@article{SchmidtKasparRichter2008,
  author    = {Schmidt, Martin and Kaspar, R{\"u}diger and Richter, Torsten},
  title     = {Ergebnisse der Tagung des BFA Mykologie (NABU) 2006 in Linowsee bei Rheinsberg/Brandenburg},
  editor    = {Kummer, Volker},
  year      = {2008},
  language  = {de}
}
@article{LauchtSkowronekBeckeretal.2008,
  author    = {Laucht, Manfred and Skowronek, Markus H. and Becker, Katja and Schulze, Thomas G. and Schmidt, Martin H. and Esser, G{\"u}nter and Rietschel, Marcella},
  title     = {Environmental risk factors and attention-deficit : hyperactivity discorder symptoms ; reply},
  issn      = {0003-990X},
  year      = {2008},
  language  = {en}
}
@article{BeckerElFaddaghSchmidtetal.2008,
  author    = {Becker, Katja and El-Faddagh, Mahha and Schmidt, Martin H. and Esser, G{\"u}nter and Laucht, Manfred},
  title     = {Interaction of dopamine transporter genotype with prenatal smoke exposure on ADHD symptoms},
  issn      = {0022-3476},
  year      = {2008},
  abstract  = {Objective To demonstrate that children homozygous for the 10-repeat allele of the common dopamine transporter (DAT1) polymorphism who were exposed to maternal prenatal smoke exhibited significantly higher hyperactivity-impulsivity than children without these environmental or genetic risks. Study design We performed a prospective longitudinal study from birth into early adulthood monitoring the long-term outcome of early risk factors. Maternal prenatal smoking was determined during a standardized interview with the mother when the child was 3 months old. At age 15 years, 305 adolescents participated in genotyping for the DAT1 40 base pair variable number of tandem repeats polymorphism and assessment of inattention, hyperactivity-impulsivity, and oppositional defiant/conduct disorder symptoms with die Kiddie- Sads-Present and Lifetime Version. Results There was no bivariate association between DAT1 genotype, prenatal smoke exposure and symptoms of attention deficit hyperactivity disorder. However, a significant interaction between DAT1 genotype and prenatal smoke exposure emerged (P =.012), indicating that males with prenatal smoke exposure who were homozygous for the DAT1 10r allele had higher hyperactivity-impulsivity than males from all other groups. In females, no significant main effects of DAT1 genotype or prenatal smoke exposure or interaction effects on any symptoms were evident (all P >.25). Conclusions This study provides further evidence for the multifactorial nature of attention deficit hyperactivity disorder and the importance of studying both genetic and environmental factors and their interaction.},
  language  = {en}
}
@article{LauchtBeckerFranketal.2008,
  author    = {Laucht, Manfred and Becker, Katja and Frank, Josef and Schmidt, Martin H. and Esser, G{\"u}nter and Treutlein, Jens and Skowronek, Markus H. and Schumann, Gunter},
  title     = {Genetic variation in dopamine pathways differentially associated with smoking progression in adolescence},
  issn      = {0890-8567},
  doi       = {10.1097/Chi.0b013e31816bff77},
  year      = {2008},
  abstract  = {Objective: To clarify the nature of the association between dopamine genes and smoking by examining whether genetic variability in components of the dopamine pathway could explain refined phenotypes in adolescent smoking progression. Method: Data are from an ongoing prospective study of the long-term outcome of early risk factors studied since birth. At age 15 years, 220 participants (108 males, 112 females) completed a self-report questionnaire measuring smoking behavior and were genotyped for five dopamine gene variants. Results: Smoking initiation was related to allelic variation in the dopamine D-4 receptor gene (DRD4), whereas smoking continuation and dependence showed association with the dopamine D-2 receptor gene (DRD2). Adolescents with the seven-repeat allele of the common DRD4 exon 3 polymorphism had rates of ever smoking that were significantly higher than in those with other genotypes. Once smoking started, carriers of the T allele of a single nucleotide polymorphism of DRD2 (rs4648317) reported higher rates of current smoking and scored higher on nicotine dependence than their allelic counterparts. Among current smokers, intention to quit was significantly lower in adolescents homozygous for the 10-repeat allele of the common dopamine transporter 3 untranslated region polymorphism. Conclusions: Our results provide preliminary evidence of genetic influences on different stages of smoking and suggest the importance of specific dopamine genes in smoking progression in adolescence.},
  language  = {en}
}
@article{EsserWyschkonSchmidtetal.2008,
  author    = {Esser, G{\"u}nter and Wyschkon, Anne and Schmidt, Martin H. and Blanz, Bernhard and Ihle, Wolfgang},
  title     = {Ein Entwicklungsmodell des Substanzmissbrauchs im fr{\"u}hen Erwachsenenalter},
  issn      = {0942-5403},
  doi       = {10.1026/0942-5403.17.1.31},
  year      = {2008},
  language  = {de}
}