@phdthesis{Muessig1999,
  author    = {M{\"u}ssig, Carsten},
  title     = {Physiologie und molekulare Wirkungsweise der Brassinosteroide},
  address   = {Potsdam},
  pages     = {vi, 92 Bl. : graph. Darst.},
  year      = {1999},
  language  = {de}
}
@article{AltmannMuessig2001,
  author    = {Altmann, Thomas and M{\"u}ssig, Carsten},
  title     = {Brassinosteroid signaling in plants},
  year      = {2001},
  language  = {en}
}
@article{AltmannSchlueterKoepkeetal.2002,
  author    = {Altmann, Thomas and Schl{\"u}ter, U. and K{\"o}pke, D. and M{\"u}ssig, Carsten},
  title     = {Analysis of carbohydrate metabolism of CPD antisense plants and the brassinosteroid-deficient cbb1 mutant},
  year      = {2002},
  language  = {en}
}
@article{AltmannMuessigFischer2002,
  author    = {Altmann, Thomas and M{\"u}ssig, Carsten and Fischer, Sabine},
  title     = {Brassinosteroid-regulated gene expression},
  year      = {2002},
  language  = {en}
}
@article{AltmannToerjekBergeretal.2003,
  author    = {Altmann, Thomas and T{\"o}rjek, Otto and Berger, Dieter and Meyer, Rhonda C. and M{\"u}ssig, Carsten and Schmidt, K. J. and Sorensen, T. R. and Weisshaar, Bernd and Olds-Mitchell, T.},
  title     = {Establishment of a high-efficiency SNP-based framework marker set for Arabidopsis},
  year      = {2003},
  language  = {en}
}
@article{MuessigShinAltmann2003,
  author    = {M{\"u}ssig, Carsten and Shin, G.-H. and Altmann, Thomas},
  title     = {Brassinosteroids promote root growth in Arabidopsis},
  year      = {2003},
  language  = {en}
}
@article{MuessigAltmann2003,
  author    = {M{\"u}ssig, Carsten and Altmann, Thomas},
  title     = {Changes in gene expression in response to altered SHL transcript levels},
  year      = {2003},
  abstract  = {The nuclear SHL protein is composed of a N-terminal BAH domain and a C-terminal PHD finger. Both domains are found in transcriptional regulators and chromatin-modifying proteins. Arabidopsis plants over-expressing SHL showed earlier flowering and senescence phenotype. To identify SHL regulated genes, expression profiles of 35S::SHL plants were established with Affymetrix ATH1 microarrays. About 130 genes showed reduced transcript levels, and about 45 genes showed increased transcript levels in 35S:: SHL plants. The up-regulated genes included AGL20 and AGL9, which most likely cause the early flowering phenotype of 35S:: SHL plants. Late-flowering SHL-antisense lines showed reduced AGL20 mRNA levels, suggesting that AGL20 gene expression depends on the SHL protein. The stronger expression of senescence- and defence-related genes (such as DIN2, DIN11 and PR-1) is in line with the early senescence phenotype of SHL-over- expressing plants. SHL-down-regulated genes included stress response genes and the PSR3.2 gene (encoding a beta- glucosidase). SHL over-expression did not alter the tissue specificity of PSR3.2 gene expression, but resulted in reduced transcript levels in both shoots and roots. Plants with glucocorticoid-inducible SHL over-expression were established and used for expression profiling as well. A subset of genes was identified, which showed consistent changes in the inducible system and in plants with constitutive SHL over-expression},
  language  = {en}
}
@article{MuessigAltmann2003,
  author    = {M{\"u}ssig, Carsten and Altmann, Thomas},
  title     = {Genomic brassinosteroid effects},
  year      = {2003},
  abstract  = {Detailed analysis of brassinosteroid (BR)-regulated genes can provide evidence of the molecular basis of BR effects. Classical techniques (such as subtractive cDNA cloning) as well as cDNA and oligonucleotide microarrays have been applied to identify genes which are upregulated or downregulated after BR treatment or are differently expressed in BR-deficient or -insensitive mutants compared with wild type plants. Genes encoding cell-wall-modifying enzymes, enzymes of the BR biosynthetic pathway, auxin response factors, and transcription factors are subject to BR regulation. Effects on several other metabolic pathways and interactions with other phytohormones have been reported as well, although some of these effects may depend on certain environmental conditions (for example, light/dark or stress), the developmental stage of the plants, and tissue types. The identification of components of the BR signal transduction pathway revealed different modes of transcriptional control in animals and plants. Steroid signaling in plants comprises the plasma membrane receptor kinases BRI1 and BAK1 and intracellular protein phosphorylations. Thus, BR signaling in plants is reminiscent of growth factor and TGF-beta signal transduction in animals. The phosphorylation cascade could be a basis of extensive signaling cross-talk and thereby explain the complexity of BR responses},
  language  = {en}
}
@article{MeyerMuessigAltmann2004,
  author    = {Meyer, Rhonda C. and M{\"u}ssig, Carsten and Altmann, Thomas},
  title     = {Genetic Diversity : Creation of novel genetic variants of arabidopsis},
  isbn      = {3-00-011587-0},
  year      = {2004},
  language  = {en}
}
@article{GollGarciaMazuchAltmannetal.2004,
  author    = {Goll-Garcia, D. and Mazuch, J. and Altmann, Thomas and M{\"u}ssig, Carsten},
  title     = {Exordium regulates brassinosteroid-responsive genes},
  year      = {2004},
  abstract  = {In a screen for potential mediators of brassinosteroid (BR) effects, the EXORDIUM (EXO) protein was identified as a regulator of BR-responsive genes. The EXO gene was characterized as a BR-up-regulated gene. EXO overexpression under the control of the 35SCaMV promoter resulted in increased transcript levels of the BR-up-regulated KCS1, Exp5, delta-TIP, and AGP4 genes, which likely are involved in the mediation of BR-promoted growth. 35S::EXO lines grown in soil or in synthetic medium showed increased vegetative growth in comparison to wild-type plants, resembling the growth phenotype of BR-treated plants. Thus, the EXO protein most likely promotes growth via the modulation of gene expression patterns. (C) 2004 Federation of European Biochemical Societies. Published by Elsevier B.V. All rights reserved},
  language  = {en}
}