@inproceedings{KurbelNowakAzodietal.2015, author = {Kurbel, Karl and Nowak, Dawid and Azodi, Amir and Jaeger, David and Meinel, Christoph and Cheng, Feng and Sapegin, Andrey and Gawron, Marian and Morelli, Frank and Stahl, Lukas and Kerl, Stefan and Janz, Mariska and Hadaya, Abdulmasih and Ivanov, Ivaylo and Wiese, Lena and Neves, Mariana and Schapranow, Matthieu-Patrick and F{\"a}hnrich, Cindy and Feinbube, Frank and Eberhardt, Felix and Hagen, Wieland and Plauth, Max and Herscheid, Lena and Polze, Andreas and Barkowsky, Matthias and Dinger, Henriette and Faber, Lukas and Montenegro, Felix and Czach{\´o}rski, Tadeusz and Nycz, Monika and Nycz, Tomasz and Baader, Galina and Besner, Veronika and Hecht, Sonja and Schermann, Michael and Krcmar, Helmut and Wiradarma, Timur Pratama and Hentschel, Christian and Sack, Harald and Abramowicz, Witold and Sokolowska, Wioletta and Hossa, Tymoteusz and Opalka, Jakub and Fabisz, Karol and Kubaczyk, Mateusz and Cmil, Milena and Meng, Tianhui and Dadashnia, Sharam and Niesen, Tim and Fettke, Peter and Loos, Peter and Perscheid, Cindy and Schwarz, Christian and Schmidt, Christopher and Scholz, Matthias and Bock, Nikolai and Piller, Gunther and B{\"o}hm, Klaus and Norkus, Oliver and Clark, Brian and Friedrich, Bj{\"o}rn and Izadpanah, Babak and Merkel, Florian and Schweer, Ilias and Zimak, Alexander and Sauer, J{\"u}rgen and Fabian, Benjamin and Tilch, Georg and M{\"u}ller, David and Pl{\"o}ger, Sabrina and Friedrich, Christoph M. and Engels, Christoph and Amirkhanyan, Aragats and van der Walt, Est{\´e}e and Eloff, J. H. P. and Scheuermann, Bernd and Weinknecht, Elisa}, title = {HPI Future SOC Lab}, editor = {Meinel, Christoph and Polze, Andreas and Oswald, Gerhard and Strotmann, Rolf and Seibold, Ulrich and Schulzki, Bernhard}, url = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-102516}, pages = {iii, 154}, year = {2015}, abstract = {Das Future SOC Lab am HPI ist eine Kooperation des Hasso-Plattner-Instituts mit verschiedenen Industriepartnern. Seine Aufgabe ist die Erm{\"o}glichung und F{\"o}rderung des Austausches zwischen Forschungsgemeinschaft und Industrie. Am Lab wird interessierten Wissenschaftlern eine Infrastruktur von neuester Hard- und Software kostenfrei f{\"u}r Forschungszwecke zur Verf{\"u}gung gestellt. Dazu z{\"a}hlen teilweise noch nicht am Markt verf{\"u}gbare Technologien, die im normalen Hochschulbereich in der Regel nicht zu finanzieren w{\"a}ren, bspw. Server mit bis zu 64 Cores und 2 TB Hauptspeicher. Diese Angebote richten sich insbesondere an Wissenschaftler in den Gebieten Informatik und Wirtschaftsinformatik. Einige der Schwerpunkte sind Cloud Computing, Parallelisierung und In-Memory Technologien. In diesem Technischen Bericht werden die Ergebnisse der Forschungsprojekte des Jahres 2015 vorgestellt. Ausgew{\"a}hlte Projekte stellten ihre Ergebnisse am 15. April 2015 und 4. November 2015 im Rahmen der Future SOC Lab Tag Veranstaltungen vor.}, language = {en} } @article{VorburgerNedielkovBrosigetal.2016, author = {Vorburger, Thomas and Nedielkov, Ruslan and Brosig, Alexander and Bok, Eva and Schunke, Emina and Steffen, Wojtek and Mayer, Sonja and Goetz, Friedrich and M{\"o}ller, Heiko Michael and Steuber, Julia}, title = {Role of the Na+-translocating NADH:quinone oxidoreductase in voltage generation and Na+ extrusion in Vibrio cholerae}, series = {Biochimica et biophysica acta : Bioenergetics}, volume = {1857}, journal = {Biochimica et biophysica acta : Bioenergetics}, publisher = {Elsevier}, address = {Amsterdam}, issn = {0005-2728}, doi = {10.1016/j.bbabio.2015.12.010}, pages = {473 -- 482}, year = {2016}, abstract = {For Vibrio cholerae, the coordinated import and export of Na+ is crucial for adaptation to habitats with different osmolarities. We investigated the Na+-extruding branch of the sodium cycle in this human pathogen by in vivo Na-23-NMR spectroscopy. The Na+ extrusion activity of cells was monitored after adding glucose which stimulated respiration via the Na+-translocating NADH:quinone oxidoreductase (Na+-NQR). In a V. cholerae deletion mutant devoid of the Na+-NQR encoding genes (nqrA-F), rates of respiratory Na+ extrusion were decreased by a factor of four, but the cytoplasmic Na+ concentration was essentially unchanged. Furthermore, the mutant was impaired in formation of transmembrane voltage (Delta psi, inside negative) and did not grow under hypoosmotic conditions at pH 8.2 or above. This growth defect could be complemented by transformation with the plasmid encoded nqr operon. In an alkaline environment, Na+/H+ antiporters acidify the cytoplasm at the expense of the transmembrane voltage. It is proposed that, at alkaline pH and limiting Na+ concentrations, the Na+-NQR is crucial for generation of a transmembrane voltage to drive the import of H+ by electrogenic Na+/H+ antiporters. Our study provides the basis to understand the role of the Na+-NQR in pathogenicity of V. cholerae and other pathogens relying on this primary Na+ pump for respiration. (C) 2015 Elsevier B.V. All rights reserved.}, language = {en} } @article{CastellanosFriedrichPetrovicetal.2020, author = {Castellanos, Reynel Urrea and Friedrich, Thomas and Petrovic, Nevena and Altmann, Simone and Brzezinka, Krzysztof and Gorka, Michal and Graf, Alexander and B{\"a}urle, Isabel}, title = {FORGETTER2 protein phosphatase and phospholipase D modulate heat stress memory in Arabidopsis}, series = {The plant journal}, volume = {104}, journal = {The plant journal}, number = {1}, publisher = {Wiley}, address = {Hoboken}, issn = {0960-7412}, doi = {10.1111/tpj.14927}, pages = {7 -- 17}, year = {2020}, abstract = {Plants can mitigate environmental stress conditions through acclimation. In the case of fluctuating stress conditions such as high temperatures, maintaining a stress memory enables a more efficient response upon recurring stress. In a genetic screen forArabidopsis thalianamutants impaired in the memory of heat stress (HS) we have isolated theFORGETTER2(FGT2) gene, which encodes a type 2C protein phosphatase (PP2C) of the D-clade.Fgt2mutants acquire thermotolerance normally; however, they are defective in the memory of HS. FGT2 interacts with phospholipase D alpha 2 (PLD alpha 2), which is involved in the metabolism of membrane phospholipids and is also required for HS memory. In summary, we have uncovered a previously unknown component of HS memory and identified the FGT2 protein phosphatase and PLD alpha 2 as crucial players, suggesting that phosphatidic acid-dependent signaling or membrane composition dynamics underlie HS memory.