@article{LissoAltmannMuessig2006,
  author    = {Lisso, Janina and Altmann, Thomas and M{\"u}ssig, Carsten},
  title     = {Metabolic changes in fruits of the tomato d(x) mutant},
  series = {Phytochemistry : an international journal of plant biochemistry},
  volume    = {67},
  journal   = {Phytochemistry : an international journal of plant biochemistry},
  number    = {20},
  publisher = {Elsevier},
  address   = {Oxford},
  issn      = {0031-9422},
  doi       = {10.1016/j.phytochem.2006.07.008},
  pages     = {2232 -- 2238},
  year      = {2006},
  language  = {en}
}
@article{LissoAltmannMuessig2006,
  author    = {Lisso, Janina and Altmann, Thomas and M{\"u}ssig, Carsten},
  title     = {The AtNFXL1 gene encodes a NF-X1 type zinc finger protein required for growth under salt stress},
  series = {FEBS letters : the journal for rapid publication of short reports in molecular biosciences},
  volume    = {580},
  journal   = {FEBS letters : the journal for rapid publication of short reports in molecular biosciences},
  number    = {22},
  publisher = {Elsevier},
  address   = {Amsterdam},
  issn      = {0014-5793},
  doi       = {10.1016/j.febslet.2006.07.079},
  pages     = {4851 -- 4856},
  year      = {2006},
  abstract  = {The human NF-X1 protein and homologous proteins in eukaryotes represent a class of transcription factors which are characterised. by NF-X1 type zinc finger motifs. The Arabidopsis genome encodes two NF-X1 homologs, which we termed AtNFXL1 and AtNFXL2. Growth and survival was impaired in atnfxl1 knock-out mutants and AtNFXL1-antisense plants under salt stress in comparison to wild-type plants. In contrast, 35S: :AtNFXL1 plants showed higher survival rates. The AtNFXL2 protein potentially plays an antagonistic role. The Arabidopsis NF-X1 type zinc finger proteins likely are part of regulatory mechanisms, which protect major processes such as photosynthesis.},
  language  = {en}
}
@phdthesis{Muessig2006,
  author    = {M{\"u}ssig, Carsten},
  title     = {Molekulare Grundlagen der wachstumsf{\"o}rdernden Wirkung der Brassinosteroide},
  address   = {Potsdam},
  pages     = {Getr. Z{\"a}hl. : graph. Darst.},
  year      = {2006},
  language  = {de}
}
@article{LissoSteinhaeuserAltmannetal.2005,
  author    = {Lisso, Janina and Steinhaeuser, Dirk and Altmann, Thomas and Kopka, Joachim and M{\"u}ssig, Carsten},
  title     = {Identification of brassinosteroid-related genes by means of transcript co-response analyses},
  issn      = {0305-1048},
  year      = {2005},
  abstract  = {The comprehensive systems-biology database (CSB.DB) was used to reveal brassinosteroid (BR)-related genes from expression profiles based on co-response analyses. Genes exhibiting simultaneous changes in transcript levels are candidates of common transcriptional regulation. Combining numerous different experiments in data matrices allows ruling out outliers and conditional changes of transcript levels. CSB.DB was queried for transcriptional co-responses with the BR-signalling components BRI1 and BAK1: 301 out of 9694 genes represented in the nasc0271 database showed co-responses with both genes. As expected, these genes comprised pathway-involved genes (e.g. 72 BR-induced genes), because the BRI1 and BAK1 proteins are required for BR-responses. But transcript co-response takes the analysis a step further compared with direct approaches because BR-related non BR-responsive genes were identified. Insights into networks and the functional context of genes are provided, because factors determining expression patterns are reflected in correlations. Our findings demonstrate that transcript co-response analysis presents a valuable resource to uncover common regulatory patterns of genes. Different data matrices in CSB.DB allow examination of specific biological questions. All matrices are publicly available through CSB.DB. This work presents one possible roadmap to use the CSB.DB resources},
  language  = {en}
}
@article{MeyerMuessigAltmann2004,
  author    = {Meyer, Rhonda C. and M{\"u}ssig, Carsten and Altmann, Thomas},
  title     = {Genetic Diversity : Creation of novel genetic variants of arabidopsis},
  isbn      = {3-00-011587-0},
  year      = {2004},
  language  = {en}
}
@article{GollGarciaMazuchAltmannetal.2004,
  author    = {Goll-Garcia, D. and Mazuch, J. and Altmann, Thomas and M{\"u}ssig, Carsten},
  title     = {Exordium regulates brassinosteroid-responsive genes},
  year      = {2004},
