@article{EsserWyschkon2001,
  author    = {Esser, G{\"u}nter and Wyschkon, Anne},
  title     = {17 Jahre danach : was wird aus Kindern mit Legasthenie?},
  year      = {2001},
  language  = {de}
}
@article{WyschkonSchulzGallitetal.2017,
  author    = {Wyschkon, Anne and Schulz, Franziska and Gallit, Finja Sunnyi and Poltz, Nadine and Kohn-Henkel, Juliane and Moraske, Svenja and Bondue, Rebecca and von Aster, Michael G. and Esser, G{\"u}nter},
  title     = {5-Jahres-Verlauf der LRS},
  series = {Zeitschrift f{\"u}r Kinder- und Jugendpsychiatrie und Psychotherapie},
  volume    = {46},
  journal   = {Zeitschrift f{\"u}r Kinder- und Jugendpsychiatrie und Psychotherapie},
  number    = {2},
  publisher = {Hogrefe},
  address   = {Bern},
  issn      = {1422-4917},
  doi       = {10.1024/1422-4917/a000535},
  pages     = {107 -- 122},
  year      = {2017},
  abstract  = {Fragestellung: Untersucht wird der Verlauf von Kindern mit Lese-Rechtschreibst{\"o}rungen (LRS) {\"u}ber gut 5 Jahre unter Ber{\"u}cksichtigung des Einflusses des Geschlechts der Betroffenen. Außerdem werden Auswirkungen der LRS auf das sp{\"a}tere Schriftsprachniveau und den Schulerfolg {\"u}berpr{\"u}ft. Methodik: Eingangs wurden 995 Sch{\"u}ler zwischen 6 und 16 Jahren untersucht. Ein Teil dieser Kinder ist nach 43 sowie 63 Monaten nachuntersucht worden. Eine LRS wurde diagnostiziert, wenn f{\"u}r das Lesen bzw. Rechtschreiben das doppelte Diskrepanzkriterium von 1.5 Standardabweichungen zur nonverbalen Intelligenz und dem Mittelwert der Klassenstufe erf{\"u}llt war und gleichzeitig keine Minderbegabung vorlag. Ergebnisse: Die LRS weist {\"u}ber einen Zeitraum von 63 Monaten eine hohe St{\"o}rungspersistenz von knapp 70 \% auf. Der 5-Jahres-Verlauf der mittleren Lese- und Rechtschreibleistungen wurde nicht vom Geschlecht beeinflusst. Trotz durchschnittlicher Intelligenz blieben die LRS-Sch{\"u}ler in der Schriftsprache mindestens eine Standardabweichung hinter durchschnittlich und etwa 0.5 Standardabweichungseinheiten hinter unterdurchschnittlich intelligenten Kindern zur{\"u}ck. Der Schulerfolg der LRS-Sch{\"u}ler glich dem unterdurchschnittlich intelligenter Kinder und fiel deutlich schlechter aus als bei durchschnittlich intelligenten Kontrollkindern. Schlussfolgerungen: Eine LRS stellt ein erhebliches Entwicklungsrisiko dar, was fr{\"u}hzeitige Diagnostik- und Therapiemaßnahmen erfordert. Daf{\"u}r sind reliable und im Hinblick auf die resultierenden Pr{\"a}valenzraten sinnvolle, allgemein anerkannte Diagnosekriterien essenziell.},
  language  = {de}
}
@misc{KuhlmannBuergerEsseretal.2015,
  author    = {Kuhlmann, Sophie Merle and B{\"u}rger, Arne and Esser, G{\"u}nter and Hammerle, Florian},
  title     = {A mindfulness-based stress prevention training for medical students (MediMind)},
  series = {Postprints der Universit{\"a}t Potsdam : Mathematisch-Naturwissenschaftliche Reihe 820},
  journal   = {Postprints der Universit{\"a}t Potsdam : Mathematisch-Naturwissenschaftliche Reihe 820},
  number    = {820},
  issn      = {1866-8372},
  doi       = {10.25932/publishup-42756},
  url       = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-427568},
  pages     = {13},
  year      = {2015},
  abstract  = {Background: Medical training is very demanding and associated with a high prevalence of psychological distress. Compared to the general population, medical students are at a greater risk of developing a psychological disorder. Various attempts of stress management training in medical school have achieved positive results on minimizing psychological distress; however, there are often limitations. Therefore, the use of a rigorous scientific method is needed. The present study protocol describes a randomized controlled trial to examine the effectiveness of a specifically developed mindfulness-based stress prevention training for medical students that includes selected elements of cognitive behavioral strategies (MediMind). Methods/Design: This study protocol presents a prospective randomized controlled trial, involving four assessment time points: baseline, post-intervention, one-year follow-up and five-year follow-up. The aims include evaluating the effect on stress, coping, psychological morbidity and personality traits with validated measures. Participants are allocated randomly to one of three conditions: MediMind, Autogenic Training or control group. Eligible participants are medical or dental students in the second or eighth semester of a German university. They form a population of approximately 420 students in each academic term. A final total sample size of 126 (at five-year follow-up) is targeted. The trainings (MediMind and Autogenic Training) comprise five weekly sessions lasting 90 minutes each. MediMind will be offered to participants of the control group once the five-year follow-up is completed. The allotment is randomized with a stratified allocation ratio by course of studies, semester, and gender. After descriptive statistics have been evaluated, inferential statistical analysis will be carried out with a repeated measures ANOVA-design with interactions between time and group. Effect sizes will be calculated using partial η-square values. Discussion: Potential limitations of this study are voluntary participation and the risk of attrition, especially concerning participants that are allocated to the control group. Strengths are the study design, namely random allocation, follow-up assessment, the use of control groups and inclusion of participants at different stages of medical training with the possibility of differential analysis.},
  language  = {en}
}
@article{KuhlmannBuergerEsseretal.2015,
  author    = {Kuhlmann, Sophie Merle and B{\"u}rger, Arne and Esser, G{\"u}nter and Hammerle, Florian},
  title     = {A mindfulness-based stress prevention training for medical students (MediMind): study protocol for a randomized controlled trial},
  series = {Trials},
  volume    = {16},
  journal   = {Trials},
  publisher = {BioMed Central},
  address   = {London},
  issn      = {1745-6215},
  doi       = {10.1186/s13063-014-0533-9},
  pages     = {11},
  year      = {2015},