}, language = {en} } @article{KabelitzBrzezinkaFriedrichetal.2016, author = {Kabelitz, Tina and Brzezinka, Krzysztof and Friedrich, Thomas and Gorka, Michal and Graf, Alexander and Kappel, Christian and B{\"a}urle, Isabel}, title = {A JUMONJI Protein with E3 Ligase and Histone H3 Binding Activities Affects Transposon Silencing in Arabidopsis}, series = {Plant physiology : an international journal devoted to physiology, biochemistry, cellular and molecular biology, biophysics and environmental biology of plants}, volume = {171}, journal = {Plant physiology : an international journal devoted to physiology, biochemistry, cellular and molecular biology, biophysics and environmental biology of plants}, publisher = {American Society of Plant Physiologists}, address = {Rockville}, issn = {0032-0889}, doi = {10.1104/pp.15.01688}, pages = {344 -- 358}, year = {2016}, abstract = {Transposable elements (TEs) make up a large proportion of eukaryotic genomes. As their mobilization creates genetic variation that threatens genome integrity, TEs are epigenetically silenced through several pathways, and this may spread to neighboring sequences. JUMONJI (JMJ) proteins can function as antisilencing factors and prevent silencing of genes next to TEs. Whether TE silencing is counterbalanced by the activity of antisilencing factors is still unclear. Here, we characterize JMJ24 as a regulator of TE silencing. We show that loss of JMJ24 results in increased silencing of the DNA transposon AtMu1c, while overexpression of JMJ24 reduces silencing. JMJ24 has a JumonjiC (JmjC) domain and two RING domains. JMJ24 autoubiquitinates in vitro, demonstrating E3 ligase activity of the RING domain(s). JMJ24-JmjC binds the N-terminal tail of histone H3, and full-length JMJ24 binds histone H3 in vivo. JMJ24 activity is anticorrelated with histone H3 Lys 9 dimethylation (H3K9me2) levels at AtMu1c. Double mutant analyses with epigenetic silencing mutants suggest that JMJ24 antagonizes histone H3K9me2 and requires H3K9 methyltransferases for its activity on AtMu1c. Genome-wide transcriptome analysis indicates that JMJ24 affects silencing at additional TEs. Our results suggest that the JmjC domain of JMJ24 has lost demethylase activity but has been retained as a binding domain for histone H3. This is in line with phylogenetic analyses indicating that JMJ24 (with the mutated JmjC domain) is widely conserved in angiosperms. Taken together, this study assigns a role in TE silencing to a conserved JmjC-domain protein with E3 ligase activity, but no demethylase activity.}, language = {en} } @phdthesis{Friedrich2020, author = {Friedrich, Alexander}, title = {Minimizers of generalized Willmore energies and applications in general relativity}, doi = {10.25932/publishup-48142}, url = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-481423}, school = {Universit{\"a}t Potsdam}, pages = {100}, year = {2020}, abstract = {Das Willmore Funktional ist eine Funktion die jeder Fl{\"a}che in einer Riemannschen Mannigfaltigkeit, ihre totale mittlere Kr{\"u}mmung zuweist. Ein klassisches Problem der Differentialgeometrie ist es geschlossene (kompakt und ohne Rand) Fl{\"a}chen zu finden die das Willmore funktional minimieren, beziehungsweise die kritische Punkte des Willmore Funktionals sind. In dieser Doktorarbeit entwickeln wir ein Konzept von verallgemeinerten Willmore Funktionalen f{\"u}r Fl{\"a}chen in Riemannschen Mannigfaltigkeiten, wobei wir uns von physikalischen Modellen leiten lassen. Insbesondere ist hier die Hawking Energie der allgemeinen Relativit{\"a}tstheorie und die Biegungsenergie von d{\"u}nnen Membranen zu nennen. F{\"u}r dieses verallgemeinerten Willmore Funktionale beweisen wir die Existenz von Minimieren mit vorgeschriebenen Fl{\"a}cheninhalt, in einer geeigneten Klasse von verallgemeinerten Fl{\"a}chen. Insbesondere konstruieren wir