed when feeding diets high in Ca and P.},
  language  = {en}
}
@article{SchellerKleinjungBieretal.1998,
  author    = {Scheller, Frieder W. and Kleinjung, Frank and Bier, Frank Fabian and Markower, Alexander and Neumann, Barbara and Wollenberger, Ursula and Kurochkin, Iliya N. and Eremenko, Arkadi V. and Barmin, Anatoli V. and Klußmann, Sven and F{\"u}rste, Jens-Peter and Erdmann, Volker A. and Mansuy, D.},
  title     = {New recognition elements in biosensing},
  year      = {1998},
  language  = {en}
}
@article{RockstroemKotzeMilutinovićetal.2024,
  author    = {Rockstr{\"o}m, Johan and Kotz{\´e}, Louis and Milutinović, Svetlana and Biermann, Frank and Brovkin, Victor and Donges, Jonathan and Ebbesson, Jonas and French, Duncan and Gupta, Joyeeta and Kim, Rakhyun and Lenton, Timothy and Lenzi, Dominic and Nakicenovic, Nebojsa and Neumann, Barbara and Schuppert, Fabian and Winkelmann, Ricarda and Bosselmann, Klaus and Folke, Carl and Lucht, Wolfgang and Schlosberg, David and Richardson, Katherine and Steffen, Will},
  title     = {The planetary commons},
  series = {Proceedings of the National Academy of Sciences of the United States of America},
  volume    = {121},
  journal   = {Proceedings of the National Academy of Sciences of the United States of America},
  number    = {5},
  publisher = {National Academy of Sciences},
  address   = {Washington, DC},
  issn      = {1091-6490},
  doi       = {10.1073/pnas.2301531121},
  pages     = {10},
  year      = {2024},
  abstract  = {The Anthropocene signifies the start of a no- analogue tra­jectory of the Earth system that is fundamentally different from the Holocene. This new trajectory is characterized by rising risks of triggering irreversible and unmanageable shifts in Earth system functioning. We urgently need a new global approach to safeguard critical Earth system regulating functions more effectively and comprehensively. The global commons framework is the closest example of an existing approach with the aim of governing biophysical systems on Earth upon which the world collectively depends. Derived during stable Holocene conditions, the global commons framework must now evolve in the light of new Anthropocene dynamics. This requires a fundamental shift from a focus only on governing shared resources beyond national jurisdiction, to one that secures critical functions of the Earth system irrespective of national boundaries. We propose a new framework—the planetary commons—which differs from the global commons frame­work by including not only globally shared geographic regions but also critical biophysical systems that regulate the resilience and state, and therefore livability, on Earth. The new planetary commons should articulate and create comprehensive stewardship obligations through Earth system governance aimed at restoring and strengthening planetary resilience and justice.},
  language  = {en}
}