  abstract  = {Background: Medical training is very demanding and associated with a high prevalence of psychological distress. Compared to the general population, medical students are at a greater risk of developing a psychological disorder. Various attempts of stress management training in medical school have achieved positive results on minimizing psychological distress; however, there are often limitations. Therefore, the use of a rigorous scientific method is needed. The present study protocol describes a randomized controlled trial to examine the effectiveness of a specifically developed mindfulness-based stress prevention training for medical students that includes selected elements of cognitive behavioral strategies (MediMind). Methods/Design: This study protocol presents a prospective randomized controlled trial, involving four assessment time points: baseline, post-intervention, one-year follow-up and five-year follow-up. The aims include evaluating the effect on stress, coping, psychological morbidity and personality traits with validated measures. Participants are allocated randomly to one of three conditions: MediMind, Autogenic Training or control group. Eligible participants are medical or dental students in the second or eighth semester of a German university. They form a population of approximately 420 students in each academic term. A final total sample size of 126 (at five-year follow-up) is targeted. The trainings (MediMind and Autogenic Training) comprise five weekly sessions lasting 90 minutes each. MediMind will be offered to participants of the control group once the five-year follow-up is completed. The allotment is randomized with a stratified allocation ratio by course of studies, semester, and gender. After descriptive statistics have been evaluated, inferential statistical analysis will be carried out with a repeated measures ANOVA-design with interactions between time and group. Effect sizes will be calculated using partial.-square values. Discussion: Potential limitations of this study are voluntary participation and the risk of attrition, especially concerning participants that are allocated to the control group. Strengths are the study design, namely random allocation, follow-up assessment, the use of control groups and inclusion of participants at different stages of medical training with the possibility of differential analysis.},
  language  = {en}
}
@article{EsserIhle2008,
  author    = {Esser, G{\"u}nter and Ihle, Wolfgang},
  title     = {Abh{\"a}ngigkeit von legalen und illegalen psychotropen Substanzen},
  year      = {2008},
  language  = {de}
}
@article{Esser2002,
  author    = {Esser, G{\"u}nter},
  title     = {Ablehnung und Vernachl{\"a}ssigung von S{\"a}uglingen},
  year      = {2002},
  language  = {de}
}
@article{KohnEsser2008,
  author    = {Kohn, Juliane and Esser, G{\"u}nter},
  title     = {ADHS im Jugend- und Erwachsenenalter},
  issn      = {0026-9298},
  doi       = {10.1007/s00112-008-1731-x},
  year      = {2008},
  language  = {de}
}
@article{IhleEsserSchmidt2005,
  author    = {Ihle, Wolfgang and Esser, G{\"u}nter and Schmidt, M. H.},
  title     = {Aggressiv-dissoziale St{\"o}rungen und rechtsextreme Einstellungen : Pr{\"a}valenz, Geschlechtsunterschiede, Verlauf und Risikofaktoren},
  year      = {2005},
  language  = {de}
}
@article{HeinrichBuchmannZohseletal.2015,
  author    = {Heinrich, Angela and Buchmann, Arlette F. and Zohsel, Katrin and Dukal, Helene and Frank, Josef and Treutlein, Jens and Nieratschker, Vanessa and Witt, Stephanie H. and Brandeis, Daniel and Schmidt, Martin H. and Esser, G{\"u}nter and Banaschewski, Tobias and Laucht, Manfred and Rietschel, Marcella},
  title     = {Alterations of Glucocorticoid Receptor Gene Methylation in Externalizing Disorders During Childhood and Adolescence},
  series = {Behavior genetics : an international journal devoted to research in the inheritance of behavior in animals and man},
  volume    = {45},
  journal   = {Behavior genetics : an international journal devoted to research in the inheritance of behavior in animals and man},
  number    = {5},
  publisher = {Springer},
  address   = {New York},
  issn      = {0001-8244},
  doi       = {10.1007/s10519-015-9721-y},
  pages     = {529 -- 536},
  year      = {2015},
  abstract  = {Epigenetic modulations are a hypothesized link between environmental factors and the development of psychiatric disorders. Research has suggested that patients with depression or bipolar disorder exhibit higher methylation levels in the glucocorticoid receptor gene NR3C1. We aimed to investigate whether NR3C1 methylation changes are similarly associated with externalizing disorders such as aggressive behavior and conduct disorder. NR3C1 exon 1F methylation was analyzed in young adults with a lifetime diagnosis of an externalizing disorder (N = 68) or a depressive disorder (N = 27) and healthy controls (N = 124) from the Mannheim Study of Children at Risk. The externalizing disorders group had significantly lower NR3C1 methylation levels than the lifetime depressive disorder group (p = 0.009) and healthy controls (p = 0.001) This report of lower methylation levels in NR3C1 in externalizing disorders may indicate a mechanism through which the differential development of externalizing disorders as opposed to depressive disorders might occur.},
  language  = {en}
}
@article{WittBuchmannBlomeyeretal.2011,
  author    = {Witt, Stephanie H. and Buchmann, Arlette F. and Blomeyer, Dorothea and Nieratschker, Vanessa and Treutlein, Jens and Esser, G{\"u}nter and Schmidt, Martin H. and Bidlingmaier, Martin and Wiedemann, Klaus and Rietschel, Marcella and Laucht, Manfred and Wuest, Stefan and Zimmermann, Ulrich S.},
  title     = {An interaction between a neuropeptide Y gene polymorphism and early adversity modulates endocrine stress responses},
  series = {Psychoneuroendocrinology},
  volume    = {36},
  journal   = {Psychoneuroendocrinology},
  number    = {7},
  publisher = {Elsevier},
  address   = {Oxford},
  issn      = {0306-4530},
  doi       = {10.1016/j.psyneuen.2010.12.015},
  pages     = {1010 -- 1020},
  year      = {2011},