Minimierer der oben erw{\"a}hnten Biegungsenergie mit vorgeschrieben Fl{\"a}cheninhalt und vorgeschriebenen, eingeschlossenem Volumen. Außerdem beweisen wir, dass kritische Punkte von verallgemeinerten Willmore Funktionalen mit vorgeschriebenen Fl{\"a}cheninhalt abseits endlich vieler Punkte glatt sind. Dabei st{\"u}tzen wir uns, wie auch im folgenden, auf die bestehende Theorie f{\"u}r das Willmore Funktional. An diese allgemeinen Resultate schließen wir eine detailliertere Analyse der Hawking Energie an. Im Kontext der allgemeinen Relativit{\"a}tstheorie beschreibt die Umgebungsmannigfaltigkeit den Raum zu einem Zeitpunkt. Daher sind wir an dem Wechselspiel zwischen der Hawking Energie und der umgebenden Mannigfaltigkeit interessiert. Wir charakterisieren Punkte in der umgebenden Mannigfaltigkeit f{\"u}r die es in jeder Umgebung eine kritische Fl{\"a}che mit vorgeschriebenem, kleinem Fl{\"a}cheninhalt gibt. Diese Punnkte werden als Konzentrationspunkte der Hawking Energie interpretiert. Außerdem berechnen wir eine Entwicklung der Hawking Energie auf kleinen, runden Sph{\"a}ren. Dadurch k{\"o}nnen wir eine Art Energiedichte der Hawking Energie identifizieren. Hierbei ist anzumerken, dass unsere Resultate im Kontrast zu Ergebnissen in der Literatur stehen. Dort wurde berechnet, dass die Entwicklung der Hawking Energie auf Sph{\"a}ren im Lichtkegel eines Punktes der umgebenden Mannigfaltigkeit in f{\"u}hrender Ordnung proportional zur der klassischen Energiedichte der allgemeinen Relativit{\"a}tstheorie ist. Zu diesem Zeitpunkt ist nicht klar wie diese Diskrepanz zu begr{\"u}nden ist. Ferner betrachten wir asymptotisch Schwarzschild Mannigfaltigkeiten. Sie sind ein Spezialfall von asymptotisch flachen Mannigfaltigkeiten, welche in der allgemeinen Relativit{\"a}tstheorie als Modelle f{\"u}r isolierte Systeme dienen. Die Schwarzschild Raumzeit selbst ist eine rotationssymmetrische Raumzeit die schwarzen Loch beschreibt. In diesen asymptotisch Schwarzschild Mannigfaltigkeiten konstruieren wir eine Bl{\"a}tterung des {\"a}ußeren Bereiches durch kritische Fl{\"a}chen der Hawking Energie mit vorgeschriebenen Fl{\"a}cheninhalt. Diese Bl{\"a}tterung kann in einem verallgemeinertem Sinne als Schwerpunkt des isolierten Systems betrachtet werden. Außerdem zeigen wir, dass die Hawking Energie entlang der Bl{\"a}tterung w{\"a}chst je gr{\"o}ßer die Fl{\"a}chen werden.}, language = {en} } @article{EngeliLehmannKaminskietal.2014, author = {Engeli, Stefan and Lehmann, Anne-Christin and Kaminski, Jana and Haas, Verena and Janke, Urgen and Janke, J{\"u}rgen and Zoerner, Alexander A. and Luft, Friedrich C. and Tsikas, Dimitrios and Jordan, Jens}, title = {Influence of dietary fat intake on the endocannabinoid system in lean and obese subjects}, series = {Obesity}, volume = {22}, journal = {Obesity}, number = {5}, publisher = {Wiley-Blackwell}, address = {Hoboken}, issn = {1930-7381}, doi = {10.1002/oby.20728}, pages = {E70 -- E76}, year = {2014}, abstract = {Objective: Endocannabinoid system (ECS) activation promotes obesity-associated metabolic disease. Increased dietary fat intake increases blood endocannabinoids and alters adipose and skeletal muscle ECS gene expression in human. Methods: Two weeks isocaloric low- (LFD) and high-fat diets (HFD) in obese (n = 12) and normal- weight (n = 17) subjects in a randomized cross-over study were compared. Blood endocannabinoids were measured in the fasting condition and after food intake using mass