  abstract  = {In a screen for potential mediators of brassinosteroid (BR) effects, the EXORDIUM (EXO) protein was identified as a regulator of BR-responsive genes. The EXO gene was characterized as a BR-up-regulated gene. EXO overexpression under the control of the 35SCaMV promoter resulted in increased transcript levels of the BR-up-regulated KCS1, Exp5, delta-TIP, and AGP4 genes, which likely are involved in the mediation of BR-promoted growth. 35S::EXO lines grown in soil or in synthetic medium showed increased vegetative growth in comparison to wild-type plants, resembling the growth phenotype of BR-treated plants. Thus, the EXO protein most likely promotes growth via the modulation of gene expression patterns. (C) 2004 Federation of European Biochemical Societies. Published by Elsevier B.V. All rights reserved},
  language  = {en}
}
@article{AltmannToerjekBergeretal.2003,
  author    = {Altmann, Thomas and T{\"o}rjek, Otto and Berger, Dieter and Meyer, Rhonda C. and M{\"u}ssig, Carsten and Schmidt, K. J. and Sorensen, T. R. and Weisshaar, Bernd and Olds-Mitchell, T.},
  title     = {Establishment of a high-efficiency SNP-based framework marker set for Arabidopsis},
  year      = {2003},
  language  = {en}
}
@article{MuessigShinAltmann2003,
  author    = {M{\"u}ssig, Carsten and Shin, G.-H. and Altmann, Thomas},
  title     = {Brassinosteroids promote root growth in Arabidopsis},
  year      = {2003},
  language  = {en}
}
@article{MuessigAltmann2003,
  author    = {M{\"u}ssig, Carsten and Altmann, Thomas},
  title     = {Changes in gene expression in response to altered SHL transcript levels},
  year      = {2003},
  abstract  = {The nuclear SHL protein is composed of a N-terminal BAH domain and a C-terminal PHD finger. Both domains are found in transcriptional regulators and chromatin-modifying proteins. Arabidopsis plants over-expressing SHL showed earlier flowering and senescence phenotype. To identify SHL regulated genes, expression profiles of 35S::SHL plants were established with Affymetrix ATH1 microarrays. About 130 genes showed reduced transcript levels, and about 45 genes showed increased transcript levels in 35S:: SHL plants. The up-regulated genes included AGL20 and AGL9, which most likely cause the early flowering phenotype of 35S:: SHL plants. Late-flowering SHL-antisense lines showed reduced AGL20 mRNA levels, suggesting that AGL20 gene expression depends on the SHL protein. The stronger expression of senescence- and defence-related genes (such as DIN2, DIN11 and PR-1) is in line with the early senescence phenotype of SHL-over- expressing plants. SHL-down-regulated genes included stress response genes and the PSR3.2 gene (encoding a beta- glucosidase). SHL over-expression did not alter the tissue specificity of PSR3.2 gene expression, but resulted in reduced transcript levels in both shoots and roots. Plants with glucocorticoid-inducible SHL over-expression were established and used for expression profiling as well. A subset of genes was identified, which showed consistent changes in the inducible system and in plants with constitutive SHL over-expression},
  language  = {en}
}
@article{MuessigAltmann2003,
  author    = {M{\"u}ssig, Carsten and Altmann, Thomas},
  title     = {Genomic brassinosteroid effects},
  year      = {2003},
  abstract  = {Detailed analysis of brassinosteroid (BR)-regulated genes can provide evidence of the molecular basis of BR effects. Classical techniques (such as subtractive cDNA cloning) as well as cDNA and oligonucleotide microarrays have been applied to identify genes which are upregulated or downregulated after BR treatment or are differently expressed in BR-deficient or -insensitive mutants compared with wild type plants. Genes encoding cell-wall-modifying enzymes, enzymes of the BR biosynthetic pathway, auxin response factors, and transcription factors are subject to BR regulation. Effects on several other metabolic pathways and interactions with other phytohormones have been reported as well, although some of these effects may depend on certain environmental conditions (for example, light/dark or stress), the developmental stage of the plants, and tissue types. The identification of components of the BR signal transduction pathway revealed different modes of transcriptional control in animals and plants. Steroid signaling in plants comprises the plasma membrane receptor kinases BRI1 and BAK1 and intracellular protein phosphorylations. Thus, BR signaling in plants is reminiscent of growth factor and TGF-beta signal transduction in animals. The phosphorylation cascade could be a basis of extensive signaling cross-talk and thereby explain the complexity of BR responses},
  language  = {en}
}