  abstract  = {Interindividual variability in the regulation of the human stress system accounts for a part of the individual's liability to stress-related diseases. These differences are influenced by environmental and genetic factors. Early childhood adversity is a well-studied environmental factor affecting an individual's stress response which has been shown to be modulated by gene environment interaction (GxE). Neuropeptide Y (NPY) plays a role in stress regulation and genetic variation in NPY may influence stress responses. In this study, we analyzed the association of a common variant in the NPY gene promoter, rs16147, with cortisol and ACTH responses to acute psychosocial stress in young adults from the Mannheim Study of Children at Risk (MARS), an ongoing epidemiological cohort study following the outcome of early adversity from birth into adulthood. We found evidence of a GxE interaction between rs16147 and early adversity significantly affecting HPA axis responses to acute psychosocial stress. These findings suggest that the neurobiological mechanisms linking early adverse experience and later neuroendocrine stress regulation are modulated by a gene variant whose functional relevance is documented by increasing convergent evidence from in vitro, animal and human studies.},
  language  = {en}
}
@article{IhleEsserSchmidtetal.1999,
  author    = {Ihle, Wolfgang and Esser, G{\"u}nter and Schmidt, Martin H. and Blanz, Bernhard and Reis, Olaf and Meyer-Probst, Bernhard},
  title     = {Angst als psychosoziales Ph{\"a}nomen und psychotherapeutisches Anliegen},
  year      = {1999},
  language  = {de}
}
@misc{EsserWyschkonSchmidt2004,
  author    = {Esser, G{\"u}nter and Wyschkon, Anne and Schmidt, Martin H.},
  title     = {Anmerkungen},
  year      = {2004},
  language  = {de}
}
@article{Esser1999,
  author    = {Esser, G{\"u}nter},
  title     = {Anmerkungen zur Kaufmann-Assessment Battery for Children (K-ABC)},
  year      = {1999},
  language  = {de}
}
@article{EsserFreybergerHoffmannetal.2006,
  author    = {Esser, G{\"u}nter and Freyberger, H. J. and Hoffmann, S. O. and Hoyer, J. and Richter, R. and Harfst, T.},
  title     = {Anpassung der Psychotherapie-Richtlinien zur Zulassung psychotherapeutischer Verfahren und Methoden},
  year      = {2006},
  language  = {de}
}
@article{NikitopoulosZohselBlomeyeretal.2014,
  author    = {Nikitopoulos, Joerg and Zohsel, Katrin and Blomeyer, Dorothea and Buchmann, Arlette F. and Schmid, Brigitte and Jennen-Steinmetz, Christine and Becker, Katja and Schmidt, Martin H. and Esser, G{\"u}nter and Brandeis, Daniel and Banaschewski, Tobias and Laucht, Manfred},
  title     = {Are infants differentially sensitive to parenting? Early maternal care, DRD4 genotype and externalizing behavior during adolescence},
  series = {Journal of psychiatric research},
  volume    = {59},
  journal   = {Journal of psychiatric research},
  publisher = {Elsevier},
  address   = {Oxford},
  issn      = {0022-3956},
  doi       = {10.1016/j.jpsychires.2014.08.012},
  pages     = {53 -- 59},
  year      = {2014},
  language  = {en}
}
@inproceedings{LauchtBlomeyerElFaddaghetal.2011,
  author    = {Laucht, Manfred and Blomeyer, Dorothea and El-Faddagh, Mahha and Esser, G{\"u}nter and Schmidt, Martin and Banaschewski, Tobias and Becker, Katja},
  title     = {Are regulatory problems in infancy precursors of ADHD in childhood? a moderating role for the dopamine D4 receptor gene},
  series = {Infant mental health journal},
  volume    = {32},
  booktitle = {Infant mental health journal},
  number    = {3},
  publisher = {Wiley-Blackwell},
  address   = {Hoboken},
  issn      = {0163-9641},
  pages     = {212 -- 212},
  year      = {2011},
  language  = {en}
}
@article{LauchtHohmEsseretal.2007,
  author    = {Laucht, Manfred and Hohm, E. and Esser, G{\"u}nter and Schmidt, Martin H. and Becker, Katja},
  title     = {Association between ADHD and smoking in adolescence : shared genetic, environmental and psychopathological factors},
  issn      = {0300-9564},
  doi       = {10.1007/s00702-007-0703-y},
  year      = {2007},
  abstract  = {The present study aimed to examine the extent to which the co-occurrence of ADHD and smoking in adolescents could be attributed to common genetic, environmental and psychopathological factors. Data are from an ongoing prospective study of the outcome of early risk factors. At age 15 years, 305 adolescents completed self-report questionnaires measuring tobacco consumption and deviant peer affiliations. Lifetime psychiatric diagnoses were obtained using standardized interviews. DNA was genotyped for the dopamine D4 receptor (DRD4) gene exon III polymorphism. Adolescents with a lifetime diagnosis of ADHD displayed significantly higher smoking activity than non-ADHD controls. A major component of this association could be accounted for by deviant peer affiliations and the comorbidity with oppositional-defiant and conduct disorder, while a minor part was attributable to DRD4 in males but not in females. These findings suggest that the association of ADHD with smoking relies on risk factors shared by the two behaviors.},
  language  = {en}
}
@article{BoeckerSchlierHolzHohmetal.2017,
  author    = {Boecker-Schlier, Regina and Holz, Nathalie E. and Hohm, Erika and Zohsel, Katrin and Blomeyer, Dorothea and Buchmann, Arlette F. and Baumeister, Sarah and Wolf, Isabella and Esser, G{\"u}nter and Schmidt, Martin H. and Meyer-Lindenberg, Andreas and Banaschewski, Tobias and Brandeis, Daniel and Laucht, Manfred},
  title     = {Association between pubertal stage at first drink and neural reward processing in early adulthood},
  series = {Addiction biology},
  volume    = {22},
  journal   = {Addiction biology},
  publisher = {Wiley},
  address   = {Hoboken},
  issn      = {1355-6215},
  doi       = {10.1111/adb.12413},
  pages     = {1402 -- 1415},
  year      = {2017},