spectrometry. Adipose and skeletal muscle gene expression was determined using real-time RT-PCR. Results: Baseline fasting plasma endocannabinoids were similar with both diets. Anandamide decreased similarly with high- or low-fat test meals in both groups. Baseline arachidonoylglycerol plasma concentrations were similar between groups and diets, and unresponsive to eating. In subcutaneous adipose tissue, DAGL-alpha mRNA was upregulated and fatty acid amide hydrolase (FAAH) and monoacylglycerol lipase (MAGL) mRNAs were down-regulated in obese subjects, but the diets had no influence. In contrast, the HFD produced pronounced reductions in skeletal muscle CB1-R and MAGL mRNA expression, whereas obesity did not affect muscular gene expression. Conclusions: Weight-neutral changes in dietary fat intake cannot explain excessive endocannabinoid availability in human obesity. Obesity and dietary fat intake affect ECS gene expression in a tissue-specific manner.}, language = {en} } @article{KathreinKipnusuKremeretal.2015, author = {Kathrein, Christine C. and Kipnusu, Wycliffe K. and Kremer, Friedrich and B{\"o}ker, Alexander}, title = {Birefringence Analysis of the Effect of Electric Fields on the Order-Disorder Transition Temperature of Lamellae Forming Block Copolymers}, series = {Macromolecules : a publication of the American Chemical Society}, volume = {48}, journal = {Macromolecules : a publication of the American Chemical Society}, number = {10}, publisher = {American Chemical Society}, address = {Washington}, issn = {0024-9297}, doi = {10.1021/acs.macromol.5b00512}, pages = {3354 -- 3359}, year = {2015}, abstract = {A detailed birefringence analysis of the effect of strong dc electric fields on the order-disorder transition temperature (T-ODT) of lamella forming block copolymers is reported. The setup presented here enabled the measurement of the T-ODT with high temperature resolution while the birefringence measurements were nondestructive and straightforward compared to alternative methods. A downward shift in the transition temperature was found for all samples upon application of the electric field. The data indicate that the dominating parameter that evokes the mixing of block copolymers when exposed to electric fields is the difference in dielectric permittivity Delta epsilon between the block copolymer constituents. The extent to which the T-ODT is shifted is furthermore influenced by the degree of polymerization N. Shifts in the transition temperature of up to 7 degrees C were found upon application of an electric field of 5 kV/mm.}, language = {en} } @misc{SanchezSanzLaudenbachWeissetal.2023, author = {Sanchez Sanz, Arturo and Laudenbach, Beno{\^i}t and Weiß, Adrian and Werner, Eva and Stachon, Markus and Anders, Friedrich and Barthel, Christian and Berrens, Dominik and Avalli, Andrea and Vandewalle, Alexander and Ferrara, Pasquale and Pohl, Patrik}, title = {Spring Issue}, series = {thersites}, volume = {2023}, journal = {thersites}, number = {16}, editor = {Amb{\"u}hl, Annemarie and Carl{\`a}-Uhink, Filippo and Rollinger, Christian and Walde, Christine}, issn = {2364-7612}, doi = {10.34679/thersites.vol16}, year = {2023}, language = {en} } @article{FriedrichOberkoflerTrindadeetal.2021, author = {Friedrich, Thomas and Oberkofler, Vicky and Trindade, In{\^e}s and Altmann, Simone and Brzezinka, Krzysztof and L{\"a}mke, J{\"o}rn S. and Gorka, Michal and Kappel, Christian and Sokolowska, Ewelina and Skirycz, Aleksandra and