  abstract  = {Puberty is a critical time period during human development. It is characterized by high levels of risk-taking behavior, such as increased alcohol consumption, and is accompanied by various neurobiological changes. Recent studies in animals and humans have revealed that the pubertal stage at first drink (PSFD) significantly impacts drinking behavior in adulthood. Moreover, neuronal alterations of the dopaminergic reward system have been associated with alcohol abuse or addiction. This study aimed to clarify the impact of PSFD on neuronal characteristics of reward processing linked to alcohol-related problems. One hundred sixty-eight healthy young adults from a prospective study covering 25 years participated in a monetary incentive delay task measured with simultaneous EEG-fMRI. PSFD was determined according to the age at menarche or Tanner stage of pubertal development, respectively. Alcohol-related problems in early adulthood were assessed with the Alcohol Use Disorder Identification Test (AUDIT). During reward anticipation, decreased fMRI activation of the frontal cortex and increased preparatory EEG activity (contingent negative variation) occurred with pubertal compared to postpubertal first alcohol intake. Moreover, alcohol-related problems during early adulthood were increased in pubertal compared to postpubertal beginners, which was mediated by neuronal activation of the right medial frontal gyrus. At reward delivery, increased fMRI activation of the left caudate and higher feedback-related EEG negativity were detected in pubertal compared to postpubertal beginners. Together with animal findings, these results implicate PSFD as a potential modulator of psychopathology, involving altered reward anticipation. Both PSFD timing and reward processing might thus be potential targets for early prevention and intervention.},
  language  = {en}
}
@article{HohmannHohmTreutleinetal.2015,
  author    = {Hohmann, Sarah and Hohm, Erika and Treutlein, Jens and Blomeyer, Dorothea and Jennen-Steinmetz, Christine and Schmidt, Martin H. and Esser, G{\"u}nter and Banaschewski, Tobias and Brandeis, Daniel and Laucht, Manfred},
  title     = {Association of norepinephrine transporter (NET, SLC6A2) genotype with ADHD-related phenotypes: Findings of a longitudinal study from birth to adolescence},
  series = {Psychiatry research : the official publication of the International Society for Neuroimaging in Psychiatry},
  volume    = {226},
  journal   = {Psychiatry research : the official publication of the International Society for Neuroimaging in Psychiatry},
  number    = {2-3},
  publisher = {Elsevier},
  address   = {Clare},
  issn      = {0165-1781},
  doi       = {10.1016/j.psychres.2014.12.029},
  pages     = {425 -- 433},
  year      = {2015},
  abstract  = {Variation in the gene encoding for the norepinephrine transporter (NET, SLC6A2) has repeatedly been linked with ADHD, although there is some inconsistency regarding the association with specific genes. The variants for which most consistent association has been found are the NET variants rs3785157 and rs28386840. Here, we tested for their association with ADHD diagnosis and ADHD-related phenotypes during development in a longitudinal German community sample. Children were followed from age 4 to age 15, using diagnostic interviews to assess ADHD. Between the ages of 8 and 15 years, the Child Behavior Checklist (CBCL) was administered to the primary caregivers. The continuous performance task (CPT) was performed at age 15. Controlling for possible confounders, we found that homozygous carriers of the major A allele of the functional promoter variant rs28386840 displayed a higher rate of ADHD lifetime diagnosis. Moreover, homozygous carriers of the minor T allele of rs3785157 were more likely to develop ADHD and showed higher scores on the CBCL externalizing behavior scales. Additionally, we found that individuals heterozygous for rs3785157 made fewer omission errors in the CPT than homozygotes. This is the first longitudinal study to report associations between specific NET variants and ADHD-related phenotypes during the course of development. (C) 2015 Elsevier Ireland Ltd. All rights reserved.},
  language  = {en}
}
@article{BlomeyerBuchmannLascorzetal.2013,
  author    = {Blomeyer, Dorothea and Buchmann, Arlette F. and Lascorz, Jesus and Zimmermann, Ulrich S. and Esser, G{\"u}nter and Desrivieres, Sylvane and Schmidt, Martin H. and Banaschewski, Tobias and Schumann, Gunter and Laucht, Manfred},
  title     = {Association of PER2 genotype and stressful life events with alcohol drinking in young adults},
  series = {PLoS one},
  volume    = {8},
  journal   = {PLoS one},
  number    = {3},
  publisher = {PLoS},
  address   = {San Fransisco},
  issn      = {1932-6203},
  doi       = {10.1371/journal.pone.0059136},
  pages     = {7},
  year      = {2013},
  abstract  = {Background: Clock genes govern circadian rhythms and shape the effect of alcohol use on the physiological system. Exposure to severe negative life events is related to both heavy drinking and disturbed circadian rhythmicity. The aim of this study was 1) to extend previous findings suggesting an association of a haplotype tagging single nucleotide polymorphism of PER2 gene with drinking patterns, and 2) to examine a possible role for an interaction of this gene with life stress in hazardous drinking. Methods: Data were collected as part of an epidemiological cohort study on the outcome of early risk factors followed since birth. At age 19 years, 268 young adults (126 males, 142 females) were genotyped for PER2 rs56013859 and were administered a 45-day alcohol timeline follow-back interview and the Alcohol Use Disorders Identification Test (AUDIT). Life stress was assessed as the number of severe negative life events during the past four years reported in a questionnaire and validated by interview. Results: Individuals with the minor G allele of rs56013859 were found to be less engaged in alcohol use, drinking at only 72\% of the days compared to homozygotes for the major A allele. Moreover, among regular drinkers, a gene x environment interaction emerged (p = .020). While no effects of genotype appeared under conditions of low stress, carriers of the G allele exhibited less hazardous drinking than those homozygous for the A allele when exposed to high stress. Conclusions: These findings may suggest a role of the circadian rhythm gene PER2 in both the drinking patterns of young adults and in moderating the impact of severe life stress on hazardous drinking in experienced alcohol users. However, in light of the likely burden of multiple tests, the nature of the measures used and the nominal evidence of interaction, replication is needed before drawing firm conclusions.},
  language  = {en}
}
@article{WeindrichJennenSteinmetzLauchtetal.1998,
  author    = {Weindrich, D. and Jennen-Steinmetz, Christine and Laucht, Manfred and Esser, G{\"u}nter and Schmidt, Martin H.},
  title     = {At risk for language disorders? : correlates and course of language disorders in preschool children born at risk},
  issn      = {0803-5253},
  year      = {1998},
  language  = {en}
}
@article{LangeEsser2005,
  author    = {Lange, Sabine and Esser, G{\"u}nter},
  title     = {Auditiv-sprachliche St{\"o}rung der Informationsverarbeitung bei Lese-Rechtschreib-St{\"o}rung.},
  isbn      = {3-89967-220-8},
  year      = {2005},
  language  = {de}
}
@article{HirschbergerEsser2005,
  author    = {Hirschberger, E. and Esser, G{\"u}nter},
  title     = {Auditive selektive Aufmerksamkeit bei geriatrischen Patienten mit einem visuellen Neglect},
  isbn      = {3-89967-220-8},
  year      = {2005},
  language  = {de}
}
@article{PolowczykTrautmannVillalbaDinterJoergetal.2000,
  author    = {Polowczyk, M. and Trautmann-Villalba, Patricia and Dinter-J{\"o}rg, Monika and Gerold, M. and Laucht, Manfred and Schmidt, Martin H. and Esser, G{\"u}nter},
  title     = {Auff{\"a}llige Mutter-Kind-Interaktion im Vorschulalter bei Kindern mit hyperkinetischen und Sozialverhaltensauff{\"a}lligkeiten},
  year      = {2000},
  language  = {de}
}
@article{LangeEsser2008,
  author    = {Lange, Sabine and Esser, G{\"u}nter},
  title     = {Ausbildung zum Kinder- und Jugendlichenpsychotherapeuten},
  isbn      = {978-3-13-126083-3},
  year      = {2008},
  language  = {de}
}
@article{WyschkonKohnBallaschketal.2008,
  author    = {Wyschkon, Anne and Kohn, Juliane and Ballaschk, Katja and Esser, G{\"u}nter},
  title     = {Basisdiagnostik Umschriebener Entwicklungsst{\"o}rungen im Grundschulalter (BUEGA)},
  issn      = {1864-6050},
  year      = {2008},
  language  = {de}
}
@book{EsserWyschkon2002,
  author    = {Esser, G{\"u}nter and Wyschkon, Anne},
  title     = {Basisdiagnostik umschriebener Entwicklungsst{\"o}rungen im Vorschulalter : (BUEVA)},
  publisher = {Beltz},
  address   = {G{\"o}ttingen},
  pages     = {84 S.},
  year      = {2002},
  abstract  = {Ziel der Vorschultestbatterie ist es, Teilleistungsst{\"o}rungen bereits vor Schulbeginn zu erkennen und damit einer fr{\"u}hzeitigen F{\"o}rderung zuzuf{\"u}hren. Das diagnostische Vorgehen zeichnet sich durch eine hohe Durchf{\"u}hrungs{\"o}konomie und einen breiten Anwendungs-bereich aus. Insgesamt werden bei Vierj{\"a}hrigen die allgemeine Intelligenz, die Artikulation, die expressive und die rezeptive Sprache sowie die Visuomotorik in 20 bis 25 Minuten erfasst, bei den F{\"u}nfj{\"a}hrigen kommen zus{\"a}tzlich noch Pr{\"u}fungen der Aufmerksamkeit hinzu, was die Durchf{\"u}hrung um f{\"u}nf bis zehn Minuten gegen{\"u}ber den Vierj{\"a}hrigen verl{\"a}ngert. Die vorliegende Testbatterie kann auch im Rahmen von Schuleingangsuntersuchungen verl{\"a}sslich eingesetzt werden.},
  language  = {de}
}
@book{EsserWyschkon2012,
  author    = {Esser, G{\"u}nter and Wyschkon, Anne},
  title     = {Basisdiagnostik umschriebener Entwicklungsst{\"o}rungen im Vorschulalter : (BUEVA) Version II},
  publisher = {Beltz},
  address   = {G{\"o}ttingen},
  pages     = {100 S.},
  year      = {2012},
  language  = {de}
}
@article{Esser2003,
  author    = {Esser, G{\"u}nter},
  title     = {Basisdiagnostik umschriebener Entwicklungsst{\"o}rungen im Vorschulalter f{\"u}r p{\"a}diatrische Praxis},
  year      = {2003},
  abstract  = {Leistungsdiagnostik in der Klinischen Kinder- und Jugendpsychologie muss neben den klassischen G{\"u}tekriterien den Beitrag eines Testverfahrens zur Diagnosestellung, die Gr{\"o}ße und Repr{\"a}sentativit{\"a}t der Normstichprobe, seine Differenzierungsf{\"a}higkeit im unteren Leistungsbereich sowie die {\"O}konomie ber{\"u}cksichtigen. Ziel der BUEVA ist es, Teilleistungsst{\"o}rungen bereits vor Schulbeginn zu erkennen und damit einer fr{\"u}hzeitigen F{\"o}rderung zuzuf{\"u}hren. Das diagnostische Vorgehen zeichnet sich durch eine hohe Durchf{\"u}hrungs{\"o}konomie aus, wodurch der verk{\"u}rzten Aufmerksamkeitsspanne der Kinder Rechnung getragen wird. Insgesamt werden bei Vierj{\"a}hrigen die allgemeine Intelligenz, die Artikulation, die expressive und die rezeptive Sprache sowie die Visuomotorik in 20 bis 25 Minuten erfasst, bei den F{\"u}nfj{\"a}hrigen kommen zus{\"a}tzlich noch Pr{\"u}fungen der Aufmerksamkeit hinzu, was die Durchf{\"u}hrung um f{\"u}nf Minuten gegen{\"u}ber den Vierj{\"a}hrigen verl{\"a}ngert. Die vorliegende Testbatterie weist eine gute pr{\"a}diktive Validit{\"a}t auf und kann auch im Rahmen von Schuleingangsuntersuchungen verl{\"a}sslich eingesetzt werden. Die Testbatterie differenziert vor allem im subnormalen Leistungsbereich. Dies wird durch eine relativ hohe Zahl leichter Items sowie durch einfache Instruktionen erreicht. Zur Diagnostik von besonderen Begabungen ist die vorliegende Testbatterie also nicht geeignet. Schl{\"u}sselw{\"o}rter: Fr{\"u}herkennung, Vorschulalter, Basisdiagnostik, allgemeine Intelligenz, Artikulation, expressive, rezeptive Sprache, Visuomotorik, Aufmerksamkeit, umschriebene Entwicklungsst{\"o}rung},
  language  = {de}
}
@article{EsserLangeWyschkon2006,
  author    = {Esser, G{\"u}nter and Lange, Sabine and Wyschkon, Anne},
  title     = {Bausteine der Diagnostik : Multimethodale Diagnostik},
  isbn      = {978-3-932096-43-3},
  year      = {2006},
  language  = {de}
}
@article{BuchmannHellwegRietscheletal.2013,
  author    = {Buchmann, Arlette F. and Hellweg, Rainer and Rietschel, Marcella and Treutlein, Jens and Witt, Stephanie H. and Zimmermann, Ulrich S. and Schmidt, Martin H. and Esser, G{\"u}nter and Banaschewski, Tobias and Laucht, Manfred and Deuschle, Michael},
  title     = {BDNF Val 66 Met and 5-HTTLPR genotype moderate the impact of early psychosocial adversity on plasma brain-derived neurotrophic factor and depressive symptoms - a prospective study},
  series = {European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology},
  volume    = {23},
  journal   = {European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology},
  number    = {8},
  publisher = {Elsevier},
  address   = {Amsterdam},