Graf, Alexander and B{\"a}urle, Isabel}, title = {Heteromeric HSFA2/HSFA3 complexes drive transcriptional memory after heat stress in Arabidopsis}, series = {Nature Communications}, volume = {12}, journal = {Nature Communications}, number = {1}, publisher = {Nature Publishing Group UK}, address = {[London]}, issn = {2041-1723}, doi = {10.1038/s41467-021-23786-6}, pages = {15}, year = {2021}, abstract = {Adaptive plasticity in stress responses is a key element of plant survival strategies. For instance, moderate heat stress (HS) primes a plant to acquire thermotolerance, which allows subsequent survival of more severe HS conditions. Acquired thermotolerance is actively maintained over several days (HS memory) and involves the sustained induction of memory-related genes. Here we show that FORGETTER3/ HEAT SHOCK TRANSCRIPTION FACTOR A3 (FGT3/HSFA3) is specifically required for physiological HS memory and maintaining high memory-gene expression during the days following a HS exposure. HSFA3 mediates HS memory by direct transcriptional activation of memory-related genes after return to normal growth temperatures. HSFA3 binds HSFA2, and in vivo both proteins form heteromeric complexes with additional HSFs. Our results indicate that only complexes containing both HSFA2 and HSFA3 efficiently promote transcriptional memory by positively influencing histone H3 lysine 4 (H3K4) hyper-methylation. In summary, our work defines the major HSF complex controlling transcriptional memory and elucidates the in vivo dynamics of HSF complexes during somatic stress memory. Moderate heat stress primes plants to acquire tolerance to subsequent, more severe heat stress. Here the authors show that the HSFA3 transcription factor forms a heteromeric complex with HSFA2 to sustain activated transcription of genes required for acquired thermotolerance by promoting H3K4 hyper-methylation.}, language = {en} } @misc{AndressDrewsGomezGutierrezetal.2016, author = {Andress, Reinhard and Drews, Julian and G{\´o}mez Guti{\´e}rrez, Alberto and Reich, Karin and Roussanova, Elena and St{\"o}ger, Alexander and Herneck, Friedrich and Schwarz, Ingo}, title = {HiN : Alexander von Humboldt im Netz}, series = {HIN : Alexander von Humboldt im Netz ; international review for Humboldtian studies}, volume = {XVII}, journal = {HIN : Alexander von Humboldt im Netz ; international review for Humboldtian studies}, number = {33}, editor = {Ette, Ottmar and Knobloch, Eberhard}, publisher = {Universit{\"a}tsverlag Potsdam}, address = {Potsdam}, issn = {1617-5239}, doi = {0.18443/hinvol17iss332016}, url = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-98944}, pages = {90}, year = {2016}, abstract = {Inhalt - Reinhard Andress: Ein unver{\"o}ffentlichter Brief Alexander von Humboldts an den Buchh{\"a}ndler Jean-Georges Treuttel - Julian Drews: {\´E}criture (auto)biographique dans l'Examen critique d'Alexandre de Humboldt - Alberto G{\´o}mez Guti{\´e}rrez: Alexander von Humboldt y la cooperaci{\´o}n transcontinental en la Geograf{\´i}a de las plantas: una nueva apreciaci{\´o}n de la obra fitogeogr{\´a}fica de Francisco Jos{\´e} de Caldas - Karin Reich und Elena Roussanova: Der Briefwechsel zwischen Karl Kreil und Alexander von Humboldt, ein wichtiger Beitrag zur Geschichte des Erdmagnetismus - Alexander St{\"o}ger: Experiment und Wissensvermittlung. Alexander von Humboldts Darstellungsmethoden in seinen Versuchen {\"u}ber die gereizte Muskel- und Nervenfaser - Friedrich Herneck und Ingo