  issn      = {0924-977X},
  doi       = {10.1016/j.euroneuro.2012.09.003},
  pages     = {902 -- 909},
  year      = {2013},
  abstract  = {Recent studies have emphasized an important role for neurotrophins, such as brain-derived neurotrophic factor (BDNF), in regulating the plasticity of neural circuits involved in the pathophysiology of stress-related diseases. The aim of the present study was to examine the interplay of the BDNF Val(66)Met and the serotonin transporter promoter (5-HTTLPR) polymorphisms in moderating the impact of early-life adversity on BDNF plasma concentration and depressive symptoms. Participants were taken from an epidemiological cohort study following the long-term outcome of early risk factors from birth into young adulthood. In 259 individuals (119 males, 140 females), genotyped for the BDNF Val(66)Met and the 5-HTTLPR polymorphisms, plasma BDNF was assessed at the age of 19 years. In addition, participants completed the Beck Depression Inventory (BDI). Early adversity was determined according to a family adversity index assessed at 3 months of age. Results indicated that individuals homozygous for both the BDNF Val and the 5-HTTLPR L allele showed significantly reduced BDNF levels following exposure to high adversity. In contrast, BDNF levels appeared to be unaffected by early psychosocial adversity in carriers of the BDNF Met or the 5-HTTLPR S allele. While the former group appeared to be most susceptible to depressive symptoms, the impact of early adversity was less pronounced in the latter group. This is the first preliminary evidence indicating that early-life adverse experiences may have lasting sequelae for plasma BDNF levels in humans, highlighting that the susceptibility to this effect is moderated by BDNF Val(66)Met and 5-HTTLPR genotype.},
  language  = {en}
}
@article{EsserDinterJoergSchmidtetal.1996,
  author    = {Esser, G{\"u}nter and Dinter-J{\"o}rg, Monika and Schmidt, Martin H. and Herrle, Johannes and Yantorno-Villalba, P. and Rose, Frauke and Laucht, Manfred},
  title     = {Bedeutung der Blickvermeidung im S{\"a}uglingsalter f{\"u}r den Entwicklungsstand des Kindes mit zwei und viereinhalb Jahren},
  year      = {1996},
  language  = {de}
}
@article{EsserWyschkon2007,
  author    = {Esser, G{\"u}nter and Wyschkon, Anne},
  title     = {Bedeutung komorbider St{\"o}rung im Kindesalter f{\"u}r den Langzeitverlauf der ADHS},
  isbn      = {978-3-17-019081-8},
  year      = {2007},
  language  = {de}
}
@article{SchmidtLayIhleetal.2001,
  author    = {Schmidt, Martin H. and Lay, Barbara and Ihle, Wolfgang and Esser, G{\"u}nter},
  title     = {Bedeutung von Dunkelfelddelikten f{\"u}r episodische und fortgesetzte Delinquenz},
  year      = {2001},
  language  = {de}
}
@article{SteigleiderLauchtEsseretal.2002,
  author    = {Steigleider, Petra and Laucht, Manfred and Esser, G{\"u}nter and Schmidt, Martin H.},
  title     = {Beeintr{\"a}chtigte kognitive und motorische Leistungen bei 8-j{\"a}hrigen Kindern mit sehr niedrigem Geburtsgewicht},
  issn      = {0084-5345},
  year      = {2002},
  language  = {de}
}
@article{HohmZohselSchmidtetal.2017,
  author    = {Hohm, Erika and Zohsel, Katrin and Schmidt, Martin H. and Esser, G{\"u}nter and Brandeis, Daniel and Banaschewski, Tobias and Laucht, Manfred},
  title     = {Beeintr{\"a}chtigter Start ins Leben},
  series = {Kindheit und Entwicklung},
  volume    = {26},
  journal   = {Kindheit und Entwicklung},
  publisher = {Hogrefe},
  address   = {G{\"o}ttingen},
  issn      = {0942-5403},
  doi       = {10.1026/0942-5403/a000234},
  pages     = {210 -- 220},
  year      = {2017},
  abstract  = {Postpartale Depressionen sind h{\"a}ufige und schwerwiegende psychische Erkrankungen mit ung{\"u}nstigem Einfluss auf die kindliche Entwicklung. Als Haupttransmissionsweg gilt die fr{\"u}he Mutter-Kind-Interaktion. {\"U}ber die langfristigen Auswirkungen auf die Kinder im Erwachsenenalter und die Rolle der Interaktion liegen kaum Ergebnisse vor. Im Rahmen der Mannheimer Risikokinderstudie wurden postpartale Depressionen bis zwei Jahre nach der Geburt erfasst. Die kindliche Entwicklung wurde fortlaufend und die Mutter-Kind-Interaktion im Alter von 3 Monaten standardisiert erhoben. 28 Kinder postpartal depressiver und 107 Kinder gesunder M{\"u}tter konnten mit 25 Jahren untersucht werden. Beeintr{\"a}chtigungen der kognitiven und psychischen Entwicklung bei Kindern postpartal depressiver M{\"u}tter waren bis ins Erwachsenenalter nachweisbar. Responsives bzw. sensitives m{\"u}tterliches Verhalten wirkte der negativen Entwicklung entgegen. Dies betont die Bedeutung einer hohen Qualit{\"a}t der Mutter-Kind-Interaktion f{\"u}r die Entwicklung von Risikokindern.},
  language  = {de}
}
@misc{HohmZohselSchmidtetal.2017,
  author    = {Hohm, Erika and Zohsel, Katrin and Schmidt, Martin H. and Esser, G{\"u}nter and Brandeis, Daniel and Banaschewski, Tobias and Laucht, Manfred},
  title     = {Beeintr{\"a}chtigter Start ins Leben},
  series = {Postprints der Universit{\"a}t Potsdam : Humanwissenschaftliche Reihe},
  journal   = {Postprints der Universit{\"a}t Potsdam : Humanwissenschaftliche Reihe},
  number    = {692},
  issn      = {1866-8364},
  doi       = {10.25932/publishup-43340},
  url       = {http://nbn-resolving.de/urn:nbn:de:kobv:517-opus4-433406},
  pages     = {37},
  year      = {2017},
  abstract  = {Postpartale Depressionen sind h{\"a}ufige und schwerwiegende psychische Erkrankungen mit ung{\"u}nstigem Einfluss auf die kindliche Entwicklung. Als Haupttransmissionsweg gilt die fr{\"u}he Mutter-Kind-Interaktion. {\"U}ber die langfristigen Auswirkungen auf die Kinder im Erwachsenenalter und die Rolle der Interaktion liegen kaum Ergebnisse vor. Im Rahmen der Mannheimer Risikokinderstudie wurden postpartale Depressionen bis zwei Jahre nach der Geburt erfasst. Die kindliche Entwicklung wurde fortlaufend und die Mutter-Kind-Interaktion im Alter von 3 Monaten standardisiert erhoben. 28 Kinder postpartal depressiver und 107 Kinder gesunder M{\"u}tter konnten mit 25 Jahren untersucht werden. Beeintr{\"a}chtigungen der kognitiven und psychischen Entwicklung bei Kindern postpartal depressiver M{\"u}tter waren bis ins Erwachsenenalter nachweisbar. Responsives bzw. sensitives m{\"u}tterliches Verhalten wirkte der negativen Entwicklung entgegen. Dies betont die Bedeutung einer hohen Qualit{\"a}t der Mutter-Kind-Interaktion f{\"u}r die Entwicklung von Risikokindern.},
  language  = {de}
}
@article{LauchtEsserHoeschetal.2000,
  author    = {Laucht, Manfred and Esser, G{\"u}nter and Hoesch, I. and Gerold, M. and Hoesch, I. and Ihle, Wolfgang and Steigleider, Petra and Stock, B. and Stoehr, R.-M. and Weindrich, D. and Schmidt, Martin H.},
  title     = {Behavioral Sequelae of Perinatal Insults and Early Family Adversity at 8 Years of Age},
  year      = {2000},
  language  = {en}
}
@misc{LauchtBlomeyerBuchmannetal.2012,
  author    = {Laucht, Manfred and Blomeyer, Dorothea and Buchmann, Arlette F. and Treutlein, Jens and Shmidt, Martin H. and Esser, G{\"u}nter and Jennen-Steinmetz, Christine and Rietschel, Marcella and Zimmermann, Ulrich S. and Banaschewski, Tobias},
  title     = {Catechol-O-methyltransferase Val158Met genotype, parenting practices and adolescent alcohol use: testing the differential susceptibility hypothesis},
  year      = {2012},
  language  = {en}
}
@article{LauchtBlomeyerBuchmannetal.2012,
  author    = {Laucht, Manfred and Blomeyer, Dorothea and Buchmann, Arlette F. and Treutlein, Jens and Schmidt, Martin H. and Esser, G{\"u}nter and Jennen-Steinmetz, Christine and Rietschel, Marcella and Zimmermann, Ulrich S. and Banaschewski, Tobias},
  title     = {Catechol-O-methyltransferase Val158Met genotype, parenting practices and adolescent alcohol use: testing the differential susceptibility hypothesis},
  series = {The journal of child psychology and psychiatry},
  volume    = {53},
  journal   = {The journal of child psychology and psychiatry},
  number    = {4},
  publisher = {Wiley-Blackwell},
  address   = {Malden},
  issn      = {0021-9630},
  doi       = {10.1111/j.1469-7610.2011.02408.x},
  pages     = {351 -- 359},
  year      = {2012},
  abstract  = {Background: Recently, first evidence has been reported for a geneparenting interaction (G x E) with regard to adolescent alcohol use. The present investigation set out to extend this research using the catechol-O-methyltransferase (COMT) Val158Met polymorphism as a genetic susceptibility factor. Moreover, the current study examined whether a potential G x E would be consistent with one of two models of geneenvironment interplay (genetic vulnerability vs. differential susceptibility). Methods: Data were collected as part of an ongoing epidemiological cohort study following the outcome of early risk factors from birth into adulthood. Two hundred and eighty-five participants (130 males, 155 females) were genotyped for the COMT Val(158) Met polymorphism and were administered an alcohol interview, providing measures of current frequency and amount of drinking at ages 15 and 19 years. Information on three dimensions of perceived parenting behavior was obtained from the 15-year-olds. Results: Adolescents homozygous for the Met allele showed higher drinking activity at age 19 years when their parents had engaged in less supervision or were less involved, while their drinking activity was reduced under conditions of favorable parenting. No such relationship was found in individuals carrying the Val allele. Conclusions: The present findings correspond with the pattern of results predicted by the differential susceptibility hypothesis, suggesting that environmental variation would have a greater impact in individuals carrying a genetic susceptibility such that, in this group, exposure to negative environmental conditions would result in more adverse outcomes and the experience of favorable conditions would lead to more positive outcomes.},
  language  = {en}
}
@article{KraheBondueHoeseetal.2015,
  author    = {Krah{\´e}, Barbara and Bond{\"u}, Rebecca and H{\"o}se, Anna and Esser, G{\"u}nter},
  title     = {Child Aggression as a Source and a Consequence of Parenting Stress: A Three-Wave Longitudinal Study},
  series = {Journal of research on adolescence : the official journal of the Society for Research on Adolescence},
  volume    = {25},
  journal   = {Journal of research on adolescence : the official journal of the Society for Research on Adolescence},
  number    = {2},
  publisher = {Wiley-Blackwell},
  address   = {Hoboken},
  issn      = {1050-8392},
  doi       = {10.1111/jora.12115},
  pages     = {328 -- 339},
  year      = {2015},
  abstract  = {This longitudinal study examined the links between child aggression and parenting stress over 4years. Child aggression was hypothesized to contribute to parenting stress, which should increase aggression. Parents and teachers of 239 German children aged between 6 and 15years completed measures of child aggression at Time 1 and Time 3, complemented by children's self-reports of aggression at Time 3. Parents rated their child-focused and parent-focused stress at an intermediate measurement Time 2. Child-focused stress mediated the path from Time 1 to Time 3 aggression in boys and girls, whereas parent-focused stress was unrelated to Time 3 aggression. The findings help to understand the continuity of aggressive behavior in childhood and adolescence and highlight the need to intervene early with families susceptible to parenting stress.},
  language  = {en}
}
@article{PitzerSchmidtEsseretal.2001,
  author    = {Pitzer, Martina and Schmidt, Martin H. and Esser, G{\"u}nter and Laucht, Manfred},
  title     = {Child development after maternal tocolysis with beta-sympathomimetic drugs},
  year      = {2001},
  abstract  = {The psycho-social development of both preterm and term children (n=347) whose mothers reported tocolytic treatment was assessed at the ages of 2, 4.5, 8 years. Term children exposed to tocolysis showed a higher rate of psychiatric disorders as well as poorer cognitive and motor performance than controls. In the preterm children no adverse impact of tocolysis could be found. The results are discussed concerning possible ways in which tocolytic treatment may influence child development. Restrictions because of the preliminary character of this study and the need of further prospective studies to clarify the developmental impact of tocolysis are also considered.},
  language  = {en}
}
@article{PitzerEsserSchmidtetal.2017,