Schwarz: Friedrich Herneck: Hegel und Alexander von Humboldt}, language = {de} } @book{RanaMohapatraSidorovaetal.2022, author = {Rana, Kaushik and Mohapatra, Durga Prasad and Sidorova, Julia and Lundberg, Lars and Sk{\"o}ld, Lars and Lopes Grim, Lu{\´i}s Fernando and Sampaio Gradvohl, Andr{\´e} Leon and Cremerius, Jonas and Siegert, Simon and Weltzien, Anton von and Baldi, Annika and Klessascheck, Finn and Kalancha, Svitlana and Lichtenstein, Tom and Shaabani, Nuhad and Meinel, Christoph and Friedrich, Tobias and Lenzner, Pascal and Schumann, David and Wiese, Ingmar and Sarna, Nicole and Wiese, Lena and Tashkandi, Araek Sami and van der Walt, Est{\´e}e and Eloff, Jan H. P. and Schmidt, Christopher and H{\"u}gle, Johannes and Horschig, Siegfried and Uflacker, Matthias and Najafi, Pejman and Sapegin, Andrey and Cheng, Feng and Stojanovic, Dragan and Stojnev Ilić, Aleksandra and Djordjevic, Igor and Stojanovic, Natalija and Predic, Bratislav and Gonz{\´a}lez-Jim{\´e}nez, Mario and de Lara, Juan and Mischkewitz, Sven and Kainz, Bernhard and van Hoorn, Andr{\´e} and Ferme, Vincenzo and Schulz, Henning and Knigge, Marlene and Hecht, Sonja and Prifti, Loina and Krcmar, Helmut and Fabian, Benjamin and Ermakova, Tatiana and Kelkel, Stefan and Baumann, Annika and Morgenstern, Laura and Plauth, Max and Eberhard, Felix and Wolff, Felix and Polze, Andreas and Cech, Tim and Danz, Noel and Noack, Nele Sina and Pirl, Lukas and Beilharz, Jossekin Jakob and De Oliveira, Roberto C. L. and Soares, F{\´a}bio Mendes and Juiz, Carlos and Bermejo, Belen and M{\"u}hle, Alexander and Gr{\"u}ner, Andreas and Saxena, Vageesh and Gayvoronskaya, Tatiana and Weyand, Christopher and Krause, Mirko and Frank, Markus and Bischoff, Sebastian and Behrens, Freya and R{\"u}ckin, Julius and Ziegler, Adrian and Vogel, Thomas and Tran, Chinh and Moser, Irene and Grunske, Lars and Sz{\´a}rnyas, G{\´a}bor and Marton, J{\´o}zsef and Maginecz, J{\´a}nos and Varr{\´o}, D{\´a}niel and Antal, J{\´a}nos Benjamin}, title = {HPI Future SOC Lab - Proceedings 2018}, number = {151}, editor = {Meinel, Christoph and Polze, Andreas and Beins, Karsten and Strotmann, Rolf and Seibold, Ulrich and R{\"o}dszus, Kurt and M{\"u}ller, J{\"u}rgen}, publisher = {Universit{\"a}tsverlag Potsdam}, address = {Potsdam}, isbn = {978-3-86956-547-7}, issn = {1613-5652}, doi = {10.25932/publishup-56371}, url = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-563712}, publisher = {Universit{\"a}t Potsdam}, pages = {x, 277}, year = {2022}, abstract = {The "HPI Future SOC Lab" is a cooperation of the Hasso Plattner Institute (HPI) and industry partners. Its mission is to enable and promote exchange and interaction between the research community and the industry partners. The HPI Future SOC Lab provides researchers with free of charge access to a complete infrastructure of state of the art hard and software. This infrastructure includes components, which might be too expensive for an ordinary research environment, such as servers with up to 64 cores and 2 TB main memory. The offerings address researchers particularly from but not limited to the areas of computer science and business information systems. Main areas of research include cloud computing, parallelization, and In-Memory technologies. This technical report presents results of research projects executed in 2018. Selected projects have presented their results on April 17th and November 14th 2017 at the Future SOC Lab Day events.}, language = {en} }