  author    = {Pitzer, Martina and Esser, G{\"u}nter and Schmidt, Martin H. and Hohm, Erika and Banaschewski, Tobias and Laucht, Manfred},
  title     = {Child regulative temperament as a mediator of parenting in the development of depressive symptoms},
  series = {Journal of neural transmission},
  volume    = {124},
  journal   = {Journal of neural transmission},
  publisher = {Springer},
  address   = {Wien},
  issn      = {0300-9564},
  doi       = {10.1007/s00702-017-1682-2},
  pages     = {631 -- 641},
  year      = {2017},
  abstract  = {Child temperament as well as parenting behaviors have been linked to adolescent depression. Beyond their main effects, the interplay between these factors is of interest. For example, in an interactive model, a differential susceptibility of temperamental variants to parenting has been suggested. However, so far, the differential susceptibility hypothesis has mostly been studied with a focus on externalizing disorders. On the other hand, parenting may shape the child's temperament and vice versa in a transactional process. In a prospective, longitudinal at-risk sample (163 boys, 176 girls), we assessed emotional (easy-difficult) and regulative (self-control) temperament at ages 4.5, and 8 years, respectively, as well as parenting quality at age 4.5 years using the HOME inventory. Hierarchical linear regression analysis was used to investigate the prediction of depressive symptoms at age 11, measured by the Child Depression Inventory, including interaction terms between the temperament variable and parenting. We additionally tested whether parenting was mediated by child temperament. As previously reported, both self-control and parenting were longitudinally associated with preadolescent depressive symptoms. There were no interactive effects between temperament and parenting. However, the effects of parenting were partly mediated by self-control. Our data do not support a differential susceptibility of temperamental variants in the development of preadolescent depression. However, our results are in line with the assumption that parenting may shape young children's temperament, with positive parenting in the early childhood fostering the development of regulative temperament.},
  language  = {en}
}
@article{SchmidHohmBlomeyeretal.2007,
  author    = {Schmid, Brigitte and Hohm, Erika and Blomeyer, Dorothea and Zimmermann, Ulrich S. and Schmidt, Martin H. and Esser, G{\"u}nter and Laucht, Manfred},
  title     = {Concurrent alcohol and tobacco use during early adolescence characterizes a group at risk},
  issn      = {0735-0414},
  doi       = {10.1093/alcalc/agm024},
  year      = {2007},
  abstract  = {Aims: To investigate whether concurrent alcohol and tobacco use during early adolescence characterizes a subgroup that differs from users of one substance only regarding several risk factors for later substance use problems. Methods: Participants were from a prospective longitudinal cohort study of 384 children at risk for later psychopathology, with the majority being born with obstetric complications and psychosocial adversities. Assessments of adolescent drug consumption and related intrapersonal characteristics were obtained at age 15. Results: Compared to consumers of alcohol only, 15-year-olds drinking and smoking during the same time period (past 4 weeks) had significantly higher levels of consumption and more excessive use of alcohol, started drinking at an earlier age, had higher scores on the Fagerstrom Test for Nicotine Dependence, and more cannabis use. This group could be distinguished from users of alcohol only by higher novelty seeking and more positive alcohol effect expectancies. Compared to consumers of tobacco only, concurrent users reported higher nicotine dependence and more cannabis use. No significant differences were observed regarding frequency and age at initiation of tobacco use, tobacco-related sensitivity, self- efficacy and instrumentality as well as novelty seeking. Conclusions: Concurrent alcohol and tobacco use during early adolescence is associated with characteristics that are well known as risk factors for later alcohol use problems and dependence and that should be targeted by prevention programs.},
  language  = {en}
}
@article{DaigMahlbergStethinetal.2012,
  author    = {Daig, Isolde and Mahlberg, Richard and Stethin, Julia and Shroeder, Franziska and Wrase, Jana and Knoll, Nina and Bschor, Tom and Esser, G{\"u}nter and Heinz, Andreas and Kienast, Thorsten},
  title     = {Decreased verbal learning but not recognition performance in alcohol-dependent individuals during early abstinence},
  year      = {2012},
  language  = {en}
}
@book{IhleGroenWalteretal.2012,
  author    = {Ihle, Wolfgang and Groen, Gunter and Walter, Daniel and Esser, G{\"u}nter and Petermann, Franz},
  title     = {Depression},
  series = {Leitfaden Kinder- und Jugendpsychotherapie},
  volume    = {16},
  journal   = {Leitfaden Kinder- und Jugendpsychotherapie},
  publisher = {Hogrefe},
  address   = {G{\"o}ttingen},
  isbn      = {978-3-8017-2381-1},
  pages     = {IX, 162 S.},
  year      = {2012},
  language  = {de}
}
@article{IhleEsserLauchtetal.2004,
  author    = {Ihle, Wolfgang and Esser, G{\"u}nter and Laucht, Manfred and Schmidt, M. H.},
  title     = {Depressive St{\"o}rungen und aggressiv-dissoziale St{\"o}rungen im Kindes- und Jugendalter : Pr{\"a}valenz, Verlauf und Risikofaktoren},
  year      = {2004},
  language  = {de}
}
@article{EsserDinterJoerg2000,
  author    = {Esser, G{\"u}nter and Dinter-J{\"o}rg, Monika},
  title     = {Der Beitrag der direkten Beobachtung der Interaktion von Mutter und Kleinkind zur Vorhersage sp{\"a}terer psychischer Auff{\"a}lligkeiten},
  isbn      = {3-934252-66-4},
  year      = {2000},
  language  = {de}
}
@article{EsserIhle2000,
  author    = {Esser, G{\"u}nter and Ihle, Wolfgang},
  title     = {Der Verlauf psychischer St{\"o}rungen vom Kindes- zum Jugendalter},
  year      = {2000},
  language  = {de}
}
@article{LauchtEsserSchmidt1997,
  author    = {Laucht, Manfred and Esser, G{\"u}nter and Schmidt, Martin H.},
  title     = {Developmental outcome of infants born with biological and psychosocial risks},
  year      = {1997},
  language  = {en}
}